Hyper-responsivity in a subset of C-fiber nociceptors in a model of painful diabetic neuropathy in the rat.
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Diabetic neuropathic pain: Physiopathology and treatmentAbnormal muscle afferent function in a model of Taxol chemotherapy-induced painful neuropathyOlesoxime (TRO19622): A Novel Mitochondrial-Targeted Neuroprotective Compound.Excitability of Aβ sensory neurons is altered in an animal model of peripheral neuropathyGastrodin inhibits allodynia and hyperalgesia in painful diabetic neuropathy rats by decreasing excitability of nociceptive primary sensory neurons.Impaired sensory nerve function and axon morphology in mice with diabetic neuropathy.Mechanically-evoked C-fiber activity in painful alcohol and AIDS therapy neuropathy in the ratMarked attenuation of inflammatory mediator-induced C-fiber sensitization for mechanical and hypotonic stimuli in TRPV4-/- mice.GDNF hyperalgesia is mediated by PLCgamma, MAPK/ERK, PI3K, CDK5 and Src family kinase signaling and dependent on the IB4-binding protein versican.Characterization of upper thoracic spinal neurons receiving noxious cardiac and/or somatic inputs in diabetic rats.Blockade of sigma 1 receptors alleviates sensory signs of diabetic neuropathy in ratsRole of Fyn-mediated NMDA receptor function in prediabetic neuropathy in mice.Cutaneous nociceptors lack sensitisation, but reveal μ-opioid receptor-mediated reduction in excitability to mechanical stimulation in neuropathy.Involvement of spinal glutamate transporter-1 in the development of mechanical allodynia and hyperalgesia associated with type 2 diabetes.Altered synaptic input and GABAB receptor function in spinal superficial dorsal horn neurons in rats with diabetic neuropathy.Characterization of upper thoracic spinal neurons responding to esophageal distension in diabetic rats.Early painful diabetic neuropathy is associated with differential changes in the expression and function of vanilloid receptor 1.Taurine replacement attenuates hyperalgesia and abnormal calcium signaling in sensory neurons of STZ-D rats.Forced-exercise delays neuropathic pain in experimental diabetes: effects on voltage-activated calcium channels.Α-Dendrotoxin-sensitive Kv1 channels contribute to conduction failure of polymodal nociceptive C-fibers from rat coccygeal nerve.Accumulation of Cav3.2 T-type Calcium Channels in the Uninjured Sural Nerve Contributes to Neuropathic Pain in Rats with Spared Nerve Injury.
P2860
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P2860
Hyper-responsivity in a subset of C-fiber nociceptors in a model of painful diabetic neuropathy in the rat.
description
2001 nî lūn-bûn
@nan
2001 թուականի Յունուարին հրատարակուած գիտական յօդուած
@hyw
2001 թվականի հունվարին հրատարակված գիտական հոդված
@hy
2001年の論文
@ja
2001年論文
@yue
2001年論文
@zh-hant
2001年論文
@zh-hk
2001年論文
@zh-mo
2001年論文
@zh-tw
2001年论文
@wuu
name
Hyper-responsivity in a subset ...... iabetic neuropathy in the rat.
@ast
Hyper-responsivity in a subset ...... iabetic neuropathy in the rat.
@en
type
label
Hyper-responsivity in a subset ...... iabetic neuropathy in the rat.
@ast
Hyper-responsivity in a subset ...... iabetic neuropathy in the rat.
@en
prefLabel
Hyper-responsivity in a subset ...... iabetic neuropathy in the rat.
@ast
Hyper-responsivity in a subset ...... iabetic neuropathy in the rat.
@en
P1433
P1476
Hyper-responsivity in a subset ...... iabetic neuropathy in the rat.
@en
P2093
P304
P356
10.1016/S0306-4522(00)00454-1
P407
P577
2001-01-01T00:00:00Z