Perturbed gap-filling synthesis in nucleotide excision repair causes histone H2AX phosphorylation in human quiescent cells. - Wikidata - wikimedia - TriplyDB

Perturbed gap-filling synthesis in nucleotide excision repair causes histone H2AX phosphorylation in human quiescent cells.

Perturbed gap-filling synthesis in nucleotide excision repair causes histone H2AX phosphorylation in human quiescent cells.

http://www.wikidata.org/entity/Q33275895

about

Disruption of TTDA results in complete nucleotide excision repair deficiency and embryonic lethality Implication of posttranslational histone modifications in nucleotide excision repair Nucleotide excision repair in eukaryotes Involvement of global genome repair, transcription coupled repair, and chromatin remodeling in UV DNA damage response changes during development Identification of Small Molecule Proliferating Cell Nuclear Antigen (PCNA) Inhibitor That Disrupts Interactions with PIP-box Proteins and Inhibits DNA Replication Replication independent ATR signalling leads to G2/M arrest requiring Nbs1, 53BP1 and MDC1.Chromatin restoration following nucleotide excision repair involves the incorporation of ubiquitinated H2A at damaged genomic sites.Physical and functional interaction between DDB and XPA in nucleotide excision repair.Repair of laser-localized DNA interstrand cross-links in G1 phase mammalian cells Kinetics of the UV-induced DNA damage response in relation to cell cycle phase. Correlation with DNA replication.Residual gammaH2AX foci as an indication of lethal DNA lesions.A novel and simple micro-irradiation technique for creating localized DNA double-strand breaks.Proliferating cell nuclear antigen-dependent rapid recruitment of Cdt1 and CRL4Cdt2 at DNA-damaged sites after UV irradiation in HeLa cells.Creating localized DNA double-strand breaks with microirradiation.The DNA damage response kinases DNA-dependent protein kinase (DNA-PK) and ataxia telangiectasia mutated (ATM) Are stimulated by bulky adduct-containing DNA.Dynamic link of DNA demethylation, DNA strand breaks and repair in mouse zygotes.The ubiquitin-selective segregase VCP/p97 orchestrates the response to DNA double-strand breaks.NER initiation factors, DDB2 and XPC, regulate UV radiation response by recruiting ATR and ATM kinases to DNA damage sites DNA repair synthesis and ligation affect the processing of excised oligonucleotides generated by human nucleotide excision repair.The contribution of mitochondrial thymidylate synthesis in preventing the nuclear genome stress UV-induced histone H2AX phosphorylation and DNA damage related proteins accumulate and persist in nucleotide excision repair-deficient XP-B cells Nucleotide excision repair-induced H2A ubiquitination is dependent on MDC1 and RNF8 and reveals a universal DNA damage response.Functional relevance of the histone gammaH2Ax in the response to DNA damaging agents.Creating a monomeric endonuclease TALE-I-SceI with high specificity and low genotoxicity in human cells.The contribution of CMP kinase to the efficiency of DNA repair BRCA1 and BRCA2 protect against oxidative DNA damage converted into double-strand breaks during DNA replication.High risk of benzo[α]pyrene-induced lung cancer in E160D FEN1 mutant mice ATP-dependent chromatin remodeling in the DNA-damage response.Reconstitution of a human ATR-mediated checkpoint response to damaged DNA.RNA polymerase: the most specific damage recognition protein in cellular responses to DNA damage?Mammalian ribonucleotide reductase subunit p53R2 is required for mitochondrial DNA replication and DNA repair in quiescent cells Bringing It All Together: Coupling Excision Repair to the DNA Damage Checkpoint.DNA excision repair: where do all the dimers go?Identification of RNF8 as a ubiquitin ligase involved in targeting the p12 subunit of DNA polymerase δ for degradation in response to DNA damage ATR Kinase Inhibition Protects Non-cycling Cells from the Lethal Effects of DNA Damage and Transcription Stress.Histone H2AX phosphorylation in response to changes in chromatin structure induced by altered osmolarity Cooperative activation of the ATR checkpoint kinase by TopBP1 and damaged DNA.ELL, a novel TFIIH partner, is involved in transcription restart after DNA repair.Human SNF5/INI1, a component of the human SWI/SNF chromatin remodeling complex, promotes nucleotide excision repair by influencing ATM recruitment and downstream H2AX phosphorylation Coupling of human DNA excision repair and the DNA damage checkpoint in a defined in vitro system.

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P2860

Perturbed gap-filling synthesis in nucleotide excision repair causes histone H2AX phosphorylation in human quiescent cells.

http://www.wikidata.org/entity/Q33275895

description

2007 nî lūn-bûn

@nan

2007 թուականի Փետրուարին հրատարակուած գիտական յօդուած

@hyw

2007 թվականի փետրվարին հրատարակված գիտական հոդված

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2007年の論文

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2007年論文

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2007年論文

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2007年論文

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2007年論文

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2007年論文

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2007年论文

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name

Perturbed gap-filling synthesi ...... tion in human quiescent cells.

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Perturbed gap-filling synthesi ...... tion in human quiescent cells.

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Perturbed gap-filling synthesi ...... tion in human quiescent cells.

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type

label

Perturbed gap-filling synthesi ...... tion in human quiescent cells.

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Perturbed gap-filling synthesi ...... tion in human quiescent cells.

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Perturbed gap-filling synthesi ...... tion in human quiescent cells.

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prefLabel

Perturbed gap-filling synthesi ...... tion in human quiescent cells.

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Perturbed gap-filling synthesi ...... tion in human quiescent cells.

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Perturbed gap-filling synthesi ...... tion in human quiescent cells.

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Perturbed gap-filling synthesi ...... tion in human quiescent cells.

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Ai Igarashi

http://www.w3.org/2001/XMLSchema#string

Kanji Ishizaki

http://www.w3.org/2001/XMLSchema#string

Katsumi Yamashita

http://www.w3.org/2001/XMLSchema#string

Kie Yaginuma

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Kuniyoshi Iwabuchi

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Manabu Inobe

http://www.w3.org/2001/XMLSchema#string

Mayumi Imura

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Megumi Matsumoto

http://www.w3.org/2001/XMLSchema#string

Mizuho Hasegawa

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Takayasu Date

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1104-1112

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10.1242/JCS.03391

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Pt 6

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120

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2007-02-27T00:00:00Z

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6479809

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17327276

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phosphorylation