Truncation in the tcdC region of the Clostridium difficile PathLoc of clinical isolates does not predict increased biological activity of Toxin B or Toxin A - Wikidata - wikimedia - TriplyDB

Truncation in the tcdC region of the Clostridium difficile PathLoc of clinical isolates does not predict increased biological activity of Toxin B or Toxin A

Truncation in the tcdC region of the Clostridium difficile PathLoc of clinical isolates does not predict increased biological activity of Toxin B or Toxin A

http://www.wikidata.org/entity/Q33475206

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The Regulatory Networks That Control Clostridium difficile Toxin Synthesis The potential for emerging therapeutic options for Clostridium difficile infection Variations in TcdB activity and the hypervirulence of emerging strains of Clostridium difficile The anti-sigma factor TcdC modulates hypervirulence in an epidemic BI/NAP1/027 clinical isolate of Clostridium difficile.TcdC does not significantly repress toxin expression in Clostridium difficile 630ΔErm.Characterization of a stable, metronidazole-resistant Clostridium difficile clinical isolate Using phenotype microarrays to determine culture conditions that induce or repress toxin production by Clostridium difficile and other microorganisms.A unique strain of community-acquired Clostridium difficile in severe complicated infection and death of a young adult.Fourteen-genome comparison identifies DNA markers for severe-disease-associated strains of Clostridium difficile Emergence of an outbreak-associated Clostridium difficile variant with increased virulence.Multiplex PCR method for detection of Clostridium difficile tcdA, tcdB, cdtA, and cdtB and internal in-frame deletion of tcdC Characterization of Clostridium difficile Strains in British Columbia, Canada: A Shift from NAP1 Majority (2008) to Novel Strain Types (2013) in One Region Variations in virulence and molecular biology among emerging strains of Clostridium difficile.The continually evolving Clostridium difficile species.Lack of association between clinical outcome of Clostridium difficile infections, strain type, and virulence-associated phenotypes.Binary toxin and its clinical importance in Clostridium difficile infection, Belgium.Differentiating virulent 027 and non-027 Clostridium difficile strains by molecular methods.Molecular epidemiology and antimicrobial susceptibility of Clostridium difficile isolated from a university teaching hospital in Japan.Diagnosis of Clostridium difficile: real-time PCR detection of toxin genes in faecal samples is more sensitive compared to toxigenic culture.Precise manipulation of the Clostridium difficile chromosome reveals a lack of association between the tcdC genotype and toxin production.Editorial commentary: Host-pathogen interactions in Clostridium difficile infection: it takes two to tango.Prophage-stimulated toxin production in Clostridium difficile NAP1/027 lysogens.Secretome analysis of Clostridium difficile strains.Clinical features of Clostridium difficile infection and molecular characterization of the isolated strains in a cohort of Danish hospitalized patients.

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P2860

Truncation in the tcdC region of the Clostridium difficile PathLoc of clinical isolates does not predict increased biological activity of Toxin B or Toxin A

http://www.wikidata.org/entity/Q33475206

description

2009 nî lūn-bûn

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2009 թուականի Յունիսին հրատարակուած գիտական յօդուած

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2009 թվականի հունիսին հրատարակված գիտական հոդված

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2009年の論文

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2009年論文

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2009年論文

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Truncation in the tcdC region ...... activity of Toxin B or Toxin A

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BMC Infectious Diseases

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Truncation in the tcdC region ...... activity of Toxin B or Toxin A

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Dave Boyd

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Michael R Mulvey

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Paul N Levett

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Ruth Murray

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P2860

Clostridium difficile toxins: mechanism of action and role in disease.

Update on Clostridium difficile associated disease.

Production of actin-specific ADP-ribosyltransferase (binary toxin) by strains of Clostridium difficile.

Characterization of a toxin A-negative, toxin B-positive strain of Clostridium difficile responsible for a nosocomial outbreak of Clostridium difficile-associated diarrhea.

Mortality attributable to nosocomial Clostridium difficile-associated disease during an epidemic caused by a hypervirulent strain in Quebec

Molecular analysis of the pathogenicity locus and polymorphism in the putative negative regulator of toxin production (TcdC) among Clostridium difficile clinical isolates.

Correlation of disease severity with fecal toxin levels in patients with Clostridium difficile-associated diarrhea and distribution of PCR ribotypes and toxin yields in vitro of corresponding isolates.

Severe Clostridium difficile-associated disease in populations previously at low risk--four states, 2005.

A predominantly clonal multi-institutional outbreak of Clostridium difficile-associated diarrhea with high morbidity and mortality.

An epidemic, toxin gene-variant strain of Clostridium difficile.

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1471-2334-9-103

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Clostridium difficile

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