Blockade of eosinophil migration and airway hyperresponsiveness by cPLA2-inhibition. - Wikidata - wikimedia - TriplyDB

Blockade of eosinophil migration and airway hyperresponsiveness by cPLA2-inhibition.

Blockade of eosinophil migration and airway hyperresponsiveness by cPLA2-inhibition.

http://www.wikidata.org/entity/Q31929666

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Maternal diet modulates placenta growth and gene expression in a mouse model of diabetic pregnancy The immunosuppressant drug FTY720 inhibits cytosolic phospholipase A2 independently of sphingosine-1-phosphate receptors Beta-escin has potent anti-allergic efficacy and reduces allergic airway inflammation.What targeting eosinophils has taught us about their role in diseases.Oral administration of cytosolic PLA2 inhibitor arachidonyl trifluoromethyl ketone ameliorates cauda equina compression injury in rats The RNA-binding protein HuR stabilizes cytosolic phospholipase A2α mRNA under interleukin-1β treatment in non-small cell lung cancer A549 Cells Multiple roles of phospholipase A2 during lung infection and inflammation.In vitro allergen challenge of peripheral blood induces differential gene expression in mononuclear cells of asthmatic patients: inhibition of cytosolic phospholipase A2alpha overcomes the asthma-associated response.The role of sphingosine-1-phosphate and its receptors in asthma.Chemotaxis of bone marrow derived eosinophils in vivo: a novel method to explore receptor-dependent trafficking in the mouse.Human macrophage ATP7A is localized in the trans-Golgi apparatus, controls intracellular copper levels, and mediates macrophage responses to dermal wounds.Cytosolic phospholipase A(2) in hypoxic pulmonary vasoconstriction.Toll-like receptor 4 signaling regulates cytosolic phospholipase A2 activation and lipid generation in lipopolysaccharide-stimulated macrophages.Platelet-activating factor-induced chemokine gene expression requires NF-kappaB activation and Ca2+/calcineurin signaling pathways. Inhibition by receptor phosphorylation and beta-arrestin recruitment.Mechanism of glutamine inhibition of cytosolic phospholipase a2 (cPLA2 ): Evidence of physical interaction between glutamine-Induced mitogen-activated protein kinase phosphatase-1 and cPLA2.Regulation of adhesion of AML14.3D10 cells by surface clustering of beta2-integrin caused by ERK-independent activation of cPLA2.CD1a presentation of endogenous antigens by group 2 innate lymphoid cells.Adenoviruses-mediated RNA interference targeting cytosolic phospholipase A2α attenuates focal ischemic brain damage in mice.

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P2860

Blockade of eosinophil migration and airway hyperresponsiveness by cPLA2-inhibition.

http://www.wikidata.org/entity/Q31929666

description

2001 nî lūn-bûn

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2001 թուականի Փետրուարին հրատարակուած գիտական յօդուած

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2001 թվականի փետրվարին հրատարակված գիտական հոդված

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2001年の論文

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2001年論文

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2001年論文

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2001年論文

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2001年論文

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2001年論文

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2001年论文

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name

Blockade of eosinophil migration and airway hyperresponsiveness by cPLA2-inhibition.

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Blockade of eosinophil migration and airway hyperresponsiveness by cPLA2-inhibition.

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Blockade of eosinophil migration and airway hyperresponsiveness by cPLA2-inhibition.

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Blockade of eosinophil migration and airway hyperresponsiveness by cPLA2-inhibition.

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prefLabel

Blockade of eosinophil migration and airway hyperresponsiveness by cPLA2-inhibition.

@ast

Blockade of eosinophil migration and airway hyperresponsiveness by cPLA2-inhibition.

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Nature Immunology

P1476

Blockade of eosinophil migration and airway hyperresponsiveness by cPLA2-inhibition.

@en

P2093

A Nonomura

http://www.w3.org/2001/XMLSchema#string

A R Leff

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H Sano

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K Kurashima

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P2860

Lipoxins and novel aspirin-triggered 15-epi-lipoxins (ATL): a jungle of cell-cell interactions or a therapeutic opportunity?

How is the level of free arachidonic acid controlled in mammalian cells?

Eosinophilic Inflammation in Asthma

Lysophosphatidylcholine: a chemotactic factor for human monocytes and its potential role in atherogenesis

Slow- and tight-binding inhibitors of the 85-kDa human phospholipase A2.

Antigen-induced generation of lyso-phospholipids in human airways.

Antibody to VLA-4, but not to L-selectin, protects neuronal M2 muscarinic receptors in antigen-challenged guinea pig airways.

Pretreatment with antibody to eosinophil major basic protein prevents hyperresponsiveness by protecting neuronal M2 muscarinic receptors in antigen-challenged guinea pigs.

New concepts in the pathogenesis of bronchial hyperresponsiveness and asthma.

Platelet activating factor: a biologically active phosphoglyceride.

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145-149

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10.1038/84244

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2

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2

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11175812

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