Multiple sclerosis: is there neurodegeneration independent from inflammation?
about
Multiple sclerosis, relapses, and the mechanism of action of adrenocorticotropic hormoneKallikreins are associated with secondary progressive multiple sclerosis and promote neurodegenerationExploring potential mechanisms of action of natalizumab in secondary progressive multiple sclerosisPharmacological Alternatives for the Treatment of Neurodegenerative Disorders: Wasp and Bee Venoms and Their Components as New Neuroactive ToolsBrain-Specific Cytoskeletal Damage Markers in Cerebrospinal Fluid: Is There a Common Pattern between Amyotrophic Lateral Sclerosis and Primary Progressive Multiple Sclerosis?Progressive multiple sclerosisPrimary progressive multiple sclerosis: current therapeutic strategies and future perspectivesSodium MRI of multiple sclerosis.CD200R1 agonist attenuates mechanisms of chronic disease in a murine model of multiple sclerosis.Proteome analysis of spinal cord during the clinical course of monophasic experimental autoimmune encephalomyelitis.Extracellular acidic pH inhibits oligodendrocyte precursor viability, migration, and differentiationOral administration of the KATP channel opener diazoxide ameliorates disease progression in a murine model of multiple sclerosis.Multimodal quantitative magnetic resonance imaging of thalamic development and aging across the human lifespan: implications to neurodegeneration in multiple sclerosis.Disruption in the Blood-Brain Barrier: The Missing Link between Brain and Body Inflammation in Bipolar Disorder?Human brain atlas-based multimodal MRI analysis of volumetry, diffusimetry, relaxometry and lesion distribution in multiple sclerosis patients and healthy adult controls: implications for understanding the pathogenesis of multiple sclerosis and consImmunosuppressive potential of human amnion epithelial cells in the treatment of experimental autoimmune encephalomyelitisMulti-modal quantitative MRI investigation of brain tissue neurodegeneration in multiple sclerosisQuantitative evaluation of cerebral white matter in patients with multiple sclerosis using multicomponent T2 mapping.Increased PK11195 PET binding in the cortex of patients with MS correlates with disabilityPharmacological Approaches to the Management of Secondary Progressive Multiple Sclerosis.Inhibition of neuronal degenerin/epithelial Na+ channels by the multiple sclerosis drug 4-aminopyridine.Neuroprotection with glatiramer acetate: evidence from the PreCISe trial.The cuprizone model for demyelination.Electrodiagnostic evaluation of peripheral nervous system changes in patients with multiple sclerosis.Advancement of therapies for neuroprotection in multiple sclerosis.Neuroinflammation, neurodegeneration and regeneration in multiple sclerosis: intercorrelated manifestations of the immune response.The remyelination Philosopher's Stone: stem and progenitor cell therapies for multiple sclerosis.Ten years of proteomics in multiple sclerosis.Thyroid Hormone Potentially Benefits Multiple Sclerosis via Facilitating Remyelination.How can proteomics elucidate the complexity of multiple sclerosis?Rapid depletion of B lymphocytes by ultra-low-dose rituximab delivered intrathecally.Topography of brain sodium accumulation in progressive multiple sclerosis.Normal-appearing white matter permeability distinguishes poor cognitive performance in processing speed and working memory.Acid-sensing ion channel-1 contributes to axonal degeneration in autoimmune inflammation of the central nervous system.Consistent induction of chronic experimental autoimmune encephalomyelitis in C57BL/6 mice for the longitudinal study of pathology and repair.Normal appearing white matter permeability: a marker of inflammation and information processing speed deficit among relapsing remitting multiple sclerosis patients.Systemic inhibition of the membrane attack complex impedes neuroinflammation in chronic relapsing experimental autoimmune encephalomyelitis.Multiple Sclerosis and Chronic Cerebrospinal Venous Insufficiency: The Neuroimaging Perspective
P2860
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P2860
Multiple sclerosis: is there neurodegeneration independent from inflammation?
description
2007 nî lūn-bûn
@nan
2007 թուականի Մարտին հրատարակուած գիտական յօդուած
@hyw
2007 թվականի մարտին հրատարակված գիտական հոդված
@hy
2007年の論文
@ja
2007年論文
@yue
2007年論文
@zh-hant
2007年論文
@zh-hk
2007年論文
@zh-mo
2007年論文
@zh-tw
2007年论文
@wuu
name
Multiple sclerosis: is there neurodegeneration independent from inflammation?
@ast
Multiple sclerosis: is there neurodegeneration independent from inflammation?
@en
type
label
Multiple sclerosis: is there neurodegeneration independent from inflammation?
@ast
Multiple sclerosis: is there neurodegeneration independent from inflammation?
@en
prefLabel
Multiple sclerosis: is there neurodegeneration independent from inflammation?
@ast
Multiple sclerosis: is there neurodegeneration independent from inflammation?
@en
P1476
Multiple sclerosis: is there neurodegeneration independent from inflammation?
@en
P2093
Hans Lassmann
P356
10.1016/J.JNS.2006.08.016
P577
2007-03-23T00:00:00Z