Blood-brain barrier invasion by group B Streptococcus depends upon proper cell-surface anchoring of lipoteichoic acid.
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Defense at the border: the blood-brain barrier versus bacterial foreignersPathogens penetrating the central nervous system: infection pathways and the cellular and molecular mechanisms of invasionHost-pathogen interactions in bacterial meningitisUnderstanding the regulation of Group B Streptococcal virulence factorsA hemolytic pigment of Group B Streptococcus allows bacterial penetration of human placentaRecent advances in understanding the molecular basis of group B Streptococcus virulenceLipoteichoic acid in Streptomyces hygroscopicus: structural model and immunomodulatory activitiesRegulation of CovR expression in Group B Streptococcus impacts blood-brain barrier penetration.Anthrax toxins inhibit neutrophil signaling pathways in brain endothelium and contribute to the pathogenesis of meningitisMacroamphiphilic components of thermophilic actinomycetes: identification of lipoteichoic acid in Thermobifida fusca.Strains of Group B streptococci from septic patients induce platelet activation via Toll-like Receptor 2.Identification of a group B streptococcal fibronectin binding protein, SfbA, that contributes to invasion of brain endothelium and development of meningitis.Analysis of two-component systems in group B Streptococcus shows that RgfAC and the novel FspSR modulate virulence and bacterial fitness.Invasion of the central nervous system by Cryptococcus neoformans requires a secreted fungal metalloprotease.Deletion of the glycosyltransferase bgsB of Enterococcus faecalis leads to a complete loss of glycolipids from the cell membrane and to impaired biofilm formationAnchors away: contribution of a glycolipid anchor to bacterial invasion of host cells.Penetration of the blood-brain barrier by Staphylococcus aureus: contribution of membrane-anchored lipoteichoic acid.Glycosaminoglycan binding facilitates entry of a bacterial pathogen into central nervous systemsEndocytosis at the blood-brain barrier: from basic understanding to drug delivery strategies.Large-scale screening of a targeted Enterococcus faecalis mutant library identifies envelope fitness factors.Activation of brain endothelium by pneumococcal neuraminidase NanA promotes bacterial internalization.Group B streptococcal serine-rich repeat proteins promote interaction with fibrinogen and vaginal colonizationCalmodulin kinase II-dependent transactivation of PDGF receptors mediates astrocytic MMP-9 expression and cell motility induced by lipoteichoic acidGroup B Streptococcus interactions with human meningeal cells and astrocytes in vitro.Binding of glycoprotein Srr1 of Streptococcus agalactiae to fibrinogen promotes attachment to brain endothelium and the development of meningitis.Association and virulence gene expression vary among serotype III group B streptococcus isolates following exposure to decidual and lung epithelial cells.Genes required for glycolipid synthesis and lipoteichoic acid anchoring in Staphylococcus aureusInteraction of neonatal phagocytes with group B streptococcus: recognition and response.Group B Streptococcus hijacks the host plasminogen system to promote brain endothelial cell invasionPenetration and activation of brain endothelium by Salmonella enterica serovar Typhimurium.The role of autophagy during group B Streptococcus infection of blood-brain barrier endotheliumGroup B Streptococcus engages an inhibitory Siglec through sialic acid mimicry to blunt innate immune and inflammatory responses in vivoThe sensor histidine kinase RgfC affects group B streptococcal virulence factor expression independent of its response regulator RgfA.Streptococcus agalactiae infection in zebrafish larvaeHost cytosolic phospholipase A₂α contributes to group B Streptococcus penetration of the blood-brain barrier.Contribution of lethal toxin and edema toxin to the pathogenesis of anthrax meningitis.Bacterial Pili exploit integrin machinery to promote immune activation and efficient blood-brain barrier penetration.Serine-rich repeat proteins and pili promote Streptococcus agalactiae colonization of the vaginal tract.Serine/threonine phosphatase Stp1 mediates post-transcriptional regulation of hemolysin, autolysis, and virulence of group B Streptococcus.Group B streptococcal infection and activation of human astrocytes.
P2860
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P2860
Blood-brain barrier invasion by group B Streptococcus depends upon proper cell-surface anchoring of lipoteichoic acid.
description
2005 nî lūn-bûn
@nan
2005 թուականի Սեպտեմբերին հրատարակուած գիտական յօդուած
@hyw
2005 թվականի սեպտեմբերին հրատարակված գիտական հոդված
@hy
2005年の論文
@ja
2005年論文
@yue
2005年論文
@zh-hant
2005年論文
@zh-hk
2005年論文
@zh-mo
2005年論文
@zh-tw
2005年论文
@wuu
name
Blood-brain barrier invasion b ...... nchoring of lipoteichoic acid.
@ast
Blood-brain barrier invasion b ...... nchoring of lipoteichoic acid.
@en
type
label
Blood-brain barrier invasion b ...... nchoring of lipoteichoic acid.
@ast
Blood-brain barrier invasion b ...... nchoring of lipoteichoic acid.
@en
prefLabel
Blood-brain barrier invasion b ...... nchoring of lipoteichoic acid.
@ast
Blood-brain barrier invasion b ...... nchoring of lipoteichoic acid.
@en
P2093
P2860
P356
P1476
Blood-brain barrier invasion b ...... nchoring of lipoteichoic acid.
@en
P2093
Andreas Peschel
Arya Khosravi
Erin J Engelson
Heather C Maisey
Iris Fedtke
Kathrin S Michelsen
Kelly S Doran
Ozlem Equils
P2860
P304
P356
10.1172/JCI23829
P407
P577
2005-09-01T00:00:00Z