Part of membrane-bound Abeta exists in rafts within senile plaques in Tg2576 mouse brain.
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Exosomes: vehicles for the transfer of toxic proteins associated with neurodegenerative diseases?Flotillins bind to the dileucine sorting motif of β-site amyloid precursor protein-cleaving enzyme 1 and influence its endosomal sortingMyeloid Extracellular Vesicles: Messengers from the Demented BrainAccumulation of amyloid-β by astrocytes result in enlarged endosomes and microvesicle-induced apoptosis of neuronsDendritic and axonal mechanisms of Ca2+ elevation impair BDNF transport in Aβ oligomer-treated hippocampal neuronsExtracellular Vesicles as a Source of Urological Biomarkers: Lessons Learned From Advances and Challenges in Clinical Applications to Major Diseases.Small amphipathic molecules modulate secondary structure and amyloid fibril-forming kinetics of Alzheimer disease peptide Aβ(1-42).Alzheimer's disease beta-amyloid peptides are released in association with exosomes.Effects of human apolipoprotein E isoforms on the amyloid beta-protein concentration and lipid composition in brain low-density membrane domainsIdentification and proteomic profiling of exosomes in human cerebrospinal fluid.Dynein dysfunction induces endocytic pathology accompanied by an increase in Rab GTPases: a potential mechanism underlying age-dependent endocytic dysfunctionEmerging roles of extracellular vesicles in neurodegenerative disorders: focus on HIV-associated neurological complications.Plasma Exosomes Spread and Cluster Around β-Amyloid Plaques in an Animal Model of Alzheimer's Disease.The secret life of extracellular vesicles in metal homeostasis and neurodegeneration.The effect of aging on brain barriers and the consequences for Alzheimer's disease development.Exosomes Secreted from HEK293-APP Swe/Ind Cells Impair the Hippocampal Neurogenesis.Exosomes and the Prion Protein: More than One Truth.Accelerated release of exosome-associated GM1 ganglioside (GM1) by endocytic pathway abnormality: another putative pathway for GM1-induced amyloid fibril formation.β-Amyloid is transmitted via neuronal connections along axonal membranes.Let's make microglia great again in neurodegenerative disorders.Hypoxia increases amyloid-β level in exosomes by enhancing the interaction between CD147 and Hook1.
P2860
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P2860
Part of membrane-bound Abeta exists in rafts within senile plaques in Tg2576 mouse brain.
description
2005 nî lūn-bûn
@nan
2005 թուականի Ապրիլին հրատարակուած գիտական յօդուած
@hyw
2005 թվականի ապրիլին հրատարակված գիտական հոդված
@hy
2005年の論文
@ja
2005年論文
@yue
2005年論文
@zh-hant
2005年論文
@zh-hk
2005年論文
@zh-mo
2005年論文
@zh-tw
2005年论文
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name
Part of membrane-bound Abeta exists in rafts within senile plaques in Tg2576 mouse brain.
@ast
Part of membrane-bound Abeta exists in rafts within senile plaques in Tg2576 mouse brain.
@en
type
label
Part of membrane-bound Abeta exists in rafts within senile plaques in Tg2576 mouse brain.
@ast
Part of membrane-bound Abeta exists in rafts within senile plaques in Tg2576 mouse brain.
@en
prefLabel
Part of membrane-bound Abeta exists in rafts within senile plaques in Tg2576 mouse brain.
@ast
Part of membrane-bound Abeta exists in rafts within senile plaques in Tg2576 mouse brain.
@en
P2093
P1476
Part of membrane-bound Abeta exists in rafts within senile plaques in Tg2576 mouse brain.
@en
P2093
Haruyasu Yamaguchi
Hideko Kokubo
J Bernd Helms
Nobuhisa Iwata
Rumi Shinohara
Takaomi C Saido
P304
P356
10.1016/J.NEUROBIOLAGING.2004.04.008
P577
2005-04-01T00:00:00Z