Gene expression patterns associated with p53 status in breast cancer.
about
Understanding and treating triple-negative breast cancerComprehensive molecular portraits of human breast tumoursThe miR-644a/CTBP1/p53 axis suppresses drug resistance by simultaneous inhibition of cell survival and epithelial-mesenchymal transition in breast cancerRNAi-mediated knock-down of arylamine N-acetyltransferase-1 expression induces E-cadherin up-regulation and cell-cell contact growth inhibitionEvidence for phenotypic plasticity in aggressive triple-negative breast cancer: human biology is recapitulated by a novel model systemDifferential pathogenesis of lung adenocarcinoma subtypes involving sequence mutations, copy number, chromosomal instability, and methylationA compact VEGF signature associated with distant metastases and poor outcomes.Current data of targeted therapies for the treatment of triple-negative advanced breast cancer: empiricism or evidence-based?Expression signatures of TP53 mutations in serous ovarian cancers.Prognostic interaction between expression of p53 and estrogen receptor in patients with node-negative breast cancer: results from IBCSG Trials VIII and IXContribution of p53 to metastasis.Building prognostic models for breast cancer patients using clinical variables and hundreds of gene expression signatures.Unraveling the regulatory connections between two controllers of breast cancer cell fateDo two machine-learning based prognostic signatures for breast cancer capture the same biological processes?Altered-function p53 missense mutations identified in breast cancers can have subtle effects on transactivation.Mitochondrial Hep27 is a c-Myb target gene that inhibits Mdm2 and stabilizes p53.Biology, metastatic patterns, and treatment of patients with triple-negative breast cancer.Multiplatform analysis of 12 cancer types reveals molecular classification within and across tissues of originTriple Negative Breast Cancer - An OverviewTreatment options for patients with triple-negative breast cancerTargeting breast cancer stem cells.Stem cell-like gene expression in ovarian cancer predicts type II subtype and prognosisNovel neoadjuvant therapy paradigms for bladder cancer: results from the National Cancer Center Institute Forum.Overexpression of miR-146a in basal-like breast cancer cells confers enhanced tumorigenic potential in association with altered p53 statusCharacterization of the p53 cistrome--DNA binding cooperativity dissects p53's tumor suppressor functions.Comparison and identification of estrogen-receptor related gene expression profiles in breast cancer of different ethnic originsIdentification of potential synthetic lethal genes to p53 using a computational biology approachCan systems biology understand pathway activation? Gene expression signatures as surrogate markers for understanding the complexity of pathway activation.An integrated genomics approach identifies drivers of proliferation in luminal-subtype human breast cancer.Pathologic progression of mammary carcinomas in a C3(1)/SV40 T/t-antigen transgenic rat model of human triple-negative and Her2-positive breast cancer.Genomic analyses across six cancer types identify basal-like breast cancer as a unique molecular entity.Effects of TP53 mutational status on gene expression patterns across 10 human cancer types.Evaluation of public cancer datasets and signatures identifies TP53 mutant signatures with robust prognostic and predictive valueTriple-Negative Breast Cancer: Clinical and Histological Correlations.Gene expression analysis of in vitro cocultures to study interactions between breast epithelium and stroma.Chemotherapy response and recurrence-free survival in neoadjuvant breast cancer depends on biomarker profiles: results from the I-SPY 1 TRIAL (CALGB 150007/150012; ACRIN 6657).p53, a target of estrogen receptor (ER) α, modulates DNA damage-induced growth suppression in ER-positive breast cancer cells.Prognostic and predictive value of TP53 mutations in node-positive breast cancer patients treated with anthracycline- or anthracycline/taxane-based adjuvant therapy: results from the BIG 02-98 phase III trial.Response and survival of breast cancer intrinsic subtypes following multi-agent neoadjuvant chemotherapy.p53 deficiency linked to B cell translocation gene 2 (BTG2) loss enhances metastatic potential by promoting tumor growth in primary and metastatic sites in patient-derived xenograft (PDX) models of triple-negative breast cancer.
P2860
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P2860
Gene expression patterns associated with p53 status in breast cancer.
description
2006 nî lūn-bûn
@nan
2006 թուականի Դեկտեմբերին հրատարակուած գիտական յօդուած
@hyw
2006 թվականի դեկտեմբերին հրատարակված գիտական հոդված
@hy
2006年の論文
@ja
2006年論文
@yue
2006年論文
@zh-hant
2006年論文
@zh-hk
2006年論文
@zh-mo
2006年論文
@zh-tw
2006年论文
@wuu
name
Gene expression patterns associated with p53 status in breast cancer.
@ast
Gene expression patterns associated with p53 status in breast cancer.
@en
type
label
Gene expression patterns associated with p53 status in breast cancer.
@ast
Gene expression patterns associated with p53 status in breast cancer.
@en
prefLabel
Gene expression patterns associated with p53 status in breast cancer.
@ast
Gene expression patterns associated with p53 status in breast cancer.
@en
P2093
P2860
P356
P1433
P1476
Gene expression patterns associated with p53 status in breast cancer
@en
P2093
Claire S Barbier
Daniel S Oh
Jason I Herschkowitz
Melissa A Troester
Xiaping He
P2860
P2888
P356
10.1186/1471-2407-6-276
P407
P577
2006-12-06T00:00:00Z
P5875
P6179
1039833406