Siglecg limits the size of B1a B cell lineage by down-regulating NFkappaB activation
about
CD24-Siglec G/10 discriminates danger- from pathogen-associated molecular patternsSiglecs and immune regulationAntigen delivery to macrophages using liposomal nanoparticles targeting sialoadhesin/CD169Decoration of T-independent antigen with ligands for CD22 and Siglec-G can suppress immunity and induce B cell tolerance in vivoSiglec-G-CD24 axis controls the severity of graft-versus-host disease in mice.Amelioration of sepsis by inhibiting sialidase-mediated disruption of the CD24-SiglecG interaction.Siglec-G/10 in self-nonself discrimination of innate and adaptive immunity.Sialoside-based pattern recognitions discriminating infections from tissue injuries.Inhibition of NF-κB signaling retards eosinophilic dermatitis in SHARPIN-deficient mice.B-1 cell lymphoma in mice lacking the steroid and xenobiotic receptor, SXR.Manipulation of the glycan-specific natural antibody repertoire for immunotherapyThe steroid and xenobiotic receptor negatively regulates B-1 cell development in the fetal liver.IL-15 temporally reorients IL-10 biased B-1a cells toward IL-12 expression.Copresentation of antigen and ligands of Siglec-G induces B cell tolerance independent of CD22.Peripheral CD4(+) T-cell tolerance is induced in vivo by rare antigen-bearing B cells in follicular, marginal zone, and B-1 subsetsCD24 and Siglec-10 selectively repress tissue damage-induced immune responses.Evolution of CD33-related siglecs: regulating host immune functions and escaping pathogen exploitation?CD22 and Siglec-G regulate inhibition of B-cell signaling by sialic acid ligand binding and control B-cell tolerance.Siglec-G is a B-1 cell inhibitory receptor and also controls B cell tolerance.Siglec-G represses DAMP-mediated effects on T cells.Sialic Acid-Binding Immunoglobulin-like Lectin G Promotes Atherosclerosis and Liver Inflammation by Suppressing the Protective Functions of B-1 Cells.Siglec-E is up-regulated and phosphorylated following lipopolysaccharide stimulation in order to limit TLR-driven cytokine production.The lectin Siglec-G inhibits dendritic cell cross-presentation by impairing MHC class I-peptide complex formation.SIGLEC-G deficiency increases susceptibility to develop B-cell lymphoproliferative disorders.Regulation of B cell functions by the sialic acid-binding receptors siglec-G and CD22.Accumulation of B1-like B cells in transgenic mice over-expressing catalytically inactive RAG1 in the periphery.
P2860
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P2860
Siglecg limits the size of B1a B cell lineage by down-regulating NFkappaB activation
description
2007 nî lūn-bûn
@nan
2007 թուականի Հոկտեմբերին հրատարակուած գիտական յօդուած
@hyw
2007 թվականի հոտեմբերին հրատարակված գիտական հոդված
@hy
2007年の論文
@ja
2007年論文
@yue
2007年論文
@zh-hant
2007年論文
@zh-hk
2007年論文
@zh-mo
2007年論文
@zh-tw
2007年论文
@wuu
name
Siglecg limits the size of B1a B cell lineage by down-regulating NFkappaB activation
@ast
Siglecg limits the size of B1a B cell lineage by down-regulating NFkappaB activation
@en
Siglecg limits the size of B1a B cell lineage by down-regulating NFkappaB activation.
@nl
type
label
Siglecg limits the size of B1a B cell lineage by down-regulating NFkappaB activation
@ast
Siglecg limits the size of B1a B cell lineage by down-regulating NFkappaB activation
@en
Siglecg limits the size of B1a B cell lineage by down-regulating NFkappaB activation.
@nl
prefLabel
Siglecg limits the size of B1a B cell lineage by down-regulating NFkappaB activation
@ast
Siglecg limits the size of B1a B cell lineage by down-regulating NFkappaB activation
@en
Siglecg limits the size of B1a B cell lineage by down-regulating NFkappaB activation.
@nl
P2093
P2860
P1433
P1476
Siglecg limits the size of B1a B cell lineage by down-regulating NFkappaB activation
@en
P2093
Bae Keun Park
Cheng Ding
Cun-Yu Wang
P2860
P356
10.1371/JOURNAL.PONE.0000997
P407
P577
2007-10-03T00:00:00Z