Good timing in the cell cycle for precise DNA repair by BRCA1.
about
Mammalian SUMO E3-ligases PIAS1 and PIAS4 promote responses to DNA double-strand breaksBRCA1: cell cycle checkpoint, genetic instability, DNA damage response and cancer evolutionMre11 and Ku regulation of double-strand break repair by gene conversion and break-induced replication.Oligo/polynucleotide-based gene modification: strategies and therapeutic potentialCohesin Is limiting for the suppression of DNA damage-induced recombination between homologous chromosomesSharp-1 modulates the cellular response to DNA damage.Translational research in the Gynecologic Oncology Group: evaluation of ovarian cancer markers, profiles, and novel therapies.Ligand-dependent differences in estrogen receptor beta-interacting proteins identified in lung adenocarcinoma cells corresponds to estrogenic responses.Oncogenic RAS regulates BRIP1 expression to induce dissociation of BRCA1 from chromatin, inhibit DNA repair, and promote senescence.Tumour suppressor genes in chemotherapeutic drug response.Photon, light ion, and heavy ion cancer radiotherapy: paths from physics and biology to clinical practice.BRCA1-Ku80 protein interaction enhances end-joining fidelity of chromosomal double-strand breaks in the G1 phase of the cell cycleBRCA1 in the DNA damage response and at telomeresFocus on histone variant H2AX: to be or not to be.Unsolved mystery: the role of BRCA1 in DNA end-joining.The topography of mutational processes in breast cancer genomes.BRCA1 expression, proliferative and apoptotic activities in ovarian epithelial inclusions.Telomerase-independent paths to immortality in predictable cancer subtypes.Current and emerging strategies to increase the efficacy of ionizing radiation in the treatment of cancer.Replication independent DNA double-strand break retention may prevent genomic instabilityProtein immunolocalization supports the presence of identical mechanisms of XY body formation in eutherians and marsupials.Low- and High-LET Ionizing Radiation Induces Delayed Homologous Recombination that Persists for Two Weeks before Resolving.The ATM inhibitor KU55933 sensitizes radioresistant bladder cancer cells with DAB2IP gene defect.Targeting BRCA1-BER deficient breast cancer by ATM or DNA-PKcs blockade either alone or in combination with cisplatin for personalized therapyRepair of chromosomal double-strand breaks by precise ligation in human cells.BRCA1 requirement for the fidelity of plasmid DNA double-strand break repair in cultured breast epithelial cells.Proteomic analysis of BRCA1-depleted cell line reveals a putative role for replication protein A2 up-regulation in BRCA1 breast tumor development.A duplex DNA-gold nanoparticle probe composed as a colorimetric biosensor for sequence-specific DNA-binding proteins.A yeast recombination assay to characterize human BRCA1 missense variants of unknown pathological significance.The brain-penetrant clinical ATM inhibitor AZD1390 radiosensitizes and improves survival of preclinical brain tumor models.
P2860
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P2860
Good timing in the cell cycle for precise DNA repair by BRCA1.
description
2005 nî lūn-bûn
@nan
2005 թուականի Սեպտեմբերին հրատարակուած գիտական յօդուած
@hyw
2005 թվականի սեպտեմբերին հրատարակված գիտական հոդված
@hy
2005年の論文
@ja
2005年論文
@yue
2005年論文
@zh-hant
2005年論文
@zh-hk
2005年論文
@zh-mo
2005年論文
@zh-tw
2005年论文
@wuu
name
Good timing in the cell cycle for precise DNA repair by BRCA1.
@ast
Good timing in the cell cycle for precise DNA repair by BRCA1.
@en
type
label
Good timing in the cell cycle for precise DNA repair by BRCA1.
@ast
Good timing in the cell cycle for precise DNA repair by BRCA1.
@en
prefLabel
Good timing in the cell cycle for precise DNA repair by BRCA1.
@ast
Good timing in the cell cycle for precise DNA repair by BRCA1.
@en
P356
P1433
P1476
Good timing in the cell cycle for precise DNA repair by BRCA1.
@en
P2093
Jac A Nickoloff
Stephen T Durant
P304
P356
10.4161/CC.4.9.2027
P577
2005-09-13T00:00:00Z