Changes in [Na(+)](i), compartmental [Ca(2+)], and NADH with dysfunction after global ischemia in intact hearts.
about
Systematic characterization of the murine mitochondrial proteome using functionally validated cardiac mitochondriaMechanisms of sudden cardiac death: oxidants and metabolismIschaemic conditioning and targeting reperfusion injury: a 30 year voyage of discoveryXanthine oxidoreductase and cardiovascular disease: molecular mechanisms and pathophysiological implications.Mitochondria and arrhythmias.Altered proteome biology of cardiac mitochondria under stress conditions.Ischemia reperfusion dysfunction changes model-estimated kinetics of myofilament interaction due to inotropic drugs in isolated hearts.Circulating levels of cytochrome c after resuscitation from cardiac arrest: a marker of mitochondrial injury and predictor of survival.Reduced mitochondrial Ca2+ loading and improved functional recovery after ischemia-reperfusion injury in old vs. young guinea pig hearts.Stretch-induced increase in cardiac contractility is independent of myocyte Ca2+ while block of stretch channels by streptomycin improves contractility after ischemic stunningReal-Time Fluorescence Measurements of ROS and [Ca2+] in Ischemic / Reperfused Rat Hearts: Detectable Increases Occur only after Mitochondrial Pore Opening and Are Attenuated by Ischemic Preconditioning.Biochemical dysfunction in heart mitochondria exposed to ischaemia and reperfusion.Protection against cardiac injury by small Ca(2+)-sensitive K(+) channels identified in guinea pig cardiac inner mitochondrial membraneMitochondrial function in vivo evaluated by NADH fluorescence: from animal models to human studies.The role of succinate and ROS in reperfusion injury - A critical appraisal.Ionic mechanisms of cardiac cell swelling induced by blocking Na+/K+ pump as revealed by experiments and simulation.Mg(2+) differentially regulates two modes of mitochondrial Ca(2+) uptake in isolated cardiac mitochondria: implications for mitochondrial Ca(2+) sequestration.Oxidative stress: Predictive marker for coronary artery disease.Ischemic and anesthetic preconditioning reduces cytosolic [Ca2+] and improves Ca(2+) responses in intact hearts.Blocking Na(+)/H(+) exchange reduces [Na(+)](i) and [Ca(2+)](i) load after ischemia and improves function in intact hearts.Cardiac preconditioning with 4-h, 17 degrees C ischemia reduces [Ca(2+)](i) load and damage in part via K(ATP) channel opening.Anesthetic preconditioning: triggering role of reactive oxygen and nitrogen species in isolated hearts.Altered NADH and improved function by anesthetic and ischemic preconditioning in guinea pig intact hearts.Cross-bridge kinetics modeled from myoplasmic [Ca2+] and LV pressure at 17 degrees C and after 37 degrees C and 17 degrees C ischemia.Myocardial contractile function during postischemic low-flow reperfusion: critical thresholds of NADH and O2 delivery.Hypothermia augments reactive oxygen species detected in the guinea pig isolated perfused heart.Negative inotropic drugs alter indexes of cytosolic [Ca(2+)]-left ventricular pressure relationships after ischemia.Warm ischemic preconditioning improves mitochondrial redox balance during and after mild hypothermic ischemia in guinea pig isolated hearts.Role of protein kinase C in metabolic regulation of the cardiac Na+ channel.Ischemia-reperfusion injury changes the dynamics of Ca2+-contraction coupling due to inotropic drugs in isolated hearts.Endogenous and Agonist-induced Opening of Mitochondrial Big Versus Small Ca2+-sensitive K+ Channels on Cardiac Cell and Mitochondrial Protection.Identity and function of a cardiac mitochondrial small conductance Ca2+-activated K+ channel splice variant.O2 delivery and redox state are determinants of compartment-specific reactive O2 species in myocardial reperfusion.
P2860
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P2860
Changes in [Na(+)](i), compartmental [Ca(2+)], and NADH with dysfunction after global ischemia in intact hearts.
description
2001 nî lūn-bûn
@nan
2001 թուականի Յունուարին հրատարակուած գիտական յօդուած
@hyw
2001 թվականի հունվարին հրատարակված գիտական հոդված
@hy
2001年の論文
@ja
2001年論文
@yue
2001年論文
@zh-hant
2001年論文
@zh-hk
2001年論文
@zh-mo
2001年論文
@zh-tw
2001年论文
@wuu
name
Changes in [Na(+)](i), compart ...... bal ischemia in intact hearts.
@ast
Changes in [Na(+)](i), compart ...... bal ischemia in intact hearts.
@en
type
label
Changes in [Na(+)](i), compart ...... bal ischemia in intact hearts.
@ast
Changes in [Na(+)](i), compart ...... bal ischemia in intact hearts.
@en
prefLabel
Changes in [Na(+)](i), compart ...... bal ischemia in intact hearts.
@ast
Changes in [Na(+)](i), compart ...... bal ischemia in intact hearts.
@en
P2093
P2860
P1476
Changes in [Na(+)](i), compart ...... bal ischemia in intact hearts.
@en
P2093
P2860
P304
P356
10.1152/AJPHEART.2001.280.1.H280
P577
2001-01-01T00:00:00Z