Cutting edge: the class II transactivator prevents activation-induced cell death by inhibiting Fas ligand gene expression.
about
MHC class II transactivator represses human IL-4 gene transcription by interruption of promoter binding with CBP/p300, STAT6 and NFAT1 via histone hypoacetylationThe RFX family interacts at the collagen (COL1A2) start site and represses transcriptionThe p53-induced gene-6 (proline oxidase) mediates apoptosis through a calcineurin-dependent pathwayThe PYRIN domain in signal transductionIdentification of CIITA regulated genetic module dedicated for antigen presentation.Many checkpoints on the road to cell death: regulation of Fas-FasL interactions and Fas signaling in peripheral immune responses.The NOD: a signaling module that regulates apoptosis and host defense against pathogens.Genome-wide CIITA-binding profile identifies sequence preferences that dictate function versus recruitment.Estrogen deficiency induces bone loss by increasing T cell proliferation and lifespan through IFN-gamma-induced class II transactivator.Failure to upregulate cell surface PD-1 is associated with dysregulated stimulation of T cells by TGN1412-like CD28 superagonist.Overexpression of CIITA in T cells aggravates Th2-mediated colitis in mice.Regulating the activity of class II transactivator by posttranslational modifications: exploring the possibilities.Self-association of class II transactivator correlates with its intracellular localization and transactivation.Kinetics of a gamma interferon response: expression and assembly of CIITA promoter IV and inhibition by methylation.Ganoderma lucidum polysaccharides counteract inhibition on CD71 and FasL expression by culture supernatant of B16F10 cells upon lymphocyte activation.CIITA mediates interferon-gamma repression of collagen transcription through phosphorylation-dependent interactions with co-repressor molecules.Collagen alpha1(I) gene (COL1A1) is repressed by RFX familyClass II major histocompatibility complex transactivator (CIITA) inhibits matrix metalloproteinase-9 gene expression.MHC class II-deficient tumor cell lines with a defective expression of the class II transactivator.Phosphorylation and ubiquitination of degron proximal residues are essential for class II transactivator (CIITA) transactivation and major histocompatibility class II expression.Attenuation of allograft rejection by intragraft inhibition of class II transcativator in high responder rat liver transplantation.Revisiting the specificity of the MHC class II transactivator CIITA in vivo.
P2860
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P2860
Cutting edge: the class II transactivator prevents activation-induced cell death by inhibiting Fas ligand gene expression.
description
2001 nî lūn-bûn
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2001 թուականի Մարտին հրատարակուած գիտական յօդուած
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2001 թվականի մարտին հրատարակված գիտական հոդված
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2001年の論文
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2001年論文
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2001年論文
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2001年論文
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2001年論文
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2001年論文
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2001年论文
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name
Cutting edge: the class II tra ...... ng Fas ligand gene expression.
@ast
Cutting edge: the class II tra ...... ng Fas ligand gene expression.
@en
type
label
Cutting edge: the class II tra ...... ng Fas ligand gene expression.
@ast
Cutting edge: the class II tra ...... ng Fas ligand gene expression.
@en
prefLabel
Cutting edge: the class II tra ...... ng Fas ligand gene expression.
@ast
Cutting edge: the class II tra ...... ng Fas ligand gene expression.
@en
P1476
Cutting edge: the class II tra ...... ng Fas ligand gene expression.
@en
P2093
P304
P356
10.4049/JIMMUNOL.166.5.2917
P407
P577
2001-03-01T00:00:00Z