Effects of adenovirus-mediated sorcin overexpression on excitation-contraction coupling in isolated rabbit cardiomyocytes.
about
Cytoplasmic tail of phospholemman interacts with the intracellular loop of the cardiac Na+/Ca2+ exchangerSorcin modulation of Ca2+ sparks in rat vascular smooth muscle cellsMechanism of calmodulin inhibition of cardiac sarcoplasmic reticulum Ca2+ release channel (ryanodine receptor)Activation of the cardiac Na(+)-Ca(2+) exchanger by sorcin via the interaction of the respective Ca(2+)-binding domains.Atrial myocyte function and Ca2+ handling is associated with inborn aerobic capacity.Genetic maneuvers to ameliorate ventricular function in heart failure: therapeutic potential and future implications.Sorcin modulates mitochondrial Ca(2+) handling and reduces apoptosis in neonatal rat cardiac myocytes.The regulation of the Na/Ca exchanger and plasmalemmal Ca2+ ATPase by other proteins.Chronic myocardial infarction promotes atrial action potential alternans, afterdepolarizations, and fibrillation.Targeting altered calcium physiology in the heart: translational approaches to excitation, contraction, and transcription.Sequestration of sorcin by aberrant forms of tau results in the defective calcium homeostasis.Sensitivity of rabbit ventricular action potential and Ca²⁺ dynamics to small variations in membrane currents and ion diffusion coefficients.Designing heart performance by gene transfer.Molecular cardiology in translation: gene, cell and chemical-based experimental therapeutics for the failing heart.Therapeutic potential of genes in cardiac repair.Sorcin, a calcium binding protein involved in the multidrug resistance mechanisms in cancer cells.Ca(2+)-binding proteins in dogs with heart failure: effects of cardiac contractility modulation electrical signals.Over-expression of FK506-binding protein FKBP12.6 alters excitation-contraction coupling in adult rabbit cardiomyocytes.Reconstitution of local Ca2+ signaling between cardiac L-type Ca2+ channels and ryanodine receptors: insights into regulation by FKBP12.6.Sorcin ablation plus β-adrenergic stimulation generate an arrhythmogenic substrate in mouse ventricular myocytes.17β-Estradiol and/or estrogen receptor alpha blocks isoproterenol-induced calcium accumulation and hypertrophy via GSK3β/PP2A/NFAT3/ANP pathway.Sorcin modulates cardiac L-type Ca2+ current by functional interaction with the alpha1C subunit in rabbits.Na(+)/Ca(2+) exchanger expression and function in a rabbit model of myocardial infarction.
P2860
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P2860
Effects of adenovirus-mediated sorcin overexpression on excitation-contraction coupling in isolated rabbit cardiomyocytes.
description
2003 nî lūn-bûn
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2003 թուականի Յունիսին հրատարակուած գիտական յօդուած
@hyw
2003 թվականի հունիսին հրատարակված գիտական հոդված
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2003年の論文
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2003年学术文章
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2003年学术文章
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2003年学术文章
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2003年学术文章
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2003年学术文章
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2003年學術文章
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name
Effects of adenovirus-mediated ...... solated rabbit cardiomyocytes.
@ast
Effects of adenovirus-mediated ...... solated rabbit cardiomyocytes.
@en
type
label
Effects of adenovirus-mediated ...... solated rabbit cardiomyocytes.
@ast
Effects of adenovirus-mediated ...... solated rabbit cardiomyocytes.
@en
prefLabel
Effects of adenovirus-mediated ...... solated rabbit cardiomyocytes.
@ast
Effects of adenovirus-mediated ...... solated rabbit cardiomyocytes.
@en
P2093
P1433
P1476
Effects of adenovirus-mediated ...... solated rabbit cardiomyocytes.
@en
P2093
Annika Burow
Astrid Kania
Christopher M Loughrey
Godfrey L Smith
Harald Kögler
Marian B Meyers
Nils Teucher
Sarah Kettlewell
Stefan Wagner
Stewart L W Miller
P304
P356
10.1161/01.RES.0000081596.90205.E2
P577
2003-06-12T00:00:00Z