Amphetamine selectively blocks inhibitory glutamate transmission in dopamine neurons.
about
Glutamatergic substrates of drug addiction and alcoholismIllicit dopamine transients: reconciling actions of abused drugsNucleus accumbens shell excitability is decreased by methamphetamine self-administration and increased by 5-HT2C receptor inverse agonism and agonism.Cocaine increases dopaminergic neuron and motor activity via midbrain α1 adrenergic signaling.Ethanol action on dopaminergic neurons in the ventral tegmental area: interaction with intrinsic ion channels and neurotransmitter inputs.Amphetamine elicits opposing actions on readily releasable and reserve pools for dopamineMice with chronic norepinephrine deficiency resemble amphetamine-sensitized animalsAn intrinsic neuronal oscillator underlies dopaminergic neuron burstingDistinct forms of Gq-receptor-dependent plasticity of excitatory transmission in the BNST are differentially affected by stress.Alpha-2 noradrenergic receptor activation inhibits the hyperpolarization-activated cation current (Ih) in neurons of the ventral tegmental area.Reduced noradrenergic innervation of ventral midbrain dopaminergic cell groups and the subthalamic nucleus in MPTP-treated parkinsonian monkeys.Dynamic regulation of midbrain dopamine neuron activity: intrinsic, synaptic, and plasticity mechanismsOrexin/hypocretin modulates response of ventral tegmental dopamine neurons to prefrontal activation: diurnal influences.Two intracellular pathways mediate metabotropic glutamate receptor-induced Ca2+ mobilization in dopamine neurons.Spontaneous opening of T-type Ca2+ channels contributes to the irregular firing of dopamine neurons in neonatal rats.There and back again: a tale of norepinephrine and drug addiction.Functional reduction of SK3-mediated currents precedes AMPA-receptor-mediated excitotoxicity in dopaminergic neurons.Heterogeneity of dopamine neuron activity across traits and states.Role of serotonin in cocaine effects in mice with reduced dopamine transporter function.Research Review: altered reward function in adolescent depression: what, when and how?Differential regulation of action potential- and metabotropic glutamate receptor-induced Ca2+ signals by inositol 1,4,5-trisphosphate in dopaminergic neurons.α-1 Adrenergic receptors are localized on presynaptic elements in the nucleus accumbens and regulate mesolimbic dopamine transmission.On the effects of psychostimulants, antidepressants, and the antiparkinsonian drug levodopa on dopamine neurons.Independent presynaptic and postsynaptic mechanisms regulate endocannabinoid signaling at multiple synapses in the ventral tegmental area.Specificity and impact of adrenergic projections to the midbrain dopamine system.Amphetamine paradoxically augments exocytotic dopamine release and phasic dopamine signals.Light and electron microscopic localization of alpha-1 adrenergic receptor immunoreactivity in the rat striatum and ventral midbrainCalcium Release from Stores Inhibits GIRKMechanisms underlying the onset and expression of levodopa-induced dyskinesia and their pharmacological manipulation.The effects of locus coeruleus and norepinephrine in methamphetamine toxicity.Norepinephrine regulates cocaine-primed reinstatement via α1-adrenergic receptors in the medial prefrontal cortex.High doses of amphetamine augment, rather than disrupt, exocytotic dopamine release in the dorsal and ventral striatum of the anesthetized rat.Computational model predicts a role for ERG current in repolarizing plateau potentials in dopamine neurons: implications for modulation of neuronal activity.CRF facilitates calcium release from intracellular stores in midbrain dopamine neurons.Social isolation rearing increases nucleus accumbens dopamine and norepinephrine responses to acute ethanol in adulthood.Noradrenergic modulation of the hyperpolarization-activated cation current (Ih) in dopamine neurons of the ventral tegmental area.Adrenergic and noradrenergic innervation of the midbrain ventral tegmental area and retrorubral field: prominent inputs from medullary homeostatic centers.Alpha1-adrenergic receptor-induced heterosynaptic long-term depression in the bed nucleus of the stria terminalis is disrupted in mouse models of affective disorders.An increase in AMPA and a decrease in SK conductance increase burst firing by different mechanisms in a model of a dopamine neuron in vivo.Repeated social defeat stress enhances glutamatergic synaptic plasticity in the VTA and cocaine place conditioning.
P2860
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P2860
Amphetamine selectively blocks inhibitory glutamate transmission in dopamine neurons.
description
2001 nî lūn-bûn
@nan
2001 թուականի Մարտին հրատարակուած գիտական յօդուած
@hyw
2001 թվականի մարտին հրատարակված գիտական հոդված
@hy
2001年の論文
@ja
2001年論文
@yue
2001年論文
@zh-hant
2001年論文
@zh-hk
2001年論文
@zh-mo
2001年論文
@zh-tw
2001年论文
@wuu
name
Amphetamine selectively blocks inhibitory glutamate transmission in dopamine neurons.
@ast
Amphetamine selectively blocks inhibitory glutamate transmission in dopamine neurons.
@en
type
label
Amphetamine selectively blocks inhibitory glutamate transmission in dopamine neurons.
@ast
Amphetamine selectively blocks inhibitory glutamate transmission in dopamine neurons.
@en
prefLabel
Amphetamine selectively blocks inhibitory glutamate transmission in dopamine neurons.
@ast
Amphetamine selectively blocks inhibitory glutamate transmission in dopamine neurons.
@en
P2093
P356
P1433
P1476
Amphetamine selectively blocks inhibitory glutamate transmission in dopamine neurons.
@en
P2093
Fiorillo CD
Morikawa H
Paladini CA
Williams JT
P2888
P304
P356
10.1038/85124
P407
P577
2001-03-01T00:00:00Z
P6179
1002043262