Shiga toxin-associated hemolytic uremic syndrome: combined cytotoxic effects of shiga toxin and lipopolysaccharide (endotoxin) on human vascular endothelial cells in vitro.
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Lipopolysaccharide from enterohemorrhagic Escherichia coli binds to platelets through TLR4 and CD62 and is detected on circulating platelets in patients with hemolytic uremic syndromeShiga toxin 2 and lipopolysaccharide induce human microvascular endothelial cells to release chemokines and factors that stimulate platelet function.Pathogenesis and diagnosis of Shiga toxin-producing Escherichia coli infectionsDNA fingerprinting of Shiga-toxin producing Escherichia coli O157 based on Multiple-Locus Variable-Number Tandem-Repeats Analysis (MLVA)Management of hemolytic-uremic syndrome in childrenInvestigation of encephalopathy caused by Shiga toxin 2c-producing Escherichia coli infection in mice.Dexamethasone rescues neurovascular unit integrity from cell damage caused by systemic administration of shiga toxin 2 and lipopolysaccharide in mice motor cortexInduction by sphingomyelinase of shiga toxin receptor and shiga toxin 2 sensitivity in human microvascular endothelial cells.Shiga toxin 2 causes apoptosis in human brain microvascular endothelial cells via C/EBP homologous protein.p38 mitogen-activated protein kinase mediates lipopolysaccharide and tumor necrosis factor alpha induction of shiga toxin 2 sensitivity in human umbilical vein endothelial cellsMonocyte chemoattractant protein 1, macrophage inflammatory protein 1 alpha, and RANTES recruit macrophages to the kidney in a mouse model of hemolytic-uremic syndromeDepletion of liver and splenic macrophages reduces the lethality of Shiga toxin-2 in a mouse model.Pretreatment of mice with lipopolysaccharide (LPS) or IL-1beta exerts dose-dependent opposite effects on Shiga toxin-2 lethality.Shiga toxin and lipopolysaccharide induce platelet-leukocyte aggregates and tissue factor release, a thrombotic mechanism in hemolytic uremic syndrome.Escherichia coli Shiga Toxin Mechanisms of Action in Renal Disease.Shiga toxin pathogenesis: kidney complications and renal failure.Complement activation associated with ADAMTS13 deficiency in human and murine thrombotic microangiopathyActivation of the Classical Mitogen-Activated Protein Kinases Is Part of the Shiga Toxin-Induced Ribotoxic Stress Response and May Contribute to Shiga Toxin-Induced Inflammation.Shiga toxin-associated hemolytic uremic syndrome: interleukin-1 beta enhancement of Shiga toxin cytotoxicity toward human vascular endothelial cells in vitroLipopolysaccharide-specific antibodies in plasma and stools of children with Shigella-associated leukemoid reaction and hemolytic-uremic syndromeComparison of the relative toxicities of Shiga-like toxins type I and type II for miceEnhancement of susceptibility to Shiga toxin-producing Escherichia coli O157:H7 by protein calorie malnutrition in mice.Role of nitric oxide in shiga toxin-2-induced premature delivery of dead fetuses in rats.The MAP kinase-activated protein kinase 2 (MK2) contributes to the Shiga toxin-induced inflammatory responseLocalization of potential binding sites for the edema disease verotoxin (VT2e) in pigsDirect evidence of neuron impairment by oral infection with verotoxin-producing Escherichia coli O157:H- in mitomycin-treated mice.Purified Shiga-like toxins induce expression of proinflammatory cytokines from murine peritoneal macrophages.Translocation of Shiga toxin across polarized intestinal cells in tissue culture.Role of TNF-α in the mechanisms responsible for preterm delivery induced by Stx2 in rats.Lipopolysaccharide Induces Human Pulmonary Micro-Vascular Endothelial Apoptosis via the YAP Signaling Pathway.The shigella paradigm and colitis due to enterohaemorrhagic Escherichia coli.Tumor necrosis factor alpha increases human cerebral endothelial cell Gb3 and sensitivity to Shiga toxinSensitization of human umbilical vein endothelial cells to Shiga toxin: involvement of protein kinase C and NF-kappaB.LPS-primed CD11b+ leukocytes serve as an effective carrier of Shiga toxin 2 to cause hemolytic uremic syndrome in mice.
P2860
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P2860
Shiga toxin-associated hemolytic uremic syndrome: combined cytotoxic effects of shiga toxin and lipopolysaccharide (endotoxin) on human vascular endothelial cells in vitro.
description
1992 nî lūn-bûn
@nan
1992 թուականի Ապրիլին հրատարակուած գիտական յօդուած
@hyw
1992 թվականի ապրիլին հրատարակված գիտական հոդված
@hy
1992年の論文
@ja
1992年論文
@yue
1992年論文
@zh-hant
1992年論文
@zh-hk
1992年論文
@zh-mo
1992年論文
@zh-tw
1992年论文
@wuu
name
Shiga toxin-associated hemolyt ...... ar endothelial cells in vitro.
@ast
Shiga toxin-associated hemolyt ...... ar endothelial cells in vitro.
@en
type
label
Shiga toxin-associated hemolyt ...... ar endothelial cells in vitro.
@ast
Shiga toxin-associated hemolyt ...... ar endothelial cells in vitro.
@en
prefLabel
Shiga toxin-associated hemolyt ...... ar endothelial cells in vitro.
@ast
Shiga toxin-associated hemolyt ...... ar endothelial cells in vitro.
@en
P2860
P921
P1476
Shiga toxin-associated hemolyt ...... lar endothelial cells in vitro
@en
P2093
P2860
P304
P407
P577
1992-04-01T00:00:00Z