Isolation of murine microglial cells for RNA analysis or flow cytometry.
about
When and how do seizures kill neurons, and is cell death relevant to epileptogenesis?A novel antagonist of p75NTR reduces peripheral expansion and CNS trafficking of pro-inflammatory monocytes and spares function after traumatic brain injury.Focal transient CNS vessel leak provides a tissue niche for sequential immune cell accumulation during the asymptomatic phase of EAE induction.Spatial and temporal profiles of growth factor expression during CNS demyelination reveal the dynamics of repair primingIdentification of a unique TGF-β-dependent molecular and functional signature in microgliaOncogenic Kras expression in postmitotic neurons leads to S100A8-S100A9 protein overexpression and gliosisIntegrated expression profiles of mRNA and miRNA in polarized primary murine microglia.Microglia change from a reactive to an age-like phenotype with the time in cultureSelective chemokine receptor usage by central nervous system myeloid cells in CCR2-red fluorescent protein knock-in mice.Microglial activation mediates host neuronal survival induced by neural stem cells.Defining the chemokine basis for leukocyte recruitment during viral encephalitis.Central nervous system (CNS)-resident natural killer cells suppress Th17 responses and CNS autoimmune pathology.CLEC5A regulates Japanese encephalitis virus-induced neuroinflammation and lethality.Cell depletion in mice that express diphtheria toxin receptor under the control of SiglecH encompasses more than plasmacytoid dendritic cellsFlow cytometry protocols for surface and intracellular antigen analyses of neural cell typesAttenuation of CNS inflammatory responses by nicotine involves α7 and non-α7 nicotinic receptorsNeurofibromatosis-1 heterozygosity increases microglia in a spatially and temporally restricted pattern relevant to mouse optic glioma formation and growth.A neurodegeneration-specific gene-expression signature of acutely isolated microglia from an amyotrophic lateral sclerosis mouse modelInterleukin-2/interleukin-2 antibody therapy induces target organ natural killer cells that inhibit central nervous system inflammation.CCR2 antagonism alters brain macrophage polarization and ameliorates cognitive dysfunction induced by traumatic brain injuryGranulocyte colony stimulating factor decreases brain amyloid burden and reverses cognitive impairment in Alzheimer's mice.The prostaglandin E2 E-prostanoid 4 receptor exerts anti-inflammatory effects in brain innate immunityF11R is a novel monocyte prognostic biomarker for malignant gliomaCranial irradiation alters the brain's microenvironment and permits CCR2+ macrophage infiltration.Nuclear receptors license phagocytosis by trem2+ myeloid cells in mouse models of Alzheimer's disease.The fractalkine receptor but not CCR2 is present on microglia from embryonic development throughout adulthood.Thrombin-processed Ecrg4 recruits myeloid cells and induces antitumorigenic inflammationIsolation of brain and spinal cord mononuclear cells using percoll gradients.Bone marrow transplantation augments the effect of brain- and spinal cord-directed adeno-associated virus 2/5 gene therapy by altering inflammation in the murine model of globoid-cell leukodystrophy.Toll/interleukin-1 receptor domain-containing adapter inducing interferon-β mediates microglial phagocytosis of degenerating axons.CD38 deficiency in the tumor microenvironment attenuates glioma progression and modulates features of tumor-associated microglia/macrophages.Microglia dictate the impact of saturated fat consumption on hypothalamic inflammation and neuronal function.Disruption of Fractalkine Signaling Leads to Microglial Activation and Neuronal Damage in the Diabetic Retina.Non-cell-autonomous effects of presenilin 1 variants on enrichment-mediated hippocampal progenitor cell proliferation and differentiation.Isolation of highly enriched primary human microglia for functional studies.Bone marrow transplantation confers modest benefits in mouse models of Huntington's disease.Isolation and Flow Cytometric Analysis of Immune Cells from the Ischemic Mouse BrainAge exacerbates the CCR2/5-mediated neuroinflammatory response to traumatic brain injury.Untangling the brain's neuroinflammatory and neurodegenerative transcriptional responses.Glucocorticoid signaling in myeloid cells worsens acute CNS injury and inflammation.
P2860
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P2860
Isolation of murine microglial cells for RNA analysis or flow cytometry.
description
2006 nî lūn-bûn
@nan
2006 թուականի Յունուարին հրատարակուած գիտական յօդուած
@hyw
2006 թվականի հունվարին հրատարակված գիտական հոդված
@hy
2006年の論文
@ja
2006年論文
@yue
2006年論文
@zh-hant
2006年論文
@zh-hk
2006年論文
@zh-mo
2006年論文
@zh-tw
2006年论文
@wuu
name
Isolation of murine microglial cells for RNA analysis or flow cytometry.
@ast
Isolation of murine microglial cells for RNA analysis or flow cytometry.
@en
Isolation of murine microglial cells for RNA analysis or flow cytometry.
@nl
type
label
Isolation of murine microglial cells for RNA analysis or flow cytometry.
@ast
Isolation of murine microglial cells for RNA analysis or flow cytometry.
@en
Isolation of murine microglial cells for RNA analysis or flow cytometry.
@nl
prefLabel
Isolation of murine microglial cells for RNA analysis or flow cytometry.
@ast
Isolation of murine microglial cells for RNA analysis or flow cytometry.
@en
Isolation of murine microglial cells for RNA analysis or flow cytometry.
@nl
P2093
P2860
P356
P1433
P1476
Isolation of murine microglial cells for RNA analysis or flow cytometry.
@en
P2093
DeRen Huang
Margaret E Sasse
Richard M Ransohoff
P2860
P2888
P304
P356
10.1038/NPROT.2006.327
P577
2006-01-01T00:00:00Z
P6179
1026108982