Golgi complex, endoplasmic reticulum exit sites, and microtubules in skeletal muscle fibers are organized by patterned activity.
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Golgi localization of Syne-1Assembly, organization, and function of the COPII coatCoupling of ER exit to microtubules through direct interaction of COPII with dynactinWho needs microtubules? Myogenic reorganization of MTOC, Golgi complex and ER exit sites persists despite lack of normal microtubule tracksVesicle budding from endoplasmic reticulum is involved in calsequestrin routing to sarcoplasmic reticulum of skeletal musclesMicrotubules that form the stationary lattice of muscle fibers are dynamic and nucleated at Golgi elements.Eukaryotic cells and their cell bodies: Cell Theory revisedLoss of a Clueless-dGRASP complex results in ER stress and blocks Integrin exit from the perinuclear endoplasmic reticulum in Drosophila larval muscle.Fiber type conversion by PGC-1α activates lysosomal and autophagosomal biogenesis in both unaffected and Pompe skeletal muscle.Neuronal activity-dependent membrane traffic at the neuromuscular junction.A new directionality tool for assessing microtubule pattern alterations.The angiotensin II type 1 receptor C-terminal Lys residues interact with tubulin and modulate receptor export traffickingAlpha2B-adrenergic receptor interaction with tubulin controls its transport from the endoplasmic reticulum to the cell surface.VCP-dependent muscle degeneration is linked to defects in a dynamic tubular lysosomal network in vivo.Regulation of α2B-Adrenerigc Receptor Export Trafficking by Specific MotifsNuclear positioning in muscle development and disease.Microtubule binding distinguishes dystrophin from utrophin.Muscle activity and muscle agrin regulate the organization of cytoskeletal proteins and attached acetylcholine receptor (AchR) aggregates in skeletal muscle fibers.A postsynaptic Pi3K-CII dependent signaling controller for presynaptic homeostatic plasticity.NADPH oxidase mediates microtubule alterations and diaphragm dysfunction in dystrophic mice.Independent variability of microtubule perturbations associated with dystrophinopathy.Increased ratio of rapsyn to ACh receptor stabilizes postsynaptic receptors at the mouse neuromuscular synapse.Deletion of the microtubule-associated protein 6 (MAP6) results in skeletal muscle dysfunction
P2860
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P2860
Golgi complex, endoplasmic reticulum exit sites, and microtubules in skeletal muscle fibers are organized by patterned activity.
description
2001 nî lūn-bûn
@nan
2001 թուականի Փետրուարին հրատարակուած գիտական յօդուած
@hyw
2001 թվականի փետրվարին հրատարակված գիտական հոդված
@hy
2001年の論文
@ja
2001年論文
@yue
2001年論文
@zh-hant
2001年論文
@zh-hk
2001年論文
@zh-mo
2001年論文
@zh-tw
2001年论文
@wuu
name
Golgi complex, endoplasmic ret ...... ganized by patterned activity.
@ast
Golgi complex, endoplasmic ret ...... ganized by patterned activity.
@en
type
label
Golgi complex, endoplasmic ret ...... ganized by patterned activity.
@ast
Golgi complex, endoplasmic ret ...... ganized by patterned activity.
@en
prefLabel
Golgi complex, endoplasmic ret ...... ganized by patterned activity.
@ast
Golgi complex, endoplasmic ret ...... ganized by patterned activity.
@en
P2093
P1476
Golgi complex, endoplasmic ret ...... rganized by patterned activity
@en
P2093
P304
P356
10.1523/JNEUROSCI.21-03-00875.2001
P407
P577
2001-02-01T00:00:00Z