Hindbrain and cranial nerve dysmorphogenesis result from acute maternal ethanol administration.
about
Definition of critical periods for Hedgehog pathway antagonist-induced holoprosencephaly, cleft lip, and cleft palateChorioallantoic fusion defects and embryonic lethality resulting from disruption of Zfp36L1, a gene encoding a CCCH tandem zinc finger protein of the Tristetraprolin familyStrain differences in developmental vulnerability to alcohol exposure via embryo culture in mice.Hedgehog pathway responsiveness correlates with the presence of primary cilia on prostate stromal cellsCommonality in Down and fetal alcohol syndromes.Molecular pathways underpinning ethanol-induced neurodegeneration.Cleft lip and palate results from Hedgehog signaling antagonism in the mouse: Phenotypic characterization and clinical implicationsAcute prenatal exposure to ethanol and social behavior: effects of age, sex, and timing of exposure.Prenatal alcohol exposure triggers ceramide-induced apoptosis in neural crest-derived tissues concurrent with defective cranial development.Time-specific effects of ethanol exposure on cranial nerve nuclei: gastrulation and neuronogenesis.Low and moderate prenatal ethanol exposures of mice during gastrulation or neurulation delays neurobehavioral development.Genesis of alcohol-induced craniofacial dysmorphism.Neuroimmune mechanisms in fetal alcohol spectrum disorderUsing drinking in the dark to model prenatal binge-like exposure to ethanol in C57BL/6J mice.Vulnerability of macaque cranial nerve neurons to ethanol is time- and site-dependent.Loss of motoneurons in the ventral compartment of the rat hypoglossal nucleus following early postnatal exposure to alcohol.Double bromodomain-containing gene Brd2 is essential for embryonic development in mouseAgrin function associated with ocular development is a target of ethanol exposure in embryonic zebrafish.Forebrain and hindbrain development in zebrafish is sensitive to ethanol exposure involving agrin, Fgf, and sonic hedgehog function.Oxidative stress and cellular and tissue damage in organogenic outbred mouse embryos after moderate perigestational alcohol intake.Embryo developmental disruption during organogenesis produced by CF-1 murine periconceptional alcohol consumption.Maternal oral intake mouse model for fetal alcohol spectrum disorders: ocular defects as a measure of effect.
P2860
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P2860
Hindbrain and cranial nerve dysmorphogenesis result from acute maternal ethanol administration.
description
2002 nî lūn-bûn
@nan
2002 թուականի Յունուարին հրատարակուած գիտական յօդուած
@hyw
2002 թվականի հունվարին հրատարակված գիտական հոդված
@hy
2002年の論文
@ja
2002年論文
@yue
2002年論文
@zh-hant
2002年論文
@zh-hk
2002年論文
@zh-mo
2002年論文
@zh-tw
2002年论文
@wuu
name
Hindbrain and cranial nerve dy ...... ternal ethanol administration.
@ast
Hindbrain and cranial nerve dy ...... ternal ethanol administration.
@en
type
label
Hindbrain and cranial nerve dy ...... ternal ethanol administration.
@ast
Hindbrain and cranial nerve dy ...... ternal ethanol administration.
@en
prefLabel
Hindbrain and cranial nerve dy ...... ternal ethanol administration.
@ast
Hindbrain and cranial nerve dy ...... ternal ethanol administration.
@en
P2093
P356
P1476
Hindbrain and cranial nerve dy ...... ternal ethanol administration.
@en
P2093
Kathleen K Sulik
Robert M Zucker
William C Dunty
P304
P356
10.1159/000066748
P577
2002-01-01T00:00:00Z