Increased endothelial expression of Toll-like receptor 2 at sites of disturbed blood flow exacerbates early atherogenic events. - Wikidata - wikimedia - TriplyDB

Increased endothelial expression of Toll-like receptor 2 at sites of disturbed blood flow exacerbates early atherogenic events.

Increased endothelial expression of Toll-like receptor 2 at sites of disturbed blood flow exacerbates early atherogenic events.

http://www.wikidata.org/entity/Q33318184

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Unexpected protective role for Toll-like receptor 3 in the arterial wall Regulated accumulation of desmosterol integrates macrophage lipid metabolism and inflammatory responses CD36 ligands promote sterile inflammation through assembly of a Toll-like receptor 4 and 6 heterodimer Endothelial dysfunction: the role of sterol regulatory element-binding protein-induced NOD-like receptor family pyrin domain-containing protein 3 inflammasome in atherosclerosis Stiffening-induced high pulsatility flow activates endothelial inflammation via a TLR2/NF-κB pathway Cholesterol-induced non-alcoholic fatty liver disease and atherosclerosis aggravated by systemic inflammation The Role of TLR2, TLR4, and TLR9 in the Pathogenesis of Atherosclerosis Cholesterol, inflammation and innate immunity Inflammatory cell recruitment in cardiovascular disease: murine models and potential clinical applications Recent insights into the cellular biology of atherosclerosis Absence of p55 TNF receptor reduces atherosclerosis, but has no major effect on angiotensin II induced aneurysms in LDL receptor deficient mice Dendritic cells in atherosclerotic disease.Vascular lipid accumulation, lipoprotein oxidation, and macrophage lipid uptake in hypercholesterolemic zebrafish.Mechanisms that regulate macrophage burden in atherosclerosis.Toll-like receptor signaling: a potential link among rheumatoid arthritis, systemic lupus, and atherosclerosis.Effects of disturbed flow on vascular endothelium: pathophysiological basis and clinical perspectives Altered in vitro endothelial repair and monocyte migration in obstructive sleep apnea: implication of VEGF and CRP.Atherogenic lipids and lipoproteins trigger CD36-TLR2-dependent apoptosis in macrophages undergoing endoplasmic reticulum stress.Role of toll-like receptor 4 in intimal foam cell accumulation in apolipoprotein E-deficient mice Oxidized low density lipoprotein induces bone morphogenetic protein-2 in coronary artery endothelial cells via Toll-like receptors 2 and 4.TLR2-deficiency of cKit+ bone marrow cells is associated with augmented potency to stimulate angiogenic processes.Spontaneously diabetic Ins2(+/Akita):apoE-deficient mice exhibit exaggerated hypercholesterolemia and atherosclerosis An LXR-NCOA5 gene regulatory complex directs inflammatory crosstalk-dependent repression of macrophage cholesterol efflux.Sparstolonin B suppresses rat vascular smooth muscle cell proliferation, migration, inflammatory response and lipid accumulation.The critical role of IL-1 receptor-associated kinase 4-mediated NF-κB activation in modified low-density lipoprotein-induced inflammatory gene expression and atherosclerosis.TLR2 and neutrophils potentiate endothelial stress, apoptosis and detachment: implications for superficial erosion.Disruption of ldlr causes increased LDL-c and vascular lipid accumulation in a zebrafish model of hypercholesterolemia.Atherosclerosis induced by endogenous and exogenous toll-like receptor (TLR)1 or TLR6 agonists.Endothelial expression of guidance cues in vessel wall homeostasis dysregulation under proatherosclerotic conditions.Macrophage-activating lipopeptide-2 downregulates the expression of ATP-binding cassette transporter A1 by activating the TLR2/NF-кB/ZNF202 pathway in THP-1 macrophages.Inflammation and endothelial dysfunction during aging: role of NF-kappaB The interleukin-1 receptor associated kinase 1 contributes to the regulation of NFAT Toll-like receptors in atherosclerosis Immune effector mechanisms implicated in atherosclerosis: from mice to humans.Identification of antigen-presenting dendritic cells in mouse aorta and cardiac valves.Emerging role of Toll-like receptors in atherosclerosis.Innate immunity and toll-like receptor antagonists: a potential role in the treatment of cardiovascular diseases.Host cholesterol and inflammation as common key regulators of toxoplasmosis and artherosclerosis development.Toll-like receptor-4 and lipoprotein accumulation in macrophages Blocking TLR2 activity diminishes and stabilizes advanced atherosclerotic lesions in apolipoprotein E-deficient mice.

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Increased endothelial expression of Toll-like receptor 2 at sites of disturbed blood flow exacerbates early atherogenic events.

http://www.wikidata.org/entity/Q33318184

description

2008 nî lūn-bûn

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2008 թուականի Փետրուարին հրատարակուած գիտական յօդուած

@hyw

2008 թվականի փետրվարին հրատարակված գիտական հոդված

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2008年の論文

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2008年論文

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2008年論文

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name

Increased endothelial expressi ...... ates early atherogenic events.

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Increased endothelial expressi ...... ates early atherogenic events.

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Increased endothelial expressi ...... ates early atherogenic events.

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Increased endothelial expressi ...... ates early atherogenic events.

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Increased endothelial expressi ...... ates early atherogenic events.

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Increased endothelial expressi ...... ates early atherogenic events.

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Increased endothelial expressi ...... ates early atherogenic events.

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Increased endothelial expressi ...... ates early atherogenic events.

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Increased endothelial expressi ...... ates early atherogenic events.

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Journal of Experimental Medicine

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Increased endothelial expressi ...... ates early atherogenic events.

@en

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Katrin Soldau

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Linda K Curtiss

http://www.w3.org/2001/XMLSchema#string

Peter S Tobias

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Thomas A Bell

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William B Kiosses

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P2860

Toll-like receptor signalling

Atherosclerosis — An Inflammatory Disease

Atherogenesis in perspective: hypercholesterolemia and inflammation as partners in crime

Toll-like receptors and innate immunity

Renal-associated TLR2 mediates ischemia/reperfusion injury in the kidney

Modulation of atherosclerosis in mice by Toll-like receptor 2

TLR2 is constitutively expressed within the kidney and participates in ischemic renal injury through both MyD88-dependent and -independent pathways.

The NF-kappa B signal transduction pathway in aortic endothelial cells is primed for activation in regions predisposed to atherosclerotic lesion formation.

Site specificity of atherosclerosis: site-selective responses to atherosclerotic modulators.

Nile red: a selective fluorescent stain for intracellular lipid droplets.

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2

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205

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5602576

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18250194

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atherosclerosis

Toll-like receptor

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2271019

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