IgG antibodies to dengue enhanced for FcγRIIIA binding determine disease severity. - Wikidata - wikimedia - TriplyDB

IgG antibodies to dengue enhanced for FcγRIIIA binding determine disease severity.

IgG antibodies to dengue enhanced for FcγRIIIA binding determine disease severity.

http://www.wikidata.org/entity/Q33438726

about

Transposon mutagenesis of the Zika virus genome highlights regions essential for RNA replication and restricted for immune evasion.Characterization of Zika virus binding and enhancement potential of a large panel of flavivirus murine monoclonal antibodies.Exploring Site-Specific N-Glycosylation of HEK293 and Plant-Produced Human IgA Isotypes.DNA-immunisation with dengue virus E protein domains I/II, but not domain III, enhances Zika, West Nile and Yellow Fever virus infection.Biological functions of fucose in mammals.Dengue Virus Evades AAV-Mediated Neutralizing Antibody Prophylaxis in Rhesus Monkeys.Anti-dengue IgG1.Decoding the Human Immunoglobulin G-Glycan Repertoire Reveals a Spectrum of Fc-Receptor- and Complement-Mediated-Effector Activities.Patients with IgG1-anti-red blood cell autoantibodies show aberrant Fc-glycosylation Neutralizing human monoclonal antibodies prevent Zika virus infection in macaques.Pro-inflammatory State in Monoclonal Gammopathy of Undetermined Significance and in Multiple Myeloma Is Characterized by Low Sialylation of Pathogen-Specific and Other Monoclonal Immunoglobulins.Engineered Sialylation of Pathogenic Antibodies In Vivo Attenuates Autoimmune Disease.Immune Response to Dengue and Zika.Small Molecules and Antibodies for Zika Therapy.Antibody-dependent enhancement of severe dengue disease in humans.Differential antibody glycosylation in autoimmunity: sweet biomarker or modulator of disease activity?Fcγ Receptor Function and the Design of Vaccination Strategies.Fc-Glycosylation in Human IgG1 and IgG3 Is Similar for Both Total and Anti-Red-Blood Cell Anti-K Antibodies.Novel Concepts of Altered Immunoglobulin G Galactosylation in Autoimmune Diseases.Diversification of IgG effector functions.Feasibility and biological rationale of repurposing sunitinib and erlotinib for dengue treatment.Modulation of Dengue/Zika Virus Pathogenicity by Antibody-Dependent Enhancement and Strategies to Protect Against Enhancement in Zika Virus Infection.A Simple Flow Cytometry Based Assay to Determine In Vitro Antibody Dependent Enhancement of Dengue Virus Using Zika Virus Convalescent Serum.Elimination of Falciparum Malaria and Emergence of Severe Dengue: An Independent or Interdependent Phenomenon?Flavivirus serocomplex cross-reactive immunity is protective by activating heterologous memory CD4 T cells.Zika convalescent macaques display delayed induction of anmanestic cross-neutralizing antibody responses after dengue infection.The Role of Fc Gamma Receptors in Broad Protection against Influenza Viruses

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P2860

IgG antibodies to dengue enhanced for FcγRIIIA binding determine disease severity.

http://www.wikidata.org/entity/Q33438726

description

2017 nî lūn-bûn

@nan

2017 թուականի Յունուարին հրատարակուած գիտական յօդուած

@hyw

2017 թվականի հունվարին հրատարակված գիտական հոդված

@hy

2017年の論文

@ja

2017年論文

@yue

2017年論文

@zh-hant

2017年論文

@zh-hk

2017年論文

@zh-mo

2017年論文

@zh-tw

2017年论文

@wuu

name

IgG antibodies to dengue enhanced for FcγRIIIA binding determine disease severity.

@ast

IgG antibodies to dengue enhanced for FcγRIIIA binding determine disease severity.

@en

type

label

IgG antibodies to dengue enhanced for FcγRIIIA binding determine disease severity.

@ast

IgG antibodies to dengue enhanced for FcγRIIIA binding determine disease severity.

@en

altLabel

IgG antibodies to dengue enhanced for FcγRIIIA binding determine disease severity

@en

prefLabel

IgG antibodies to dengue enhanced for FcγRIIIA binding determine disease severity.

@ast

IgG antibodies to dengue enhanced for FcγRIIIA binding determine disease severity.

@en

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SCIENCE.AAI8128

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IgG antibodies to dengue enhanced for FcγRIIIA binding determine disease severity.

@en

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Benjamin A Pinsky

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Jaturong Sewatanon

http://www.w3.org/2001/XMLSchema#string

Jeffrey V Ravetch

http://www.w3.org/2001/XMLSchema#string

Kulkanya Chokephaibulkit

http://www.w3.org/2001/XMLSchema#string

Nattawat Onlamoon

http://www.w3.org/2001/XMLSchema#string

Rafi Ahmed

http://www.w3.org/2001/XMLSchema#string

Siddhartha Kumar Bhaumik

http://www.w3.org/2001/XMLSchema#string

Stylianos Bournazos

http://www.w3.org/2001/XMLSchema#string

Taia T Wang

http://www.w3.org/2001/XMLSchema#string

P2860

Subversion of innate defenses by the interplay between DENV and pre-existing enhancing antibodies: TLRs signaling collapse

Observations related to pathogenesis of dengue hemorrhagic fever. I. Experience with classification of dengue viruses

Human antibody responses after dengue virus infection are highly cross-reactive to Zika virus

Antibody-Dependent Enhancement of Dengue Virus Infection in Primary Human Macrophages; Balancing Higher Fusion against Antiviral Responses

Dengue virus structural differences that correlate with pathogenesis.

Primary Human Splenic Macrophages, but Not T or B Cells, Are the Principal Target Cells for Dengue Virus Infection In Vitro

Dengue viremia titer, antibody response pattern, and virus serotype correlate with disease severity

Origins of dengue type 2 viruses associated with increased pathogenicity in the Americas

Evidence that maternal dengue antibodies are important in the development of dengue hemorrhagic fever in infants

A prospective seroepidemiologic study on dengue in children four to nine years of age in Yogyakarta, Indonesia I. studies in 1995-1996.

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6323

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355

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Jeffery Taubenberger

Matthew J Memoli

Kovit Pattanapanyasat

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2017-01-01T00:00:00Z

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28126818

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