C5a regulates IL-12+ DC migration to induce pathogenic Th1 and Th17 cells in sepsis. - Wikidata - wikimedia - TriplyDB

C5a regulates IL-12+ DC migration to induce pathogenic Th1 and Th17 cells in sepsis.

C5a regulates IL-12+ DC migration to induce pathogenic Th1 and Th17 cells in sepsis.

http://www.wikidata.org/entity/Q31126184

about

Activated Complement Factors as Disease Markers for Sepsis Attenuation of cGVHD by C5a/C5aR blockade is associated with increased frequency of Treg Alterations of dendritic cells in sepsis: featured role in immunoparalysis.T-cell autophagy deficiency increases mortality and suppresses immune responses after sepsis.Effect of tumor necrosis factor-α induced protein 8 like-2 on immune function of dendritic cells in mice following acute insults.Mechanism of C5a-induced immunologic derangement in sepsis.Antiseptic effect of sea cucumber (Holothuria atra) against multi-organ failure induced by sepsis: Molecular and histopathological study.Dendritic Cells in Sepsis: Pathological Alterations and Therapeutic Implications.Complement After Trauma: Suturing Innate and Adaptive Immunity

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C5a regulates IL-12+ DC migration to induce pathogenic Th1 and Th17 cells in sepsis.

http://www.wikidata.org/entity/Q31126184

description

2013 nî lūn-bûn

@nan

2013 թուականի Յուլիսին հրատարակուած գիտական յօդուած

@hyw

2013 թվականի հուլիսին հրատարակված գիտական հոդված

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2013年の論文

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2013年論文

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2013年論文

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2013年論文

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2013年論文

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2013年論文

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2013年论文

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name

C5a regulates IL-12+ DC migration to induce pathogenic Th1 and Th17 cells in sepsis.

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C5a regulates IL-12+ DC migration to induce pathogenic Th1 and Th17 cells in sepsis.

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type

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C5a regulates IL-12+ DC migration to induce pathogenic Th1 and Th17 cells in sepsis.

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C5a regulates IL-12+ DC migration to induce pathogenic Th1 and Th17 cells in sepsis.

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C5a regulates IL-12+ DC migration to induce pathogenic Th1 and Th17 cells in sepsis.

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C5a regulates IL-12+ DC migration to induce pathogenic Th1 and Th17 cells in sepsis.

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C5a regulates IL-12+ DC migration to induce pathogenic Th1 and Th17 cells in sepsis.

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Beifen Shen

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Bernadette Marrero

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Chen Xing

http://www.w3.org/2001/XMLSchema#string

Chunmei Hou

http://www.w3.org/2001/XMLSchema#string

Gencheng Han

http://www.w3.org/2001/XMLSchema#string

Guojiang Chen

http://www.w3.org/2001/XMLSchema#string

He Xiao

http://www.w3.org/2001/XMLSchema#string

Ke Wang

http://www.w3.org/2001/XMLSchema#string

Ning Ma

http://www.w3.org/2001/XMLSchema#string

Yan Li

http://www.w3.org/2001/XMLSchema#string

P2860

Definitions for sepsis and organ failure and guidelines for the use of innovative therapies in sepsis. The ACCP/SCCM Consensus Conference Committee. American College of Chest Physicians/Society of Critical Care Medicine

Complement-induced impairment of innate immunity during sepsis

The dark side of C5a in sepsis

Expression and function of the C5a receptor in rat alveolar epithelial cells

Immunobiology of dendritic cells

The epidemiology of sepsis in the United States from 1979 through 2000

2001 SCCM/ESICM/ACCP/ATS/SIS International Sepsis Definitions Conference

Absence of signaling into CD4⁺ cells via C3aR and C5aR enables autoinductive TGF-β1 signaling and induction of Foxp3⁺ regulatory T cells.

Dendritic cells as sensors of infection.

Endotoxin-induced shock in the rat. A role for C5a

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