Attenuation of antigen-induced airway hyperresponsiveness in CGRP-deficient mice. - Wikidata - wikimedia - TriplyDB

Attenuation of antigen-induced airway hyperresponsiveness in CGRP-deficient mice.

Attenuation of antigen-induced airway hyperresponsiveness in CGRP-deficient mice.

http://www.wikidata.org/entity/Q33184839

about

The GPCR modulator protein RAMP2 is essential for angiogenesis and vascular integrity Sensory detection and responses to toxic gases: mechanisms, health effects, and countermeasures.Calcitonin gene-related peptide: physiology and pathophysiology Deficiency of RAMP1 attenuates antigen-induced airway hyperresponsiveness in mice Calcitonin gene-related peptide stimulates proliferation of alveolar epithelial cells.The neuropeptide neuromedin U activates eosinophils and is involved in allergen-induced eosinophilia.Greater involvement of neurokinins found in Guinea pig models of severe asthma compared with mild asthma.Neuro-immune interactions in allergic diseases: novel targets for therapeutics.Adrenomedullin insufficiency increases allergen-induced airway hyperresponsiveness in mice.Calcitonin gene-related peptide mediates acid-induced lung injury in mice.Airway expression of calcitonin gene-related peptide in T-cell peptide-induced late asthmatic reactions in atopics.

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P2860

Attenuation of antigen-induced airway hyperresponsiveness in CGRP-deficient mice.

http://www.wikidata.org/entity/Q33184839

description

2002 nî lūn-bûn

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2002 թուականի Նոյեմբերին հրատարակուած գիտական յօդուած

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2002 թվականի նոյեմբերին հրատարակված գիտական հոդված

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2002年の論文

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2002年論文

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2002年論文

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2002年論文

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2002年論文

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2002年論文

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2002年论文

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name

Attenuation of antigen-induced airway hyperresponsiveness in CGRP-deficient mice.

@ast

Attenuation of antigen-induced airway hyperresponsiveness in CGRP-deficient mice.

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type

label

Attenuation of antigen-induced airway hyperresponsiveness in CGRP-deficient mice.

@ast

Attenuation of antigen-induced airway hyperresponsiveness in CGRP-deficient mice.

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prefLabel

Attenuation of antigen-induced airway hyperresponsiveness in CGRP-deficient mice.

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Attenuation of antigen-induced airway hyperresponsiveness in CGRP-deficient mice.

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AJPLUNG.00130.2002

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American Journal of Physiology - Lung Cellular and Molecular Physiology

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Attenuation of antigen-induced airway hyperresponsiveness in CGRP-deficient mice.

@en

P2093

Eijiro Ohga

http://www.w3.org/2001/XMLSchema#string

Hiroki Kurihara

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Hiroshi Yamamoto

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Ryozo Nagai

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Takahide Nagase

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Takayuki Shindo

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Tetsuji Tomita

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Tomoko Aoki-Nagase

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Yasuhiro Yamaguchi

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Yasuyoshi Ouchi

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P2860

RAMPs regulate the transport and ligand specificity of the calcitonin-receptor-like receptor

Alternative RNA processing in calcitonin gene expression generates mRNAs encoding different polypeptide products

Production of a novel neuropeptide encoded by the calcitonin gene via tissue-specific RNA processing

Calcitonin gene-related peptide is a potent vasodilator

Elevated sympathetic nervous activity in mice deficient in alphaCGRP

Expression of the cysteinyl leukotriene 1 receptor in normal human lung and peripheral blood leukocytes.

Bronchial hyperreactivity, increased endotoxin lethality and melanocytic tumorigenesis in transgenic mice overexpressing platelet-activating factor receptor.

Effect of a 5-lipoxygenase inhibitor and leukotriene antagonist (PF 5901) on antigen-induced airway responses in neonatally immunized rabbits.

E-type prostaglandins enhance local oedema formation and neutrophil accumulation but suppress eosinophil accumulation in guinea-pig skin.

Impaired anaphylactic responses with intact sensitivity to endotoxin in mice lacking a platelet-activating factor receptor.

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