Increased expression of placenta growth factor in proliferative diabetic retinopathy.
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Placental growth factor contributes to micro-vascular abnormalization and blood-retinal barrier breakdown in diabetic retinopathySustained inflammation after pericyte depletion induces irreversible blood-retina barrier breakdownImmunolocalisation of the VEGF receptors FLT-1, KDR, and FLT-4 in diabetic retinopathy.Alterations in the intraocular cytokine milieu after intravitreal bevacizumab.Placenta growth factor-1 exerts time-dependent stabilization of adherens junctions following VEGF-induced vascular permeability.An anti-angiogenic state in mice and humans with retinal photoreceptor cell degeneration.Genetic and environmental factors influencing the Placental Growth Factor (PGF) variation in two populations.Prevention of ischemia-induced retinopathy by the natural ocular antiangiogenic agent pigment epithelium-derived factor.Deletion of placental growth factor prevents diabetic retinopathy and is associated with Akt activation and HIF1α-VEGF pathway inhibition.Vascular endothelial growth factor C promotes survival of retinal vascular endothelial cells via vascular endothelial growth factor receptor-2.Placental growth factor and its potential role in diabetic retinopathy and other ocular neovascular diseases.A chronic grey matter penumbra, lateral microvascular intussusception and venous peduncular avulsion underlie diabetic vitreous haemorrhageActivation of the Wnt pathway plays a pathogenic role in diabetic retinopathy in humans and animal models.PlGF: a multitasking cytokine with disease-restricted activity.Diabetic Retinopathy: Vascular and Inflammatory Disease.Central Role of Metabolism in Endothelial Cell Function and Vascular Disease.Ranibizumab efficiently blocks migration but not proliferation induced by growth factor combinations including VEGF in retinal endothelial cells.Diabetic retinopathy: current understanding, mechanisms, and treatment strategiesHypoxia increases placenta growth factor expression in human myocardium and cultured neonatal rat cardiomyocytes.Binding of VEGF-A is sufficient to abrogate the disturbing effects of VEGF-B together with VEGF-A on retinal endothelial cells.Aflibercept versus ranibizumab for treating persistent diabetic macular oedema.Kaempferol inhibited VEGF and PGF expression and in vitro angiogenesis of HRECs under diabetic-like environment.Vitreous levels of placental growth factor correlate with activity of proliferative diabetic retinopathy and are not influenced by bevacizumab treatment.Anti-inflammatory cytokine and angiogenic factors levels in vitreous samples of diabetic retinopathy patients.The Novel Pathogenesis of Retinopathy Mediated by Multiple RTK Signals is Uncovered in Newly Developed Mouse Model.
P2860
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P2860
Increased expression of placenta growth factor in proliferative diabetic retinopathy.
description
1998 nî lūn-bûn
@nan
1998 թուականի Յունուարին հրատարակուած գիտական յօդուած
@hyw
1998 թվականի հունվարին հրատարակված գիտական հոդված
@hy
1998年の論文
@ja
1998年論文
@yue
1998年論文
@zh-hant
1998年論文
@zh-hk
1998年論文
@zh-mo
1998年論文
@zh-tw
1998年论文
@wuu
name
Increased expression of placenta growth factor in proliferative diabetic retinopathy.
@ast
Increased expression of placenta growth factor in proliferative diabetic retinopathy.
@en
type
label
Increased expression of placenta growth factor in proliferative diabetic retinopathy.
@ast
Increased expression of placenta growth factor in proliferative diabetic retinopathy.
@en
prefLabel
Increased expression of placenta growth factor in proliferative diabetic retinopathy.
@ast
Increased expression of placenta growth factor in proliferative diabetic retinopathy.
@en
P2093
P1476
Increased expression of placenta growth factor in proliferative diabetic retinopathy
@en
P2093
P304
P577
1998-01-01T00:00:00Z