Factors larger than 100 kd in post-hemorrhagic shock mesenteric lymph are toxic for endothelial cells.
about
The digestive tract as the origin of systemic inflammationEnrichment of the lung microbiome with gut bacteria in sepsis and the acute respiratory distress syndromeThe intestinal microbiome and surgical disease.Trauma hemorrhagic shock-induced lung injury involves a gut-lymph-induced TLR4 pathway in miceGastrointestinal lymphatics in health and diseaseHeparin use in a rat hemorrhagic shock model induces biologic activity in mesenteric lymph separate from shockActivation of toll-like receptor 4 is necessary for trauma hemorrhagic shock-induced gut injury and polymorphonuclear neutrophil primingIntestinal crosstalk: a new paradigm for understanding the gut as the "motor" of critical illness.Effects of intestinal lymph on expression of neutrophil adhesion factors and lung injury after trauma-induced shock.Role of lipase-generated free fatty acids in converting mesenteric lymph from a noncytotoxic to a cytotoxic fluid.Impact of intestinal ischemia/reperfusion and lymph drainage on distant organs in ratsIntroduction of an agent-based multi-scale modular architecture for dynamic knowledge representation of acute inflammationRole of gut-lymph factors in the induction of burn-induced and trauma-shock-induced acute heart failure.A journey with Tony Hugli up the inflammatory cascade towards the auto-digestion hypothesisMesenteric lymph duct ligation prevents trauma/hemorrhage shock-induced cardiac contractile dysfunction.Trauma-hemorrhagic shock-induced pulmonary epithelial and endothelial cell injury utilizes different programmed cell death signaling pathways.Regional susceptibility to stress-induced intestinal injury in the mouse.A sphingosine-1 phosphate agonist (FTY720) limits trauma/hemorrhagic shock-induced multiple organ dysfunction syndrome.Cross-transfusion of postshock mesenteric lymph provokes acute lung injury2008 Landis Award lecture. Inflammation and the autodigestion hypothesis.Trauma-hemorrhagic shock mesenteric lymph induces endothelial apoptosis that involves both caspase-dependent and caspase-independent mechanisms.Bacterial translocation in an experimental intestinal obstruction model. C-reactive protein reliability?The proteome of rodent mesenteric lymph.Albumin protects against gut-induced lung injury in vitro and in vivo.Biomechanical aspects of the auto-digestion theory.
P2860
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P2860
Factors larger than 100 kd in post-hemorrhagic shock mesenteric lymph are toxic for endothelial cells.
description
2001 nî lūn-bûn
@nan
2001 թուականի Մարտին հրատարակուած գիտական յօդուած
@hyw
2001 թվականի մարտին հրատարակված գիտական հոդված
@hy
2001年の論文
@ja
2001年論文
@yue
2001年論文
@zh-hant
2001年論文
@zh-hk
2001年論文
@zh-mo
2001年論文
@zh-tw
2001年论文
@wuu
name
Factors larger than 100 kd in ...... e toxic for endothelial cells.
@ast
Factors larger than 100 kd in ...... e toxic for endothelial cells.
@en
type
label
Factors larger than 100 kd in ...... e toxic for endothelial cells.
@ast
Factors larger than 100 kd in ...... e toxic for endothelial cells.
@en
prefLabel
Factors larger than 100 kd in ...... e toxic for endothelial cells.
@ast
Factors larger than 100 kd in ...... e toxic for endothelial cells.
@en
P2093
P356
P1433
P1476
Factors larger than 100 kd in ...... e toxic for endothelial cells.
@en
P2093
P304
P356
10.1067/MSY.2001.111698
P407
P577
2001-03-01T00:00:00Z