UVA1 genotoxicity is mediated not by oxidative damage but by cyclobutane pyrimidine dimers in normal mouse skin. - Wikidata - wikimedia - TriplyDB

UVA1 genotoxicity is mediated not by oxidative damage but by cyclobutane pyrimidine dimers in normal mouse skin.

UVA1 genotoxicity is mediated not by oxidative damage but by cyclobutane pyrimidine dimers in normal mouse skin.

http://www.wikidata.org/entity/Q33324825

about

The mechanisms of UV mutagenesis.Aging and chronic sun exposure cause distinct epigenetic changes in human skin.Unravelling UVA-induced mutagenesis.Comparison of DNA damage responses following equimutagenic doses of UVA and UVB: a less effective cell cycle arrest with UVA may render UVA-induced pyrimidine dimers more mutagenic than UVB-induced ones.A life history perspective on skin cancer and the evolution of skin pigmentation.Analysis of CPD ultraviolet lesion bypass in chicken DT40 cells: polymerase η and PCNA ubiquitylation play identical roles Solar-UV-signature mutation prefers TCG to CCG: extrapolative consideration from UVA1-induced mutation spectra in mouse skin.DNA damage as a biological sensor for environmental sunlight.UV signature mutations.Patterns and processes of somatic mutations in nine major cancers.Cyclobutane Pyrimidine Dimer Density as a Predictive Biomarker of the Biological Effects of Ultraviolet Radiation in Normal Human Fibroblast UV wavelength-dependent DNA damage and human non-melanoma and melanoma skin cancer.The photobiology of melanocytes modulates the impact of UVA on sunlight-induced melanoma.Photoinduced damage to cellular DNA: direct and photosensitized reactions.The maximal cumulative solar UVB dose allowed to maintain healthy and young skin and prevent premature photoaging.Phosphoinositide 3-Kinase-Dependent Signalling Pathways in Cutaneous Squamous Cell Carcinomas.Somatic cell mutations caused by 365 nm LED-UVA due to DNA double-strand breaks through oxidative damage.Tissue-specific and time-dependent clonal expansion of ENU-induced mutant cells in gpt delta mice.Roles of UVA radiation and DNA damage responses in melanoma pathogenesis.Wavelength dependence of biological damage induced by UV radiation on bacteria UVA (UVA-1) therapy for the treatment of acute GVHD of the skin

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UVA1 genotoxicity is mediated not by oxidative damage but by cyclobutane pyrimidine dimers in normal mouse skin.

http://www.wikidata.org/entity/Q33324825

description

2008 nî lūn-bûn

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2008 թուականի Մարտին հրատարակուած գիտական յօդուած

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2008 թվականի մարտին հրատարակված գիտական հոդված

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2008年の論文

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2008年論文

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2008年論文

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2008年論文

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2008年論文

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2008年論文

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2008年论文

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UVA1 genotoxicity is mediated ...... e dimers in normal mouse skin.

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UVA1 genotoxicity is mediated ...... e dimers in normal mouse skin.

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UVA1 genotoxicity is mediated ...... e dimers in normal mouse skin.

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UVA1 genotoxicity is mediated ...... e dimers in normal mouse skin.

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UVA1 genotoxicity is mediated ...... e dimers in normal mouse skin.

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UVA1 genotoxicity is mediated ...... e dimers in normal mouse skin.

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P1433

Journal of Investigative Dermatology

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UVA1 genotoxicity is mediated ...... e dimers in normal mouse skin.

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Hiroshi Kasai

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Jun-ichiro Komura

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Kazuaki Kawai

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Kazuo Yamamoto

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Ko Sakatsume

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Kotaro Hieda

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Masakatsu Watanabe

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Masaru Imai

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Osamu Nikaido

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Shoichi Higashi

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2289-2296

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scholarly article

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10.1038/JID.2008.61

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9

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128

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Hironobu Ikehata

Liangcheng Wang

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2008-03-20T00:00:00Z

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5495273

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