Mechanisms of orthostatic intolerance following very prolonged exercise. - Wikidata - wikimedia - TriplyDB

Mechanisms of orthostatic intolerance following very prolonged exercise.

Mechanisms of orthostatic intolerance following very prolonged exercise.

http://www.wikidata.org/entity/Q33156281

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Music Attenuated a Decrease in Parasympathetic Nervous System Activity after Exercise.Initial orthostatic hypotension is unrelated to orthostatic tolerance in healthy young subjects.Influence of age on syncope following prolonged exercise: differential responses but similar orthostatic intolerance.Diurnal variation in time to presyncope and associated circulatory changes during a controlled orthostatic challenge.Syncope is unrelated to supine and postural hypotension following prolonged exercise.Postexercise orthostatic intolerance: influence of exercise intensity.Influence of regular exercise training on post-exercise hemodynamic regulation to orthostatic challenge.Crawling to the finish line: why do endurance runners collapse? Implications for understanding of mechanisms underlying pacing and fatigue.Slow breathing as a means to improve orthostatic tolerance: a randomized sham-controlled trial.

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Mechanisms of orthostatic intolerance following very prolonged exercise.

http://www.wikidata.org/entity/Q33156281

description

2008 nî lūn-bûn

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2008 թուականի Մայիսին հրատարակուած գիտական յօդուած

@hyw

2008 թվականի մայիսին հրատարակված գիտական հոդված

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2008年の論文

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2008年論文

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2008年論文

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2008年論文

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2008年論文

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2008年論文

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2008年论文

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name

Mechanisms of orthostatic intolerance following very prolonged exercise.

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Mechanisms of orthostatic intolerance following very prolonged exercise.

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type

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Mechanisms of orthostatic intolerance following very prolonged exercise.

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Mechanisms of orthostatic intolerance following very prolonged exercise.

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Mechanisms of orthostatic intolerance following very prolonged exercise.

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Mechanisms of orthostatic intolerance following very prolonged exercise.

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JAPPLPHYSIOL.00175.2008

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Journal of Applied Physiology

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Mechanisms of orthostatic intolerance following very prolonged exercise

@en

P2093

Carissa Murrell

http://www.w3.org/2001/XMLSchema#string

David Gaze

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James D Cotter

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Keith P George

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Philip N Ainslie

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P2860

Middle cerebral artery blood velocity is reduced with hyperthermia during prolonged exercise in humans

Heart rate variability: standards of measurement, physiological interpretation and clinical use. Task Force of the European Society of Cardiology and the North American Society of Pacing and Electrophysiology

Decrease in cardiac output and muscle sympathetic activity during vasovagal syncope.

Management of vasovagal syncope: controlling or aborting faints by leg crossing and muscle tensing.

Syncope, cerebral perfusion, and oxygenation.

Leg crossing, muscle tensing, squatting, and the crash position are effective against vasovagal reactions solely through increases in cardiac output.

The venous system is the main determinant of hypotension in patients with vasovagal syncope.

Abnormal forearm vascular responses during dynamic leg exercise in patients with vasovagal syncope.

Impaired immediate vasoconstrictor responses in patients with recurrent neurally mediated syncope.

Failure of reflex venoconstriction during exercise in patients with vasovagal syncope.

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213-225

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scholarly article

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10.1152/JAPPLPHYSIOL.00175.2008

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1

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105

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Samuel J.E. Lucas

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2008-05-15T00:00:00Z

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5368846

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18483169

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