Specificity of RGS10A as a key component in the RANKL signaling mechanism for osteoclast differentiation
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Cellular deficiency in the RGS10 protein facilitates chemoresistant ovarian cancerDownregulation of both gene expression and activity of Hsp27 improved maturation of mouse oocyte in vitroRNAi-mediated silencing of Atp6i and Atp6i haploinsufficiency prevents both bone loss and inflammation in a mouse model of periodontal diseaseAtp6v0d2 is an essential component of the osteoclast-specific proton pump that mediates extracellular acidification in bone resorption.Targeting Atp6v1c1 Prevents Inflammation and Bone Erosion Caused by Periodontitis and Reveals Its Critical Function in OsteoimmunologySignaling networks that control the lineage commitment and differentiation of bone cells.Ac45 silencing mediated by AAV-sh-Ac45-RNAi prevents both bone loss and inflammation caused by periodontitis.Collagen XXIV (Col24α1) promotes osteoblastic differentiation and mineralization through TGF-β/Smads signaling pathway.IL-1R/TLR2 through MyD88 Divergently Modulates Osteoclastogenesis through Regulation of Nuclear Factor of Activated T Cells c1 (NFATc1) and B Lymphocyte-induced Maturation Protein-1 (Blimp1).Functions of RANKL/RANK/OPG in bone modeling and remodeling.Role of Regulators of G Protein Signaling Proteins in Bone Physiology and Pathophysiology.RNA interference-mediated silencing of Atp6i prevents both periapical bone erosion and inflammation in the mouse model of endodontic diseaseAtp6v1c1 may regulate filament actin arrangement in breast cancer cells.Role of regulator of G protein signaling proteins in bone.Inhibition of Rgs10 Expression Prevents Immune Cell Infiltration in Bacteria-induced Inflammatory Lesions and Osteoclast-mediated Bone Destruction.Alveolar bone loss: mechanisms, potential therapeutic targets, and interventions.Galactosemia, a single gene disorder with epigenetic consequences.Osteoclast proton pump regulator Atp6v1c1 enhances breast cancer growth by activating the mTORC1 pathway and bone metastasis by increasing V-ATPase activity.Atp6v1c1 is an essential component of the osteoclast proton pump and in F-actin ring formation in osteoclasts.MSRA polymorphism is associated with the risk of rheumatoid arthritis in a Chinese population.
P2860
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P2860
Specificity of RGS10A as a key component in the RANKL signaling mechanism for osteoclast differentiation
description
2007 nî lūn-bûn
@nan
2007 թուականի Հոկտեմբերին հրատարակուած գիտական յօդուած
@hyw
2007 թվականի հոտեմբերին հրատարակված գիտական հոդված
@hy
2007年の論文
@ja
2007年論文
@yue
2007年論文
@zh-hant
2007年論文
@zh-hk
2007年論文
@zh-mo
2007年論文
@zh-tw
2007年论文
@wuu
name
Specificity of RGS10A as a key ...... for osteoclast differentiation
@ast
Specificity of RGS10A as a key ...... for osteoclast differentiation
@en
Specificity of RGS10A as a key ...... or osteoclast differentiation.
@nl
type
label
Specificity of RGS10A as a key ...... for osteoclast differentiation
@ast
Specificity of RGS10A as a key ...... for osteoclast differentiation
@en
Specificity of RGS10A as a key ...... or osteoclast differentiation.
@nl
prefLabel
Specificity of RGS10A as a key ...... for osteoclast differentiation
@ast
Specificity of RGS10A as a key ...... for osteoclast differentiation
@en
Specificity of RGS10A as a key ...... or osteoclast differentiation.
@nl
P2093
P2860
P356
P1476
Specificity of RGS10A as a key ...... for osteoclast differentiation
@en
P2093
Philip Stashenko
Shuying Yang
Yi-Ping Li
P2860
P304
P356
10.1242/JCS.008300
P407
P577
2007-10-01T00:00:00Z