A trans-activator function is generated by integration of hepatitis B virus preS/S sequences in human hepatocellular carcinoma DNA.
about
Hepatocellular carcinoma and hepatitis B surface proteinStudy of transactivating effect of pre-S2 protein of hepatitis B virus and cloning of genes transactivated by pre-S2 protein with suppression subtractive hybridization.Hepatitis B viral surface mutations in patients with adefovir resistant chronic hepatitis B with A181T/V polymerase mutations.Hepatitis B virus-induced oncogenesis.Pathogenesis of hepatitis B virus infectionInteraction of Hepatitis B virus with cellular processes in liver carcinogenesis.Novel F141L pre-S2 mutation in hepatitis B virus increases the risk of hepatocellular carcinoma in patients with chronic genotype C infections.Clinical characteristics and outcome of a cohort of 101 patients with hepatocellular carcinoma.Male-specific hepatitis B virus large surface protein variant W4P potentiates tumorigenicity and induces gender disparityIdentification of transforming hepatitis B virus S gene nonsense mutations derived from freely replicative viruses in hepatocellular carcinomaHepatitis B virus infection and primary hepatocellular carcinoma.Hepatitis B virus preS1 deletion is related to viral replication increase and disease progressionClinical relevance of hepatitis B virus variantsAssociation of preS/S Mutations with Occult Hepatitis B Virus (HBV) Infection in South Korea: Transmission Potential of Distinct Occult HBV Variants.The 1995 Walter Hubert Lecture--molecular epidemiology of human cancer: insights from the mutational analysis of the p53 tumour-suppressor gene.The molecular diagnosis of hepatitis B virus-associated hepatocellular carcinoma.Analysis of integrated ground squirrel hepatitis virus and flanking host DNA in two hepatocellular carcinomas.Up-regulation of intercellular adhesion molecule 1 transcription by hepatitis B virus X protein.Male-specific W4P/R mutation in the pre-S1 region of hepatitis B virus, increasing the risk of progression of liver diseases in chronic patients.Identification and characterization of genotype A and D recombinant hepatitis B virus from Indian chronic HBV isolates.Hepatocarcinogenesis in transgenic mice carrying hepatitis B virus pre-S/S gene with the sW172* mutation.Hepatitis B virus mutations related to liver disease progression of Korean patients.Frequent amplification of c-myc in ground squirrel liver tumors associated with past or ongoing infection with a hepadnavirus.Hepatitis B virus surface (S) transactivator with DNA-binding properties.Hepatitis B virus transactivator MHBst: activation of NF-kappa B, selective inhibition by antioxidants and integral membrane localization.Expression profile of nine novel genes differentially expressed in hepatitis B virus-associated hepatocellular carcinomas.HBx triggers either cellular senescence or cell proliferation depending on cellular phenotype.Molecular analysis of Hepatitis B virus sub-genotypes and incidence of preS1/preS2 region mutations in HBV-infected Egyptian patients from Mansoura.HCC-Associated Single Nucleotide Variants and Deletions Identified Using Genome-Wide High Throughput Analysis of HBV.Dysregulated surface gene expression from disrupted hepatitis B virus genomes.Analysis of the complete hepatitis B virus genome in patients with genotype C chronic hepatitis and hepatocellular carcinoma.A molecular epidemiology study investigating familial clustering of hepatitis B virus infection in families with unfavorable prognoses in Northwest China.The role of hepatitis B virus in development of primary hepatocellular carcinoma: Part II.The molecular pathogenesis of hepatocellular carcinoma.Hepatitis B virus transcriptional activators: mechanisms and possible role in oncogenesis.Chromosome alterations in human hepatocellular carcinomas correlate with aetiology and histological grade--results of an explorative CGH meta-analysis.Cloning of differentially expressed genes in human hepatocellular carcinoma and nontumor liver.Characteristics of human hepatocellular carcinoma cell lines (Hep-KANO) derived from a non-hepatitic, non-cirrhotic hepatitis B virus carrier.Differentially expressed genes in hepatocellular carcinoma induced by woodchuck hepatitis B virus in mice.Clinical significance of pre-S mutations in patients with genotype C hepatitis B virus infection.
P2860
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P2860
A trans-activator function is generated by integration of hepatitis B virus preS/S sequences in human hepatocellular carcinoma DNA.
description
1990 nî lūn-bûn
@nan
1990 թուականի Ապրիլին հրատարակուած գիտական յօդուած
@hyw
1990 թվականի ապրիլին հրատարակված գիտական հոդված
@hy
1990年の論文
@ja
1990年論文
@yue
1990年論文
@zh-hant
1990年論文
@zh-hk
1990年論文
@zh-mo
1990年論文
@zh-tw
1990年论文
@wuu
name
A trans-activator function is ...... hepatocellular carcinoma DNA.
@ast
A trans-activator function is ...... hepatocellular carcinoma DNA.
@en
type
label
A trans-activator function is ...... hepatocellular carcinoma DNA.
@ast
A trans-activator function is ...... hepatocellular carcinoma DNA.
@en
prefLabel
A trans-activator function is ...... hepatocellular carcinoma DNA.
@ast
A trans-activator function is ...... hepatocellular carcinoma DNA.
@en
P2093
P2860
P356
P1476
A trans-activator function is ...... hepatocellular carcinoma DNA.
@en
P2093
P2860
P304
P356
10.1073/PNAS.87.8.2970
P407
P577
1990-04-01T00:00:00Z