Caspases and calpain are independent mediators of cisplatin-induced endothelial cell necrosis.
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Mechanism of Platinum Derivatives Induced Kidney InjuryMechanisms of Cisplatin nephrotoxicityEndoplasmic reticulum stress-induced cell death in dopaminergic cells: effect of resveratrolCoupling endoplasmic reticulum stress to the cell death program in dopaminergic cells: effect of paraquatPathophysiology of cisplatin-induced acute kidney injury.An mTOR anti-sense oligonucleotide decreases polycystic kidney disease in mice with a targeted mutation in Pkd2.An mTOR kinase inhibitor slows disease progression in a rat model of polycystic kidney diseaseIL-33 exacerbates acute kidney injuryβ-Caryophyllene ameliorates cisplatin-induced nephrotoxicity in a cannabinoid 2 receptor-dependent manner.Evaluation of urine biomarkers of kidney injury in polycystic kidney disease.Incidence of Venous Thromboembolism in cancer patients treated with Cisplatin based chemotherapy - a cohort study.zVAD-fmk prevents cisplatin-induced cleavage of autophagy proteins but impairs autophagic flux and worsens renal function.Vegfa protects the glomerular microvasculature in diabetes.Interleukin-18 binding protein transgenic mice are protected against ischemic acute kidney injury.Macrophages are not the source of injurious interleukin-18 in ischemic acute kidney injury in miceThe caspase 3 sensor Phiphilux G2D2 is activated non-specifically in S1 renal proximal tubules.NLRP3 inflammasome knockout mice are protected against ischemic but not cisplatin-induced acute kidney injury.Induction of hypoxia and necrosis in multicellular tumor spheroids is associated with resistance to chemotherapy treatment.Tumor promotion via injury- and death-induced inflammationAn Early and Robust Activation of Caspases Heads Cells for a Regulated Form of Necrotic-like Cell Death.CD4 T cell knockout does not protect against kidney injury and worsens cancer.The mode of cisplatin-induced cell death in CYP2E1-overexpressing HepG2 cells: modulation by ERK, ROS, glutathione, and thioredoxin.Role of IL-1α in cisplatin-induced acute renal failure in mice.Effect of pomegranate flower extract on cisplatin-induced nephrotoxicity in rats.Oxaliplatin-induced transient ischaemic attack.IL-33 deficiency slows cancer growth but does not protect against cisplatin-induced AKI in mice with cancer.Thromboembolism in Patients with Bladder Cancer: Incidence, Risk Factors and Prevention.
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P2860
Caspases and calpain are independent mediators of cisplatin-induced endothelial cell necrosis.
description
2006 nî lūn-bûn
@nan
2006 թուականի Ապրիլին հրատարակուած գիտական յօդուած
@hyw
2006 թվականի ապրիլին հրատարակված գիտական հոդված
@hy
2006年の論文
@ja
2006年論文
@yue
2006年論文
@zh-hant
2006年論文
@zh-hk
2006年論文
@zh-mo
2006年論文
@zh-tw
2006年论文
@wuu
name
Caspases and calpain are indep ...... ced endothelial cell necrosis.
@ast
Caspases and calpain are indep ...... ced endothelial cell necrosis.
@en
type
label
Caspases and calpain are indep ...... ced endothelial cell necrosis.
@ast
Caspases and calpain are indep ...... ced endothelial cell necrosis.
@en
prefLabel
Caspases and calpain are indep ...... ced endothelial cell necrosis.
@ast
Caspases and calpain are indep ...... ced endothelial cell necrosis.
@en
P2093
P2860
P1476
Caspases and calpain are indep ...... ced endothelial cell necrosis.
@en
P2093
Belda Dursun
Charles L Edelstein
Dong-Jin Oh
Hilary Somerset
Sarah Faubel
P2860
P304
P356
10.1152/AJPRENAL.00455.2005
P577
2006-04-18T00:00:00Z