Leptin deficiency produces contrasting phenotypes in bones of the limb and spine.
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Central leptin gene therapy corrects skeletal abnormalities in leptin-deficient ob/ob miceBone Marrow Adipose Tissue: To Be or Not To Be a Typical Adipose Tissue?The alliance of mesenchymal stem cells, bone, and diabetesAnorexia Nervosa and Its Associated Endocrinopathy in Young PeopleAdipokines as potential prognostic biomarkers in patients with acute knee injuryLipid metabolism disorders and bone dysfunction--interrelated and mutually regulated (review)Evidence for pleiotropic factors in genetics of the musculoskeletal systemGenetics of the musculoskeletal system: a pleiotropic approachPathogenesis of adolescent idiopathic scoliosis in girls - a double neuro-osseous theory involving disharmony between two nervous systems, somatic and autonomic expressed in the spine and trunk: possible dependency on sympathetic nervous system andTargeted disruption of the Lasp-1 gene is linked to increases in histamine-stimulated gastric HCl secretionThe effects of graded levels of calorie restriction: I. impact of short term calorie and protein restriction on body composition in the C57BL/6 mouse.Morbid obesity attenuates the skeletal abnormalities associated with leptin deficiency in miceAn age-dependent interaction with leptin unmasks ghrelin's bone-protective effectsPreadipocyte factor-1 is associated with marrow adiposity and bone mineral density in women with anorexia nervosaHormone predictors of abnormal bone microarchitecture in women with anorexia nervosaExtreme obesity due to impaired leptin signaling in mice does not cause knee osteoarthritis.Anorexia nervosa and bone.Serum levels of leptin and high molecular weight adiponectin are inversely associated with radiographic spinal progression in patients with ankylosing spondylitis: results from the ENRADAS trialImpaired fracture healing with high non-union rates remains irreversible after traumatic brain injury in leptin-deficient mice.Short-term re-feeding of previously energy-restricted C57BL/6 male mice restores body weight and body fat and attenuates the decline in natural killer cell function after primary influenza infectionBone mineral content in patients with congenital generalized lipodystrophy is unaffected by metreleptin replacement therapy.Leptin functions peripherally to regulate differentiation of mesenchymal progenitor cells.Moderate weight gain does not influence bone metabolism in skeletally mature female rats.Caloric restriction leads to high marrow adiposity and low bone mass in growing miceRecent progress in histochemistry and cell biology.Leptin receptor (Lepr) is a negative modulator of bone mechanosensitivity and genetic variations in Lepr may contribute to the differential osteogenic response to mechanical stimulation in the C57BL/6J and C3H/HeJ pair of mouse strains.Expansion of Bone Marrow Adipose Tissue During Caloric Restriction Is Associated With Increased Circulating Glucocorticoids and Not With Hypoleptinemia.Skeletal aging and the adipocyte program: New insights from an "old" molecule.Diet and gene interactions influence the skeletal response to polyunsaturated fatty acidsChronic central administration of Ghrelin increases bone mass through a mechanism independent of appetite regulation.Leptin's metabolic and immune functions can be uncoupled at the ligand/receptor interaction levelAbsence of functional leptin receptor isoforms in the POUND (Lepr(db/lb)) mouse is associated with muscle atrophy and altered myoblast proliferation and differentiation.Leptin may play a role in bone microstructural alterations in obese children.Hypothalamic leptin gene therapy prevents weight gain without long-term detrimental effects on bone in growing and skeletally mature female rats.Role of WNT16 in the regulation of periosteal bone formation in female mice.Are volumetric bone mineral density and bone micro-architecture associated with leptin and soluble leptin receptor levels in adolescent idiopathic scoliosis?--A case-control studyEffects of obesity on bone metabolism.Effects of resveratrol treatment on bone and cartilage in obese diabetic miceWhy does starvation make bones fat?The aromatic amino acid tryptophan stimulates skeletal muscle IGF1/p70s6k/mTor signaling in vivo and the expression of myogenic genes in vitro.
P2860
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P2860
Leptin deficiency produces contrasting phenotypes in bones of the limb and spine.
description
2004 nî lūn-bûn
@nan
2004 թուականի Մարտին հրատարակուած գիտական յօդուած
@hyw
2004 թվականի մարտին հրատարակված գիտական հոդված
@hy
2004年の論文
@ja
2004年論文
@yue
2004年論文
@zh-hant
2004年論文
@zh-hk
2004年論文
@zh-mo
2004年論文
@zh-tw
2004年论文
@wuu
name
Leptin deficiency produces contrasting phenotypes in bones of the limb and spine.
@ast
Leptin deficiency produces contrasting phenotypes in bones of the limb and spine.
@en
type
label
Leptin deficiency produces contrasting phenotypes in bones of the limb and spine.
@ast
Leptin deficiency produces contrasting phenotypes in bones of the limb and spine.
@en
prefLabel
Leptin deficiency produces contrasting phenotypes in bones of the limb and spine.
@ast
Leptin deficiency produces contrasting phenotypes in bones of the limb and spine.
@en
P2093
P1433
P1476
Leptin deficiency produces contrasting phenotypes in bones of the limb and spine.
@en
P2093
P304
P356
10.1016/J.BONE.2003.11.020
P577
2004-03-01T00:00:00Z