Genetic background determines response to hemostasis and thrombosis. - Wikidata - wikimedia - TriplyDB

Genetic background determines response to hemostasis and thrombosis.

Genetic background determines response to hemostasis and thrombosis.

http://www.wikidata.org/entity/Q33259449

about

Genetic dissection of the Canq1 locus governing variation in extent of the collateral circulation Hybrid mouse diversity panel: a panel of inbred mouse strains suitable for analysis of complex genetic traits.Genomic survey of prepulse inhibition in mouse chromosome substitution strains.Murine strain differences in hemostasis and thrombosis and tissue factor pathway inhibitor Wide genetic variation in the native pial collateral circulation is a major determinant of variation in severity of stroke.Strain and model dependent differences in inflammatory cell recruitment in mice Genetic determinants of atherosclerosis, obesity, and energy balance in consomic mice.Genetic dissection of quantitative trait Loci for hemostasis and thrombosis on mouse chromosomes 11 and 5 using congenic and subcongenic strains.EMILIN2 regulates platelet activation, thrombus formation, and clot retraction.Quantitative trait locus analysis for hemostasis and thrombosis Mouse models for studies of cardiovascular complications of type 1 diabetes.Ablation of promyelocytic leukemia protein (PML) re-patterns energy balance and protects mice from obesity induced by a Western diet A nonsynonymous SNP within PCDH15 is associated with lipid traits in familial combined hyperlipidemia.Does plasmin have anticoagulant activity?Analysis of Fyn function in hemostasis and alphaIIbbeta3-integrin signaling.Mouse chromosome 17 candidate modifier genes for thrombosis.EMILIN2 (Elastin microfibril interface located protein), potential modifier of thrombosis.Influence of genetic background on bleeding phenotype in the tail-tip bleeding model and recommendations for standardization: communication from the SSC of the ISTH.

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P2860

Genetic background determines response to hemostasis and thrombosis.

http://www.wikidata.org/entity/Q33259449

description

2006 nî lūn-bûn

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2006 թուականի Հոկտեմբերին հրատարակուած գիտական յօդուած

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2006 թվականի հոտեմբերին հրատարակված գիտական հոդված

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2006年の論文

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2006年論文

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2006年論文

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2006年論文

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2006年論文

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2006年論文

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2006年论文

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name

Genetic background determines response to hemostasis and thrombosis.

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Genetic background determines response to hemostasis and thrombosis.

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Genetic background determines response to hemostasis and thrombosis.

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type

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Genetic background determines response to hemostasis and thrombosis.

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Genetic background determines response to hemostasis and thrombosis.

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Genetic background determines response to hemostasis and thrombosis.

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Genetic background determines response to hemostasis and thrombosis.

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Genetic background determines response to hemostasis and thrombosis.

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Genetic background determines response to hemostasis and thrombosis.

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BMC Blood Disorders

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Genetic background determines response to hemostasis and thrombosis.

@en

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Aleksey Shchurin

http://www.w3.org/2001/XMLSchema#string

Annie E Hill

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Erika Hart

http://www.w3.org/2001/XMLSchema#string

Jane Hoover-Plow

http://www.w3.org/2001/XMLSchema#string

Jingfeng Sha

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Jonathan B Singer

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Joseph H Nadeau

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P2860

Atherosclerosis

Remodeling of the vessel wall after copper-induced injury is highly attenuated in mice with a total deficiency of plasminogen activator inhibitor-1

Leptin-dependent platelet aggregation and arterial thrombosis suggests a mechanism for atherothrombotic disease in obesity

Two quantitative trait loci for prepulse inhibition of startle identified on mouse chromosome 16 using chromosome substitution strains

Vascular-bed--specific hemostasis and hypercoagulable states.

Characterization of the cDNA coding for mouse plasminogen and localization of the gene to mouse chromosome 17.

Analysing complex genetic traits with chromosome substitution strains.

Plasminogen activator inhibitor-1 gene-deficient mice. I. Generation by homologous recombination and characterization.

Thrombin and fibrinolysis.

The study of gene-environment interactions that influence thrombosis and fibrinolysis. Genetic variation at the loci for factor VII and plasminogen activator inhibitor-1.

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