Perturbed gap-filling synthesis in nucleotide excision repair causes histone H2AX phosphorylation in human quiescent cells.
about
Disruption of TTDA results in complete nucleotide excision repair deficiency and embryonic lethalityImplication of posttranslational histone modifications in nucleotide excision repairNucleotide excision repair in eukaryotesInvolvement of global genome repair, transcription coupled repair, and chromatin remodeling in UV DNA damage response changes during developmentIdentification of Small Molecule Proliferating Cell Nuclear Antigen (PCNA) Inhibitor That Disrupts Interactions with PIP-box Proteins and Inhibits DNA ReplicationReplication independent ATR signalling leads to G2/M arrest requiring Nbs1, 53BP1 and MDC1.Chromatin restoration following nucleotide excision repair involves the incorporation of ubiquitinated H2A at damaged genomic sites.Physical and functional interaction between DDB and XPA in nucleotide excision repair.Repair of laser-localized DNA interstrand cross-links in G1 phase mammalian cellsKinetics of the UV-induced DNA damage response in relation to cell cycle phase. Correlation with DNA replication.Residual gammaH2AX foci as an indication of lethal DNA lesions.A novel and simple micro-irradiation technique for creating localized DNA double-strand breaks.Proliferating cell nuclear antigen-dependent rapid recruitment of Cdt1 and CRL4Cdt2 at DNA-damaged sites after UV irradiation in HeLa cells.Creating localized DNA double-strand breaks with microirradiation.The DNA damage response kinases DNA-dependent protein kinase (DNA-PK) and ataxia telangiectasia mutated (ATM) Are stimulated by bulky adduct-containing DNA.Dynamic link of DNA demethylation, DNA strand breaks and repair in mouse zygotes.The ubiquitin-selective segregase VCP/p97 orchestrates the response to DNA double-strand breaks.NER initiation factors, DDB2 and XPC, regulate UV radiation response by recruiting ATR and ATM kinases to DNA damage sitesDNA repair synthesis and ligation affect the processing of excised oligonucleotides generated by human nucleotide excision repair.The contribution of mitochondrial thymidylate synthesis in preventing the nuclear genome stressUV-induced histone H2AX phosphorylation and DNA damage related proteins accumulate and persist in nucleotide excision repair-deficient XP-B cellsNucleotide excision repair-induced H2A ubiquitination is dependent on MDC1 and RNF8 and reveals a universal DNA damage response.Functional relevance of the histone gammaH2Ax in the response to DNA damaging agents.Creating a monomeric endonuclease TALE-I-SceI with high specificity and low genotoxicity in human cells.The contribution of CMP kinase to the efficiency of DNA repairBRCA1 and BRCA2 protect against oxidative DNA damage converted into double-strand breaks during DNA replication.High risk of benzo[α]pyrene-induced lung cancer in E160D FEN1 mutant miceATP-dependent chromatin remodeling in the DNA-damage response.Reconstitution of a human ATR-mediated checkpoint response to damaged DNA.RNA polymerase: the most specific damage recognition protein in cellular responses to DNA damage?Mammalian ribonucleotide reductase subunit p53R2 is required for mitochondrial DNA replication and DNA repair in quiescent cellsBringing It All Together: Coupling Excision Repair to the DNA Damage Checkpoint.DNA excision repair: where do all the dimers go?Identification of RNF8 as a ubiquitin ligase involved in targeting the p12 subunit of DNA polymerase δ for degradation in response to DNA damageATR Kinase Inhibition Protects Non-cycling Cells from the Lethal Effects of DNA Damage and Transcription Stress.Histone H2AX phosphorylation in response to changes in chromatin structure induced by altered osmolarityCooperative activation of the ATR checkpoint kinase by TopBP1 and damaged DNA.ELL, a novel TFIIH partner, is involved in transcription restart after DNA repair.Human SNF5/INI1, a component of the human SWI/SNF chromatin remodeling complex, promotes nucleotide excision repair by influencing ATM recruitment and downstream H2AX phosphorylationCoupling of human DNA excision repair and the DNA damage checkpoint in a defined in vitro system.
P2860
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P2860
Perturbed gap-filling synthesis in nucleotide excision repair causes histone H2AX phosphorylation in human quiescent cells.
description
2007 nî lūn-bûn
@nan
2007 թուականի Փետրուարին հրատարակուած գիտական յօդուած
@hyw
2007 թվականի փետրվարին հրատարակված գիտական հոդված
@hy
2007年の論文
@ja
2007年論文
@yue
2007年論文
@zh-hant
2007年論文
@zh-hk
2007年論文
@zh-mo
2007年論文
@zh-tw
2007年论文
@wuu
name
Perturbed gap-filling synthesi ...... tion in human quiescent cells.
@ast
Perturbed gap-filling synthesi ...... tion in human quiescent cells.
@en
Perturbed gap-filling synthesi ...... tion in human quiescent cells.
@nl
type
label
Perturbed gap-filling synthesi ...... tion in human quiescent cells.
@ast
Perturbed gap-filling synthesi ...... tion in human quiescent cells.
@en
Perturbed gap-filling synthesi ...... tion in human quiescent cells.
@nl
prefLabel
Perturbed gap-filling synthesi ...... tion in human quiescent cells.
@ast
Perturbed gap-filling synthesi ...... tion in human quiescent cells.
@en
Perturbed gap-filling synthesi ...... tion in human quiescent cells.
@nl
P2093
P356
P1476
Perturbed gap-filling synthesi ...... tion in human quiescent cells.
@en
P2093
Ai Igarashi
Kanji Ishizaki
Katsumi Yamashita
Kie Yaginuma
Kuniyoshi Iwabuchi
Manabu Inobe
Mayumi Imura
Megumi Matsumoto
Mizuho Hasegawa
Takayasu Date
P304
P356
10.1242/JCS.03391
P407
P577
2007-02-27T00:00:00Z