Antagomir-17-5p abolishes the growth of therapy-resistant neuroblastoma through p21 and BIM - Wikidata - wikimedia - TriplyDB

Antagomir-17-5p abolishes the growth of therapy-resistant neuroblastoma through p21 and BIM

Antagomir-17-5p abolishes the growth of therapy-resistant neuroblastoma through p21 and BIM

http://www.wikidata.org/entity/Q33336561

about

Inhibition of microRNA function by antimiR oligonucleotides MicroRNA-184 inhibits neuroblastoma cell survival through targeting the serine/threonine kinase AKT2 The inhibition of the highly expressed miR-221 and miR-222 impairs the growth of prostate carcinoma xenografts in mice Nanoparticle-based therapy in an in vivo microRNA-155 (miR-155)-dependent mouse model of lymphoma Mechanism of human Hb switching: a possible role of the kit receptor/miR 221-222 complex Feud or Friend? The Role of the miR-17-92 Cluster in Tumorigenesis miR-9, a MYC/MYCN-activated microRNA, regulates E-cadherin and cancer metastasis.Germline deletion of the miR-17∼92 cluster causes skeletal and growth defects in humans Repression of the miR-17-92 cluster by p53 has an important function in hypoxia-induced apoptosis Genetic dissection of the miR-17~92 cluster of microRNAs in Myc-induced B-cell lymphomas The Quest for Targets Executing MYC-Dependent Cell Transformation Oncogenic MicroRNAs: Key Players in Malignant Transformation Chemoresistance, cancer stem cells, and miRNA influences: the case for neuroblastoma MicroRNAs in breast cancer initiation and progression Significance and therapeutic value of miRNAs in embryonal neural tumors MicroRNAs in the pathogenesis of cystic kidney disease Epigenetic deregulation of microRNAs in rhabdomyosarcoma and neuroblastoma and translational perspectives MicroRNA-regulated pathways in hematological malignancies: how to avoid cells playing out of tune The miR-17/92 cluster: a comprehensive update on its genomics, genetics, functions and increasingly important and numerous roles in health and disease Inhibition of neuroblastoma tumor growth by targeted delivery of microRNA-34a using anti-disialoganglioside GD2 coated nanoparticles Examination of the expanding pathways for the regulation of p21 expression and activity.miR-155: A Novel Target in Allergic Asthma The myc-miR-17~92 axis blunts TGF{beta} signaling and production of multiple TGF{beta}-dependent antiangiogenic factors The miR-17-92 cluster regulates FOG-2 expression and inhibits proliferation of mouse embryonic cardiomyocytes Analysis of the miRNA-mRNA-lncRNA networks in ER+ and ER- breast cancer cell lines Global MYCN transcription factor binding analysis in neuroblastoma reveals association with distinct E-box motifs and regions of DNA hypermethylation.Unveiling MYCN regulatory networks in neuroblastoma via integrative analysis of heterogeneous genomics data.The Effects of Antisense miRNA-20a Alone or in Combination with Imatinib on K562 Cell Proliferation Right into the heart of microRNA-133a.A 3'-untranslated region (3'UTR) induces organ adhesion by regulating miR-199a* functions.A novel and universal method for microRNA RT-qPCR data normalization.Widespread dysregulation of MiRNAs by MYCN amplification and chromosomal imbalances in neuroblastoma: association of miRNA expression with survival.miRNA expression profiling enables risk stratification in archived and fresh neuroblastoma tumor samples.Deep sequencing reveals differential expression of microRNAs in favorable versus unfavorable neuroblastoma.Micromanaging vascular biology: tiny microRNAs play big band MYCN-regulated microRNAs repress estrogen receptor-alpha (ESR1) expression and neuronal differentiation in human neuroblastoma Molecular characterization of aromatase inhibitor-resistant, tamoxifen-resistant and LTEDaro cell lines MicroRNA-19a mediates the suppressive effect of laminar flow on cyclin D1 expression in human umbilical vein endothelial cells.Imperfect centered miRNA binding sites are common and can mediate repression of target mRNAs Investigation gene and microRNA expression in glioblastoma.

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Antagomir-17-5p abolishes the growth of therapy-resistant neuroblastoma through p21 and BIM

http://www.wikidata.org/entity/Q33336561

description

2008 nî lūn-bûn

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2008 թուականի Մայիսին հրատարակուած գիտական յօդուած

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2008 թվականի մայիսին հրատարակված գիտական հոդված

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2008年の論文

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2008年論文

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2008年論文

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2008年論文

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2008年論文

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2008年論文

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2008年论文

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name

Antagomir-17-5p abolishes the ...... roblastoma through p21 and BIM

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Antagomir-17-5p abolishes the ...... roblastoma through p21 and BIM

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Antagomir-17-5p abolishes the ...... oblastoma through p21 and BIM.

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Antagomir-17-5p abolishes the ...... roblastoma through p21 and BIM

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Antagomir-17-5p abolishes the ...... roblastoma through p21 and BIM

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Antagomir-17-5p abolishes the ...... roblastoma through p21 and BIM

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Antagomir-17-5p abolishes the ...... roblastoma through p21 and BIM

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Antagomir-17-5p abolishes the ...... roblastoma through p21 and BIM

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Alberto Donfrancesco

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Cesare Peschle

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Laura Fontana

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Renata Boldrini

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Serena Giovinazzi

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Sonia Albini

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Valentina Federici

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P2860

Human MicroRNA targets

Differential targeting of prosurvival Bcl-2 proteins by their BH3-only ligands allows complementary apoptotic function

Apoptosis initiated when BH3 ligands engage multiple Bcl-2 homologs, not Bax or Bak

The p21 Cdk-interacting protein Cip1 is a potent inhibitor of G1 cyclin-dependent kinases

Bim: a novel member of the Bcl-2 family that promotes apoptosis

MicroRNA genes are transcribed by RNA polymerase II

Transgenic over-expression of the microRNA miR-17-92 cluster promotes proliferation and inhibits differentiation of lung epithelial progenitor cells

Prediction of mammalian microRNA targets

Combinatorial microRNA target predictions

A microRNA polycistron as a potential human oncogene

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e2236

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10.1371/JOURNAL.PONE.0002236

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5

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Patrizio Giacomini

Loredana Cifaldi

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2008-05-21T00:00:00Z

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5355946

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18493594

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2375057

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