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Induction of blood brain barrier tight junction protein alterations by CD8 T cells.

Induction of blood brain barrier tight junction protein alterations by CD8 T cells.

http://www.wikidata.org/entity/Q33363111

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Cerebral malaria: mysteries at the blood-brain barrier Perivascular Arrest of CD8+ T Cells Is a Signature of Experimental Cerebral Malaria Breakdown of the blood-brain barrier during tick-borne encephalitis in mice is not dependent on CD8+ T-cells Claudin-1 induced sealing of blood-brain barrier tight junctions ameliorates chronic experimental autoimmune encephalomyelitis Induction of microglia activation after infection with the non-neurotropic A/CA/04/2009 H1N1 influenza virus.Novel magnetic/ultrasound focusing system enhances nanoparticle drug delivery for glioma treatment.Imaging Plasmodium immunobiology in the liver, brain, and lung.Trafficking of immune cells in the central nervous system CD8 T cell-initiated vascular endothelial growth factor expression promotes central nervous system vascular permeability under neuroinflammatory conditions.Interferons and interferon regulatory factors in malaria Rapid formation of extended processes and engagement of Theiler's virus-infected neurons by CNS-infiltrating CD8 T cells.Contrasting roles for CD4 vs. CD8 T-cells in a murine model of virally induced "T1 black hole" formation.Progesterone and allopregnanolone attenuate blood-brain barrier dysfunction following permanent focal ischemia by regulating the expression of matrix metalloproteinases.Intratumoral IL-12 gene therapy results in the crosspriming of Tc1 cells reactive against tumor-associated stromal antigens.Quantitative trait loci analysis reveals candidate genes implicated in regulating functional deficit and CNS vascular permeability in CD8 T cell-initiated blood-brain barrier disruption Elevation of matrix metalloproteinase-9 level in cerebrospinal fluid of tick-borne encephalitis patients is associated with IgG extravassation and disease severity.Blood cells and endothelial barrier function.Perforin competent CD8 T cells are sufficient to cause immune-mediated blood-brain barrier disruption.Zonula occludens-1, occludin and E-cadherin expression and organization in salivary glands with Sjögren's syndrome.Activated Brain Endothelial Cells Cross-Present Malaria Antigen Engaging neuroscience to advance translational research in brain barrier biology.A hematopoietic contribution to microhemorrhage formation during antiviral CD8 T cell-initiated blood-brain barrier disruption.Viral disruption of the blood-brain barrier.CD8 T cell-initiated blood-brain barrier disruption is independent of neutrophil support Preserved vascular integrity and enhanced survival following neuropilin-1 inhibition in a mouse model of CD8 T cell-initiated CNS vascular permeability Novel 3D analysis of Claudin-5 reveals significant endothelial heterogeneity among CNS microvessels.Blocking neurogenic inflammation for the treatment of acute disorders of the central nervous system.Modulation of peripheral cytotoxic cells and ictogenesis in a model of seizures.Neuronal uptake of serum albumin is associated with neuron damage during the development of epilepsy Microbial induction of vascular pathology in the CNS.Modulatory effects of perforin gene dosage on pathogen-associated blood-brain barrier (BBB) disruption.Optical imaging to map blood-brain barrier leakage A new hypothesis on the manifestation of cerebral malaria: the secret is in the liver Theiler's murine encephalomyelitis virus as an experimental model system to study the mechanism of blood-brain barrier disruption.Targeting Angiotensin II Type-1 Receptor (AT1R) Inhibits the Harmful Phenotype of Plasmodium-Specific CD8+ T Cells during Blood-Stage Malaria.Peroxisome proliferator activating receptor (PPAR) in cerebral malaria (CM): a novel target for an additional therapy.How do immune cells overcome the blood-brain barrier in multiple sclerosis?Are you in or out? Leukocyte, ion, and neurotransmitter permeability across the epileptic blood-brain barrier.Immunobiology of Plasmodium in liver and brain.Host matrix metalloproteinases in cerebral malaria: new kids on the block against blood-brain barrier integrity?

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P2860

Induction of blood brain barrier tight junction protein alterations by CD8 T cells.

http://www.wikidata.org/entity/Q33363111

description

2008 nî lūn-bûn

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2008 թուականի Օգոստոսին հրատարակուած գիտական յօդուած

@hyw

2008 թվականի օգոստոսին հրատարակված գիտական հոդված

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2008年の論文

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2008年論文

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2008年論文

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2008年論文

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2008年論文

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2008年論文

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2008年论文

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name

Induction of blood brain barrier tight junction protein alterations by CD8 T cells.

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Induction of blood brain barrier tight junction protein alterations by CD8 T cells.

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Induction of blood brain barrier tight junction protein alterations by CD8 T cells.

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Induction of blood brain barrier tight junction protein alterations by CD8 T cells.

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Induction of blood brain barrier tight junction protein alterations by CD8 T cells.

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Induction of blood brain barrier tight junction protein alterations by CD8 T cells.

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Induction of blood brain barrier tight junction protein alterations by CD8 T cells.

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Aaron J Johnson

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Georgette L Suidan

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Istvan Pirko

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Jeremiah R Mcdole

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Yi Chen

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P2860

Hepatocyte growth factor/scatter factor decreases the expression of occludin and transendothelial resistance (TER) and increases paracellular permeability in human vascular endothelial cells

Size-selective loosening of the blood-brain barrier in claudin-5-deficient mice

Hepatocyte growth factor/scatter factor decreases the expression of occludin and transendothelial resistance (TER) and increases paracellular permeability in human vascular endothelial cells

Peptide mapping by limited proteolysis in sodium dodecyl sulfate and analysis by gel electrophoresis

The blood-brain barrier/neurovascular unit in health and disease

The blood-brain barrier: an overview: structure, regulation, and clinical implications

Original antigenic sin and apoptosis in the pathogenesis of dengue hemorrhagic fever

Inflammatory pain alters blood-brain barrier permeability and tight junctional protein expression.

Prevalent class I-restricted T-cell response to the Theiler's virus epitope Db:VP2121-130 in the absence of endogenous CD4 help, tumor necrosis factor alpha, gamma interferon, perforin, or costimulation through CD28.

Granzyme A, a noncytolytic component of CD8(+) cell granules, restricts the spread of herpes simplex virus in the peripheral nervous systems of experimentally infected mice.

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18725947

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blood–brain barrier

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