A critical role for peptidoglycan N-deacetylation in Listeria evasion from the host innate immune system. - Wikidata - wikimedia - TriplyDB

A critical role for peptidoglycan N-deacetylation in Listeria evasion from the host innate immune system.

A critical role for peptidoglycan N-deacetylation in Listeria evasion from the host innate immune system.

http://www.wikidata.org/entity/Q33268810

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Rhombencephalitis Caused by Listeria monocytogenes in Humans and Ruminants: A Zoonosis on the Rise?Function of Nod-like receptors in microbial recognition and host defense Bacterial peptidoglycan degrading enzymes and their impact on host muropeptide detection Peptidoglycan remodeling by the coordinated action of multispecific enzymes Subversion of host recognition and defense systems by Francisella spp Streptococcus mutans SMU.623c Codes for a Functional, Metal-Dependent Polysaccharide Deacetylase That Modulates Interactions with Salivary Agglutinin Structural and functional characterization of a putative polysaccharide deacetylase of the human parasiteEncephalitozoon cuniculi The Structure of Helicobacter pylori HP0310 Reveals an Atypical Peptidoglycan Deacetylase Structure determination of BA0150, a putative polysaccharide deacetylase fromBacillus anthracis Characterization of the divalent metal binding site of bacterial polysaccharide deacetylase using crystallography and quantum chemical calculations International Union of Basic and Clinical Pharmacology. XCVI. Pattern recognition receptors in health and disease Transcytosis of Listeria monocytogenes across the intestinal barrier upon specific targeting of goblet cell accessible E-cadherin Protective and pro-inflammatory roles of intestinal bacteria Pseudomonas aeruginosa exploits lipid A and muropeptides modification as a strategy to lower innate immunity during cystic fibrosis lung infection L-Rhamnosylation of Listeria monocytogenes Wall Teichoic Acids Promotes Resistance to Antimicrobial Peptides by Delaying Interaction with the Membrane Nod1 signaling overcomes resistance of S. pneumoniae to opsonophagocytic killing.Nod-like receptors: cytosolic watchdogs for immunity against pathogens.Distinct TLR- and NLR-mediated transcriptional responses to an intracellular pathogen.Resistance to mucosal lysozyme compensates for the fitness deficit of peptidoglycan modifications by Streptococcus pneumoniae.Oxidative stress-induced peptidoglycan deacetylase in Helicobacter pylori.In vivo transcriptional profiling of Listeria monocytogenes and mutagenesis identify new virulence factors involved in infection.NOD2, RIP2 and IRF5 play a critical role in the type I interferon response to Mycobacterium tuberculosis.The bacterial pathogen Listeria monocytogenes and the interferon family: type I, type II and type III interferons How Listeria monocytogenes organizes its surface for virulence Identification of genetic loci in Lactobacillus plantarum that modulate the immune response of dendritic cells using comparative genome hybridization Clostridium difficile extracytoplasmic function σ factor σV regulates lysozyme resistance and is necessary for pathogenesis in the hamster model of infection.Comparative experimental infection of Listeria monocytogenes and Listeria ivanovii in bovine trophoblasts.The Mycobacterium tuberculosis protein LdtMt2 is a nonclassical transpeptidase required for virulence and resistance to amoxicillin.Mycobacterium tuberculosis Rv1096 protein: gene cloning, protein expression, and peptidoglycan deacetylase activity.The novel polysaccharide deacetylase homologue Pdi contributes to virulence of the aquatic pathogen Streptococcus iniae.Transcriptomic and phenotypic responses of Listeria monocytogenes strains possessing different growth efficiencies under acidic conditions.Host-guest chemistry of the peptidoglycan The Listeria monocytogenes InlC protein interferes with innate immune responses by targeting the I{kappa}B kinase subunit IKK{alpha}.Both TLR2 and TRIF contribute to interferon-β production during Listeria infection The timing of IFNβ production affects early innate responses to Listeria monocytogenes and determines the overall outcome of lethal infection.Immune receptors involved in Streptococcus suis recognition by dendritic cells.Listeria monocytogenes is resistant to lysozyme through the regulation, not the acquisition, of cell wall-modifying enzymes.SpoVG Is a Conserved RNA-Binding Protein That Regulates Listeria monocytogenes Lysozyme Resistance, Virulence, and Swarming Motility.Modifications to the peptidoglycan backbone help bacteria to establish infection Antimicrobial Peptide Resistance Mechanisms of Gram-Positive Bacteria.

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P2860

A critical role for peptidoglycan N-deacetylation in Listeria evasion from the host innate immune system.

http://www.wikidata.org/entity/Q33268810

description

2007 nî lūn-bûn

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2007 թուականի Յունուարին հրատարակուած գիտական յօդուած

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2007 թվականի հունվարին հրատարակված գիտական հոդված

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2007年の論文

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2007年論文

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2007年論文

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2007年論文

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2007年論文

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2007年論文

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2007年论文

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A critical role for peptidogly ...... the host innate immune system.

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A critical role for peptidogly ...... the host innate immune system.

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A critical role for peptidogly ...... the host innate immune system.

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A critical role for peptidogly ...... the host innate immune system.

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A critical role for peptidogly ...... the host innate immune system.

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A critical role for peptidogly ...... the host innate immune system.

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Anthony Coyle

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Dana J Philpott

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Emmanuel Psylinakis

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Jean-Claude Rousselle

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John Bertin

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Marco Giovannini

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Marie-Anne Nahori

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Marie-Christine Prévost

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Michel Chignard

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Nadège Cayet

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P2860

Nod1, an Apaf-1-like activator of caspase-9 and nuclear factor-kappaB

The complete genome sequence of the gastric pathogen Helicobacter pylori

RICK/Rip2/CARDIAK mediates signalling for receptors of the innate and adaptive immune systems

Host recognition of bacterial muramyl dipeptide mediated through NOD2. Implications for Crohn's disease

CARD4/Nod1 mediates NF-kappaB and JNK activation by invasive Shigella flexneri.

Nod2-dependent regulation of innate and adaptive immunity in the intestinal tract

NOD2 is a negative regulator of Toll-like receptor 2-mediated T helper type 1 responses

Analysis of gene control signals by DNA fusion and cloning in Escherichia coli

Nod1 detects a unique muropeptide from gram-negative bacterial peptidoglycan

Detailed structural analysis of the peptidoglycan of the human pathogen Neisseria meningitidis.

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997-1002

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104

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Pascale Cossart

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6586857

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