Uncoupling protein-2 prevents neuronal death and diminishes brain dysfunction after stroke and brain trauma. - Wikidata - wikimedia - TriplyDB

Uncoupling protein-2 prevents neuronal death and diminishes brain dysfunction after stroke and brain trauma.

Uncoupling protein-2 prevents neuronal death and diminishes brain dysfunction after stroke and brain trauma.

http://www.wikidata.org/entity/Q31149457

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Persistent nuclear factor-kappa B activation in Ucp2-/- mice leads to enhanced nitric oxide and inflammatory cytokine production The neuroprotective properties of calorie restriction, the ketogenic diet, and ketone bodies Role of sirtuins in ischemia-reperfusion injury Mitochondria in traumatic brain injury and mitochondrial-targeted multipotential therapeutic strategies The Timing of Raf/ERK and AKT Activation in Protecting PC12 Cells against Oxidative Stress Amitriptyline improves motor function via enhanced neurotrophin signaling and mitochondrial functions in the murine N171-82Q Huntington disease model.Uncoupling protein 2 in the glial response to stress: implications for neuroprotection Molecular mechanisms of ischemia-reperfusion injury in brain: pivotal role of the mitochondrial membrane potential in reactive oxygen species generation Changes in components of energy regulation in mouse cortex with increases in wakefulness Genipin-induced inhibition of uncoupling protein-2 sensitizes drug-resistant cancer cells to cytotoxic agents Pathway analysis reveals common pro-survival mechanisms of metyrapone and carbenoxolone after traumatic brain injury Resveratrol pretreatment protects rat brain from cerebral ischemic damage via a sirtuin 1-uncoupling protein 2 pathway Heterogeneous nuclear ribonucleoprotein K enhances insulin-induced expression of mitochondrial UCP2 protein UCP2 protects hypothalamic cells from TNF-alpha-induced damage Cyanide-induced death of dopaminergic cells is mediated by uncoupling protein-2 up-regulation and reduced Bcl-2 expression Developmental Sex Differences in the Metabolism of Cardiolipin in Mouse Cerebral Cortex Mitochondria Sirtuins in mammals: insights into their biological function Negative regulation of UCP2 by TGFβ signaling characterizes low and intermediate-grade primary breast cancer.Neurometabolism in human epilepsy Cloning, ontogenesis, and localization of an atypical uncoupling protein 4 in Xenopus laevis.Overexpression of UCP2 protects thalamic neurons following global ischemia in the mouse.Reduced antioxidant capacity and diet-induced atherosclerosis in uncoupling protein-2-deficient mice.Persistent oxidative stress due to absence of uncoupling protein 2 associated with impaired pancreatic beta-cell function.Both ischemic preconditioning and ghrelin administration protect hippocampus from ischemia/reperfusion and upregulate uncoupling protein-2 Dietary curcumin supplementation counteracts reduction in levels of molecules involved in energy homeostasis after brain trauma Perspective: Does brown fat protect against diseases of aging?Cancer stem cell theory and the warburg effect, two sides of the same coin?Mitochondrial Uncoupler Prodrug of 2,4-Dinitrophenol, MP201, Prevents Neuronal Damage and Preserves Vision in Experimental Optic Neuritis Selective neuronal vulnerability to oxidative stress in the brain.ER-mitochondrial calcium flow underlies vulnerability of mechanosensory hair cells to damage.Deletion of UCP2 in iNOS deficient mice reduces the severity of the disease during experimental autoimmune encephalomyelitis Mitochondria contribute to LPS-induced MAPK activation via uncoupling protein UCP2 in macrophages Role of uncoupling protein UCP2 in cell-mediated immunity: how macrophage-mediated insulitis is accelerated in a model of autoimmune diabetes.Mitochondria: the missing link between preconditioning and neuroprotection.Mitochondrial uncoupling protein-2 (UCP2) mediates leptin protection against MPP+ toxicity in neuronal cells.Overexpression of mitochondrial uncoupling protein 2 inhibits inflammatory cytokines and activates cell survival factors after cerebral ischemia.Deletion of mitochondrial uncoupling protein-2 increases ischemic brain damage after transient focal ischemia by altering gene expression patterns and enhancing inflammatory cytokines Sodium butyrate reverses the inhibition of Krebs cycle enzymes induced by amphetamine in the rat brain.Quantification of uncoupling protein 2 reveals its main expression in immune cells and selective up-regulation during T-cell proliferation Analysis of uncoupling protein 2-deficient mice upon anaesthesia and sedation revealed a role for UCP2 in locomotion.

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P2860

Uncoupling protein-2 prevents neuronal death and diminishes brain dysfunction after stroke and brain trauma.

http://www.wikidata.org/entity/Q31149457

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2003 nî lūn-bûn

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2003 թուականի Յուլիսին հրատարակուած գիտական յօդուած

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