Cerebellar gene expression profiles of mouse models for Rett syndrome reveal novel MeCP2 targets. - Wikidata - wikimedia - TriplyDB

Cerebellar gene expression profiles of mouse models for Rett syndrome reveal novel MeCP2 targets.

Cerebellar gene expression profiles of mouse models for Rett syndrome reveal novel MeCP2 targets.

http://www.wikidata.org/entity/Q33288477

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DLX5 and DLX6 expression is biallelic and not modulated by MeCP2 deficiency Developmental Dynamics of Rett Syndrome In Sickness and in Health: Perineuronal Nets and Synaptic Plasticity in Psychiatric Disorders BDNF deregulation in Rett syndrome Genetic modifiers of MeCP2 function in Drosophila.Microglia as a critical player in both developmental and late-life CNS pathologies.Phosphorylation of MeCP2 at Serine 80 regulates its chromatin association and neurological function.Induced chromosome deletion in a Williams-Beuren syndrome mouse model causes cardiovascular abnormalities.Mecp2-null mice provide new neuronal targets for Rett syndrome.Neonatal maternal deprivation response and developmental changes in gene expression revealed by hypothalamic gene expression profiling in mice.Improved MECP2 Gene Therapy Extends the Survival of MeCP2-Null Mice without Apparent Toxicity after Intracisternal Delivery.Global transcriptional and translational repression in human-embryonic-stem-cell-derived Rett syndrome neurons.MeCP2 mutation results in compartment-specific reductions in dendritic branching and spine density in layer 5 motor cortical neurons of YFP-H mice.GluD1 is a common altered player in neuronal differentiation from both MECP2-mutated and CDKL5-mutated iPS cells.Phospholemman: a novel cardiac stress protein.Disruption of DNA-methylation-dependent long gene repression in Rett syndrome.Setdb1-mediated histone H3K9 hypermethylation in neurons worsens the neurological phenotype of Mecp2-deficient mice.Fetal alcohol exposure alters proopiomelanocortin gene expression and hypothalamic-pituitary-adrenal axis function via increasing MeCP2 expression in the hypothalamus.The role of MeCP2 in CNS development and function.Complexities of Rett syndrome and MeCP2 Neuronal MeCP2 is expressed at near histone-octamer levels and globally alters the chromatin state.The protocadherins, PCDHB1 and PCDH7, are regulated by MeCP2 in neuronal cells and brain tissues: implication for pathogenesis of Rett syndrome.Brain region-specific expression of Fxyd1, an Mecp2 target gene, is regulated by epigenetic mechanisms.Synaptic dysfunction in neurodevelopmental disorders associated with autism and intellectual disabilities.MECP2 Is a Frequently Amplified Oncogene with a Novel Epigenetic Mechanism That Mimics the Role of Activated RAS in Malignancy Linking epigenetics to human disease and Rett syndrome: the emerging novel and challenging concepts in MeCP2 research.Effects of Mecp2 loss of function in embryonic cortical neurons: a bioinformatics strategy to sort out non-neuronal cells variability from transcriptome profiling.Depression in pregnancy, infant birth weight and DNA methylation of imprint regulatory elements Correcting deregulated Fxyd1 expression ameliorates a behavioral impairment in a mouse model of Rett syndrome.MeCP2 modulates gene expression pathways in astrocytes Impaired in vivo binding of MeCP2 to chromatin in the absence of its DNA methyl-binding domain MECP2 genomic structure and function: insights from ENCODE Breathing dysfunction in Rett syndrome: understanding epigenetic regulation of the respiratory network Mouse models of MeCP2 disorders share gene expression changes in the cerebellum and hypothalamus.Cell cloning-based transcriptome analysis in Rett patients: relevance to the pathogenesis of Rett syndrome of new human MeCP2 target genes.DNA methylation and methyl-CpG binding proteins: developmental requirements and function.MECP2 promotes the growth of gastric cancer cells by suppressing miR-338-mediated antiproliferative effect.Enhanced dense core granule function and adrenal hypersecretion in a mouse model of Rett syndrome.Negative Allosteric Modulation of mGluR5 Partially Corrects Pathophysiology in a Mouse Model of Rett Syndrome.Promoter-like sequences regulating transcriptional activity in neurexin and neuroligin genes

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Cerebellar gene expression profiles of mouse models for Rett syndrome reveal novel MeCP2 targets.

http://www.wikidata.org/entity/Q33288477

description

2007 nî lūn-bûn

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2007 թուականի Յունիսին հրատարակուած գիտական յօդուած

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2007 թվականի հունիսին հրատարակված գիտական հոդված

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2007年の論文

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Cerebellar gene expression pro ...... me reveal novel MeCP2 targets.

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Cerebellar gene expression pro ...... me reveal novel MeCP2 targets.

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Cerebellar gene expression pro ...... me reveal novel MeCP2 targets.

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Cerebellar gene expression pro ...... me reveal novel MeCP2 targets.

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Cerebellar gene expression pro ...... me reveal novel MeCP2 targets.

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ChaRandle Jordan

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Helen C Kwan

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Hong Hua Li

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Uta Francke

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P2860

Reeler/Disabled-like disruption of neuronal migration in knockout mice lacking the VLDL receptor and ApoE receptor 2

Rett syndrome is caused by mutations in X-linked MECP2, encoding methyl-CpG-binding protein 2

Brahma links the SWI/SNF chromatin-remodeling complex with MeCP2-dependent transcriptional silencing

A tissue-specific MAR/SAR DNA-binding protein with unusual binding site recognition

Reelin and glutamic acid decarboxylase67 promoter remodeling in an epigenetic methionine-induced mouse model of schizophrenia

On the epigenetic regulation of the human reelin promoter

Expression of MeCP2 in postmitotic neurons rescues Rett syndrome in mice

Significance analysis of microarrays applied to the ionizing radiation response

Cluster analysis and display of genome-wide expression patterns

Histone hyperacetylation induces demethylation of reelin and 67-kDa glutamic acid decarboxylase promoters

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