Prevention of influenza-induced lung injury in mice overexpressing extracellular superoxide dismutase.
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Overexpression of extracellular superoxide dismutase reduces acute radiation induced lung toxicityProtective immunity and safety of a genetically modified influenza virus vaccineN-acetylcysteine synergizes with oseltamivir in protecting mice from lethal influenza infectionParadoxical Roles of Antioxidant Enzymes: Basic Mechanisms and Health ImplicationsA Role for Neutrophils in Viral Respiratory Disease.Progress of small molecular inhibitors in the development of anti-influenza virus agents.Possible roles of proinflammatory and chemoattractive cytokines produced by human fetal membrane cells in the pathology of adverse pregnancy outcomes associated with influenza virus infection.RNA Viruses: ROS-Mediated Cell Death.Mouse Models of Oxidative Stress Indicate a Role for Modulating Healthy Aging.8-nitroguanosine formation in viral pneumonia and its implication for pathogenesis.Methamphetamine reduces human influenza A virus replication.A new mouse model for temporal- and tissue-specific control of extracellular superoxide dismutase.Elevated mitochondrial superoxide disrupts normal T cell development, impairing adaptive immune responses to an influenza challenge.Inhibitory effect of glutathione on oxidative liver injury induced by dengue virus serotype 2 infections in mice.Extracellular superoxide dismutase in macrophages augments bacterial killing by promoting phagocytosis.Viral-mediated inhibition of antioxidant enzymes contributes to the pathogenesis of severe respiratory syncytial virus bronchiolitis.Immune response in a wild bird is predicted by oxidative status, but does not cause oxidative stress.Quercetin 7-rhamnoside reduces porcine epidemic diarrhea virus replication via independent pathway of viral induced reactive oxygen species.Pleiotropic Effects of Levofloxacin, Fluoroquinolone Antibiotics, against Influenza Virus-Induced Lung InjuryPreventive Activity against Influenza (H1N1) Virus by Intranasally Delivered RNA-Hydrolyzing Antibody in Respiratory Epithelial Cells of Mice.Synergistic protection against hyperoxia-induced lung injury by neutrophils blockade and EC-SOD overexpression.Oxygen free radical involvement in acute lung injury induced by H5N1 virus in miceNADPH oxidases: an overview from structure to innate immunity-associated pathologies.Respiratory syncytial virus infection: mechanisms of redox control and novel therapeutic opportunitiesResolution of immune response by recombinant transforming growth factor-beta (rTGF-β) during influenza A virus infectionThe Environmental Pollutant Cadmium Promotes Influenza Virus Replication in MDCK Cells by Altering Their Redox StateThe requirement of reactive oxygen intermediates for lymphocytic choriomeningitis virus binding and growth.Extracellular superoxide dismutase haplotypes are associated with acute lung injury and mortality.Antioxidant treatment ameliorates respiratory syncytial virus-induced disease and lung inflammation.Genome-wide fitness profiling reveals adaptations required by Haemophilus in coinfection with influenza A virus in the murine lung.Neonatal hyperoxia alters the host response to influenza A virus infection in adult mice through multiple pathways.Role of glutathione in immunity and inflammation in the lung.Intracellular redox state as target for anti-influenza therapy: are antioxidants always effective?The Influenza Virus H5N1 Infection Can Induce ROS Production for Viral Replication and Host Cell Death in A549 Cells Modulated by Human Cu/Zn Superoxide Dismutase (SOD1) Overexpression.Respiratory syncytial virus induces oxidative stress by modulating antioxidant enzymes.Respiratory Viral Infections and Subversion of Cellular Antioxidant Defenses.Therapeutic impact of human serum albumin-thioredoxin fusion protein on influenza virus-induced lung injury mice.Modulation of human beta-defensin-1 (hBD-1) in plasmacytoid dendritic cells (PDC), monocytes, and epithelial cells by influenza virus, Herpes simplex virus, and Sendai virus and its possible role in innate immunity.Inflammatory cells as a source of airspace extracellular superoxide dismutase after pulmonary injury.Activation of the aryl hydrocarbon receptor increases pulmonary neutrophilia and diminishes host resistance to influenza A virus.
P2860
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P2860
Prevention of influenza-induced lung injury in mice overexpressing extracellular superoxide dismutase.
description
2001 nî lūn-bûn
@nan
2001 թուականի Յունուարին հրատարակուած գիտական յօդուած
@hyw
2001 թվականի հունվարին հրատարակված գիտական հոդված
@hy
2001年の論文
@ja
2001年論文
@yue
2001年論文
@zh-hant
2001年論文
@zh-hk
2001年論文
@zh-mo
2001年論文
@zh-tw
2001年论文
@wuu
name
Prevention of influenza-induce ...... cellular superoxide dismutase.
@ast
Prevention of influenza-induce ...... cellular superoxide dismutase.
@en
type
label
Prevention of influenza-induce ...... cellular superoxide dismutase.
@ast
Prevention of influenza-induce ...... cellular superoxide dismutase.
@en
prefLabel
Prevention of influenza-induce ...... cellular superoxide dismutase.
@ast
Prevention of influenza-induce ...... cellular superoxide dismutase.
@en
P2093
P2860
P1476
Prevention of influenza-induce ...... cellular superoxide dismutase.
@en
P2093
P2860
P304
P356
10.1152/AJPLUNG.2001.280.1.L69
P577
2001-01-01T00:00:00Z