Reduced dendritic spine density on cerebral cortical pyramidal neurons in schizophrenia - Wikidata - wikimedia - TriplyDB

Reduced dendritic spine density on cerebral cortical pyramidal neurons in schizophrenia

Reduced dendritic spine density on cerebral cortical pyramidal neurons in schizophrenia

http://www.wikidata.org/entity/Q32000373

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GABAergic interneuron origin of schizophrenia pathophysiology.DNA-methyltransferase 1 mRNA is selectively overexpressed in telencephalic GABAergic interneurons of schizophrenia brains Glutamatergic deficits and parvalbumin-containing inhibitory neurons in the prefrontal cortex in schizophrenia Reduced dendritic spine density in auditory cortex of subjects with schizophrenia Colocalization of integrin receptors and reelin in dendritic spine postsynaptic densities of adult nonhuman primate cortex Theories of schizophrenia: a genetic-inflammatory-vascular synthesis Modulation of actin dynamics by Rac1 to target cognitive function Dendritic spine pathology in schizophrenia Anatomy and physiology of the thick-tufted layer 5 pyramidal neuron Deciphering the role of docosahexaenoic acid in brain maturation and pathology with magnetic resonance imaging Brain-specific Crmp2 deletion leads to neuronal development deficits and behavioural impairments in mice Reduced fronto-temporal and limbic connectivity in the 22q11.2 deletion syndrome: vulnerability markers for developing schizophrenia?Fragmentation: loss of global coherence or breakdown of modularity in functional brain architecture?Altered immunoreactivity of complexin protein in prefrontal cortex in severe mental illness Schizophrenia risk from complex variation of complement component 4 Disrupted-in-Schizophrenia 1 (DISC1) regulates spines of the glutamate synapse via Rac1 The schizophrenia susceptibility gene dysbindin regulates dendritic spine dynamics A dual comparative approach: integrating lines of evidence from human evolutionary neuroanatomy and neurodevelopmental disorders Dysconnection in schizophrenia: from abnormal synaptic plasticity to failures of self-monitoring Schizophrenia genes, gene expression, and neuropathology: on the matter of their convergence Synaptic Basis for Cross-modal Plasticity: Enhanced Supragranular Dendritic Spine Density in Anterior Ectosylvian Auditory Cortex of the Early Deaf Cat.Altered glutamate protein co-expression network topology linked to spine loss in the auditory cortex of schizophrenia.Molecular substrates of schizophrenia: homeostatic signaling to connectivity Antipsychotic drugs: comparison in animal models of efficacy, neurotransmitter regulation, and neuroprotection Glutamatergic synaptic dysregulation in schizophrenia: therapeutic implications.Molecular aspects of glutamate dysregulation: implications for schizophrenia and its treatment.Reduction in phencyclidine induced sensorimotor gating deficits in the rat following increased system xc⁻ activity in the medial prefrontal cortex.Modeling psychiatric disorders at the cellular and network levels Chronic administration of the neurotrophic agent cerebrolysin ameliorates the behavioral and morphological changes induced by neonatal ventral hippocampus lesion in a rat model of schizophrenia Review of pathological hallmarks of schizophrenia: comparison of genetic models with patients and nongenetic models Altered volume and hemispheric asymmetry of the superficial cortical layers in the schizophrenia planum temporale.Schizophrenia from a neural circuitry perspective: advancing toward rational pharmacological therapies Why do many psychiatric disorders emerge during adolescence?Age-dependent regulation of synaptic connections by dopamine D2 receptors.Remodeling of axo-spinous synapses in the pathophysiology and treatment of depression.What happens in the brain of schizophrenia patients?: an investigation from the viewpoint of neuropathology.Development of antipsychotic medications with novel mechanisms of action based on computational modeling of hippocampal neuropathology.Dysfunctional and compensatory prefrontal cortical systems, genes and the pathogenesis of schizophrenia.Increased diffusivity in superior temporal gyrus in patients with schizophrenia: a Diffusion Tensor Imaging study Kalirin loss results in cortical morphological alterations

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P2860

Reduced dendritic spine density on cerebral cortical pyramidal neurons in schizophrenia

http://www.wikidata.org/entity/Q32000373

description

1998 nî lūn-bûn

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1998 թուականի Հոկտեմբերին հրատարակուած գիտական յօդուած

@hyw

1998 թվականի հոտեմբերին հրատարակված գիտական հոդված

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1998年の論文

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1998年論文

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1998年論文

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1998年論文

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1998年論文

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1998年論文

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1998年论文

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name

Reduced dendritic spine density on cerebral cortical pyramidal neurons in schizophrenia

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Reduced dendritic spine density on cerebral cortical pyramidal neurons in schizophrenia

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type

label

Reduced dendritic spine density on cerebral cortical pyramidal neurons in schizophrenia

@ast

Reduced dendritic spine density on cerebral cortical pyramidal neurons in schizophrenia

@en

prefLabel

Reduced dendritic spine density on cerebral cortical pyramidal neurons in schizophrenia

@ast

Reduced dendritic spine density on cerebral cortical pyramidal neurons in schizophrenia

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Journal of Neurology, Neurosurgery and Psychiatry

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Reduced dendritic spine density on cerebral cortical pyramidal neurons in schizophrenia

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P2093

Barnes TR

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Davis A

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Garey LJ

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Hirsch SR

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Patel TS

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P2860

Cerebral ventricular size and cognitive impairment in chronic schizophrenia

Reduction of cortical volume in schizophrenia on magnetic resonance imaging.

Patterns of cerebral blood flow in schizophrenia.

Low cerebrospinal fluid glutamate in schizophrenic patients and a new hypothesis on schizophrenia.

Temporal lobe pathology in schizophrenia: a quantitative magnetic resonance imaging study.

Golgi preparations as a tool in neuropathology with particular reference to investigations of the human telencephalic cortex.

Lateral cerebral ventricular enlargement in chronic schizophrenia.

Progressive dendritic changes in aging human cortex.

Glutamate: a neurotransmitter in mammalian brain.

Is schizophrenia due to excessive synaptic pruning in the prefrontal cortex? The Feinberg hypothesis revisited.

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