Ectopic expression of epidermal growth factor receptors induces hormone independence in ZR-75-1 human breast cancer cells.
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Breast cancer oestrogen independence mediated by BCAR1 or BCAR3 genes is transmitted through mechanisms distinct from the oestrogen receptor signalling pathway or the epidermal growth factor receptor signalling pathwayIdentification of BCAR3 by a random search for genes involved in antiestrogen resistance of human breast cancer cellsAND-34/BCAR3 differs from other NSP homologs in induction of anti-estrogen resistance, cyclin D1 promoter activation and altered breast cancer cell morphologyEpidermal growth factor receptor signalling in human breast cancer cells operates parallel to estrogen receptor α signalling and results in tamoxifen insensitive proliferationRelevance of BCAR4 in tamoxifen resistance and tumour aggressiveness of human breast cancer.Differential expression of estrogen, progesterone, and epidermal growth factor receptors in normal, benign, and malignant human breast tissues using dual staining immunohistochemistry.Tyrosine dephosphorylation enhances the therapeutic target activity of epidermal growth factor receptor (EGFR) by disrupting its interaction with estrogen receptor (ER).cDNA transfection followed by the isolation of a MCF-7 breast cell line resistant to tamoxifen in vitro and in vivo.Expression of aromatase and estrogen receptor alpha in chondrosarcoma, but no beneficial effect of inhibiting estrogen signaling both in vitro and in vivo.Cyclin E overexpression, a negative prognostic factor in breast cancer with strong correlation to oestrogen receptor status.BCAR4 induces antioestrogen resistance but sensitises breast cancer to lapatinibModels and Mechanisms of Acquired Antihormone Resistance in Breast Cancer: Significant Clinical Progress Despite Limitations.Additive antitumour effect of the epidermal growth factor receptor tyrosine kinase inhibitor gefitinib (Iressa, ZD1839) and the antioestrogen fulvestrant (Faslodex, ICI 182,780) in breast cancer cellsGenome-wide analysis of BP1 transcriptional targets in breast cancer cell line Hs578T.Salinomycin co-treatment enhances tamoxifen cytotoxicity in luminal A breast tumor cells by facilitating lysosomal degradation of receptor tyrosine kinases.
P2860
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P2860
Ectopic expression of epidermal growth factor receptors induces hormone independence in ZR-75-1 human breast cancer cells.
description
1992 nî lūn-bûn
@nan
1992 թուականի Սեպտեմբերին հրատարակուած գիտական յօդուած
@hyw
1992 թվականի սեպտեմբերին հրատարակված գիտական հոդված
@hy
1992年の論文
@ja
1992年論文
@yue
1992年論文
@zh-hant
1992年論文
@zh-hk
1992年論文
@zh-mo
1992年論文
@zh-tw
1992年论文
@wuu
name
Ectopic expression of epiderma ...... 5-1 human breast cancer cells.
@ast
Ectopic expression of epiderma ...... 5-1 human breast cancer cells.
@en
type
label
Ectopic expression of epiderma ...... 5-1 human breast cancer cells.
@ast
Ectopic expression of epiderma ...... 5-1 human breast cancer cells.
@en
prefLabel
Ectopic expression of epiderma ...... 5-1 human breast cancer cells.
@ast
Ectopic expression of epiderma ...... 5-1 human breast cancer cells.
@en
P2093
P1433
P1476
Ectopic expression of epiderma ...... 5-1 human breast cancer cells.
@en
P2093
Dorssers LC
Foekens JA
Portengen H
van Agthoven T
van Agthoven TL
P304
P407
P577
1992-09-01T00:00:00Z