Generation and phenotypic analysis of protein S-deficient mice. - Wikidata - wikimedia - TriplyDB

Generation and phenotypic analysis of protein S-deficient mice.

Generation and phenotypic analysis of protein S-deficient mice.

http://www.wikidata.org/entity/Q33385212

about

Tyro3, Axl, and Mertk receptor signaling in inflammatory bowel disease and colitis-associated cancer Protein S protects neurons from excitotoxic injury by activating the TAM receptor Tyro3-phosphatidylinositol 3-kinase-Akt pathway through its sex hormone-binding globulin-like region Activated protein C cofactor function of protein S: a critical role for Asp95 in the EGF1-like domain T cell-derived protein S engages TAM receptor signaling in dendritic cells to control the magnitude of the immune response.Protein S is a cofactor for platelet and endothelial tissue factor pathway inhibitor-α but not for cell surface-associated tissue factor pathway inhibitor Protein S controls hypoxic/ischemic blood-brain barrier disruption through the TAM receptor Tyro3 and sphingosine 1-phosphate receptor.Dendritic cells in systemic lupus erythematosus Targeted mutagenesis of zebrafish antithrombin III triggers disseminated intravascular coagulation and thrombosis, revealing insight into function.The protein C pathway in tissue inflammation and injury: pathogenic role and therapeutic implications Regulation of phagocytosis by TAM receptors and their ligands.Protein S blocks the extrinsic apoptotic cascade in tissue plasminogen activator/N-methyl D-aspartate-treated neurons via Tyro3-Akt-FKHRL1 signaling pathway.Systemic autoimmunity in TAM triple knockout mice causes inflammatory brain damage and cell death.ELISA-Based Detection System for Protein S K196E Mutation, a Genetic Risk Factor for Venous Thromboembolism.TAM receptor signaling in immune homeostasis.Exacerbated venous thromboembolism in mice carrying a protein S K196E mutation Vitamin K and the nervous system: an overview of its actions.Phosphorylation of protein S by platelet kinases enhances its activated protein C cofactor activity.TAM receptor signaling and autoimmune disease.TAM receptor tyrosine kinase function and the immunopathology of liver disease.Lack of protein S in mice causes embryonic lethal coagulopathy and vascular dysgenesis Molecular basis of protein S deficiency in China.Targeting anticoagulant protein S to improve hemostasis in hemophilia.Protein S Protects against Podocyte Injury in Diabetic Nephropathy.Plasma protein S residues 37-50 mediate its binding to factor Va and inhibition of blood coagulation.

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P2860

Generation and phenotypic analysis of protein S-deficient mice.

http://www.wikidata.org/entity/Q33385212

description

2009 nî lūn-bûn

@nan

2009 թուականի Յունիսին հրատարակուած գիտական յօդուած

@hyw

2009 թվականի հունիսին հրատարակված գիտական հոդված

@hy

2009年の論文

@ja

2009年論文

@yue

2009年論文

@zh-hant

2009年論文

@zh-hk

2009年論文

@zh-mo

2009年論文

@zh-tw

2009年论文

@wuu

name

Generation and phenotypic analysis of protein S-deficient mice.

@ast

Generation and phenotypic analysis of protein S-deficient mice.

@en

type

label

Generation and phenotypic analysis of protein S-deficient mice.

@ast

Generation and phenotypic analysis of protein S-deficient mice.

@en

prefLabel

Generation and phenotypic analysis of protein S-deficient mice.

@ast

Generation and phenotypic analysis of protein S-deficient mice.

@en

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BLOOD-2009-03-209031

P1433

P1476

Generation and phenotypic analysis of protein S-deficient mice

@en

P2093

Anne C Brisset

http://www.w3.org/2001/XMLSchema#string

José A Fernández

http://www.w3.org/2001/XMLSchema#string

Marc Schapira

http://www.w3.org/2001/XMLSchema#string

Monica Azevedo

http://www.w3.org/2001/XMLSchema#string

Svetlana N Tchaikovski

http://www.w3.org/2001/XMLSchema#string

Tilman M Hackeng

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P2860

Protein S binds to and inhibits factor Xa

Inactivation of the gene for anticoagulant protein C causes lethal perinatal consumptive coagulopathy in mice

Zfp423 is required for normal cerebellar development.

Homeostatic regulation of the immune system by receptor tyrosine kinases of the Tyro 3 family

Effects of protein S and factor Xa on peptide bond cleavages during inactivation of factor Va and factor VaR506Q by activated protein C

A highly efficient recombineering-based method for generating conditional knockout mutations

A highly efficient Escherichia coli-based chromosome engineering system adapted for recombinogenic targeting and subcloning of BAC DNA

Key role of the P2Y(1) receptor in tissue factor-induced thrombin-dependent acute thromboembolism: studies in P2Y(1)-knockout mice and mice treated with a P2Y(1) antagonist.

Deficiency or inhibition of Gas6 causes platelet dysfunction and protects mice against thrombosis.

Mice with a severe deficiency in protein C display prothrombotic and proinflammatory phenotypes and compromised maternal reproductive capabilities.

P304

2307-2314

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scholarly article

P356

10.1182/BLOOD-2009-03-209031

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P433

11

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P478

114

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Anne Angelillo-Scherrer

François Saller

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2009-06-30T00:00:00Z

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26331774

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19567881

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2745849

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