Host transcript accumulation during lytic KSHV infection reveals several classes of host responses
about
An integrated approach to elucidate the intra-viral and viral-cellular protein interaction networks of a gamma-herpesvirusShutoff of Host Gene Expression in Influenza A Virus and Herpesviruses: Similar Mechanisms and Common ThemesA ribonucleoprotein complex protects the interleukin-6 mRNA from degradation by distinct herpesviral endonucleasesProgressive accumulation of activated ERK2 within highly stable ORF45-containing nuclear complexes promotes lytic gammaherpesvirus infectionAberrant herpesvirus-induced polyadenylation correlates with cellular messenger RNA destruction.Epstein-Barr virus DNase (BGLF5) induces genomic instability in human epithelial cells.KSHV and the pathogenesis of Kaposi sarcoma: listening to human biology and medicine.Deep sequencing reveals direct targets of gammaherpesvirus-induced mRNA decay and suggests that multiple mechanisms govern cellular transcript escape.The lytic transcriptome of Kaposi's sarcoma-associated herpesvirus reveals extensive transcription of noncoding regions, including regions antisense to important genes.A viral nuclear noncoding RNA binds re-localized poly(A) binding protein and is required for late KSHV gene expression.Red1 promotes the elimination of meiosis-specific mRNAs in vegetatively growing fission yeast.Transcriptome-Wide Cleavage Site Mapping on Cellular mRNAs Reveals Features Underlying Sequence-Specific Cleavage by the Viral Ribonuclease SOX.The "Bridge" in the Epstein-Barr virus alkaline exonuclease protein BGLF5 contributes to shutoff activity during productive infection.Regulation of the retinoblastoma proteins by the human herpesviruses.Divergent MicroRNA targetomes of closely related circulating strains of a polyomavirus.Comprehensive mapping and analysis of Kaposi's sarcoma-associated herpesvirus 3' UTRs identify differential posttranscriptional control of gene expression in lytic versus latent infection.The role of Kaposi sarcoma-associated herpesvirus in the pathogenesis of Kaposi sarcoma.An RNA element in human interleukin 6 confers escape from degradation by the gammaherpesvirus SOX protein.Nuclease escape elements protect messenger RNA against cleavage by multiple viral endonucleases.Kaposi's sarcoma-associated herpesvirus G-protein-coupled receptor prevents AU-rich-element-mediated mRNA decay.Raman tweezers provide the fingerprint of cells supporting the late stages of KSHV reactivation.Viral and cellular N(6)-methyladenosine and N(6),2'-O-dimethyladenosine epitranscriptomes in the KSHV life cycle.
P2860
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P2860
Host transcript accumulation during lytic KSHV infection reveals several classes of host responses
description
2007 nî lūn-bûn
@nan
2007 թուականի Օգոստոսին հրատարակուած գիտական յօդուած
@hyw
2007 թվականի օգոստոսին հրատարակված գիտական հոդված
@hy
2007年の論文
@ja
2007年学术文章
@wuu
2007年学术文章
@zh-cn
2007年学术文章
@zh-hans
2007年学术文章
@zh-my
2007年学术文章
@zh-sg
2007年學術文章
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name
Host transcript accumulation d ...... eral classes of host responses
@ast
Host transcript accumulation d ...... eral classes of host responses
@en
Host transcript accumulation d ...... ral classes of host responses.
@nl
type
label
Host transcript accumulation d ...... eral classes of host responses
@ast
Host transcript accumulation d ...... eral classes of host responses
@en
Host transcript accumulation d ...... ral classes of host responses.
@nl
prefLabel
Host transcript accumulation d ...... eral classes of host responses
@ast
Host transcript accumulation d ...... eral classes of host responses
@en
Host transcript accumulation d ...... ral classes of host responses.
@nl
P2860
P1433
P1476
Host transcript accumulation d ...... eral classes of host responses
@en
P2093
P2860
P356
10.1371/JOURNAL.PONE.0000811
P407
P577
2007-08-29T00:00:00Z