Increased hemorrhagic transformation and altered infarct size and localization after experimental stroke in a rat model type 2 diabetes. - Wikidata - wikimedia - TriplyDB

Increased hemorrhagic transformation and altered infarct size and localization after experimental stroke in a rat model type 2 diabetes.

Increased hemorrhagic transformation and altered infarct size and localization after experimental stroke in a rat model type 2 diabetes.

http://www.wikidata.org/entity/Q33302694

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Recommendations for preclinical research in hemorrhagic transformation Obesity and stroke: Can we translate from rodents to patients?Diabetes Mellitus and Blood-Brain Barrier Dysfunction: An Overview Acute treatment with rosuvastatin protects insulin resistant (C57BL/6J ob/ob) mice against transient cerebral ischemia.Adaptive cerebral neovascularization in a model of type 2 diabetes: relevance to focal cerebral ischemia.Hyperglycemia, acute ischemic stroke, and thrombolytic therapy Diabetes mellitus aggravates hemorrhagic transformation after ischemic stroke via mitochondrial defects leading to endothelial apoptosis.Different strokes for different folks: the rich diversity of animal models of focal cerebral ischemia Vascular protection in diabetic stroke: role of matrix metalloprotease-dependent vascular remodeling Exacerbated brain damage, edema and inflammation in type-2 diabetic mice subjected to focal ischemia.Angiopoietin/Tie2 pathway mediates type 2 diabetes induced vascular damage after cerebral stroke Persistent cerebrovascular damage after stroke in type two diabetic rats measured by magnetic resonance imaging.Neurovascular injury in acute hyperglycemia and diabetes: A comparative analysis in experimental stroke.Metformin treatment in the period after stroke prevents nitrative stress and restores angiogenic signaling in the brain in diabetes.Comparative Analysis of Different Methods of Ischemia/Reperfusion in Hyperglycemic Stroke Outcomes: Interaction with tPA.Niaspan enhances vascular remodeling after stroke in type 1 diabetic rats.Sex-independent neuroprotection with minocycline after experimental thromboembolic stroke.Cell Treatment for Stroke in Type Two Diabetic Rats Improves Vascular Permeability Measured by MRI.Enhanced cerebral but not peripheral angiogenesis in the Goto-Kakizaki model of type 2 diabetes involves VEGF and peroxynitrite signaling.Twisted blood vessels: symptoms, etiology and biomechanical mechanisms Angiogenesis: a harmonized target for recovery after stroke.Mechanisms of acute neurovascular protection with AT1 blockade after stroke: Effect of prestroke hypertension.Obesity and hyperglycemia lead to impaired post-ischemic recovery after permanent ischemia in mice.Matrix Metalloprotease 3 Exacerbates Hemorrhagic Transformation and Worsens Functional Outcomes in Hyperglycemic Stroke Cerebral neovascularization and remodeling patterns in two different models of type 2 diabetes.Targets of vascular protection in acute ischemic stroke differ in type 2 diabetes.Early effects of high-fat diet on neurovascular function and focal ischemic brain injury.Hyperglycemia, diabetes and stroke: focus on the cerebrovasculature.Cerebrovascular complications of diabetes: focus on stroke.The effect of endothelin receptor A antagonism on basilar artery endothelium-dependent relaxation after ischemic stroke MRI-based selection for intra-arterial stroke therapy: value of pretreatment diffusion-weighted imaging lesion volume in selecting patients with acute stroke who will benefit from early recanalization.Comparative analysis of the neurovascular injury and functional outcomes in experimental stroke models in diabetic Goto-Kakizaki rats.Diet-induced obesity causes cerebral vessel remodeling and increases the damage caused by ischemic stroke.The influence of stroke risk factors and comorbidities on assessment of stroke therapies in humans and animals.Cerebral neovascularization in diabetes: implications for stroke recovery and beyond.Impact of Comorbidities on Acute Injury and Recovery in Preclinical Stroke Research: Focus on Hypertension and Diabetes.Diabetes Worsens Functional Outcomes in Young Female Rats: Comparison of Stroke Models, Tissue Plasminogen Activator Effects, and Sexes.Cerebrovascular complications of diabetes: focus on cognitive dysfunction Diabetic aggravation of stroke and animal models.Brain injury with diabetes mellitus: evidence, mechanisms and treatment implications.

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Increased hemorrhagic transformation and altered infarct size and localization after experimental stroke in a rat model type 2 diabetes.

http://www.wikidata.org/entity/Q33302694

description

2007 nî lūn-bûn

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2007 թուականի Հոկտեմբերին հրատարակուած գիտական յօդուած

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2007 թվականի հոտեմբերին հրատարակված գիտական հոդված

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2007年の論文

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Increased hemorrhagic transfor ...... n a rat model type 2 diabetes.

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Adviye Ergul

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Anna Kozak

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Christiana Hall

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Hazem Elewa

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Jeffrey A Switzer

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P2860

Effect of neutrophil depletion on gelatinase expression, edema formation and hemorrhagic transformation after focal ischemic stroke.

Regional metabolic disturbances and cerebrovascular anatomy after permanent middle cerebral artery occlusion in C57black/6 and SV129 mice.

Vascular endothelial growth factor and diabetic retinopathy: role of oxidative stress.

Stress hyperglycemia and prognosis of stroke in nondiabetic and diabetic patients: a systematic overview.

Cerebral ischemic preconditioning. An experimental phenomenon or a clinical important entity of stroke prevention?

Cerebral microvessel responses to focal ischemia.

Diabetes and cardiovascular disease: the road to cardioprotection.

Nitric oxide and cerebral ischemic preconditioning.

Targets for vascular protection after acute ischemic stroke.

Hyperglycemia in acute ischemic stroke: a vascular perspective.

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