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Evasion and Immuno-Endocrine Regulation in Parasite Infection: Two Sides of the Same Coin in Chagas Disease?Immune Evasion Strategies of Trypanosoma cruziModulation of immune response in experimental Chagas diseaseTrypanosomiasis-induced megacolon illustrates how myenteric neurons modulate the risk for colon cancer in rats and humansPathogenesis of chagas' disease: parasite persistence and autoimmunityAltered bone marrow lymphopoiesis and interleukin-6-dependent inhibition of thymocyte differentiation contribute to thymic atrophy during Trypanosoma cruzi infection.BAFF mediates splenic B cell response and antibody production in experimental Chagas disease.Unraveling Chagas disease transmission through the oral route: Gateways to Trypanosoma cruzi infection and target tissues.Chagasic thymic atrophy does not affect negative selection but results in the export of activated CD4+CD8+ T cells in severe forms of human disease.Different infective forms trigger distinct immune response in experimental Chagas diseaseThe receptor Slamf1 on the surface of myeloid lineage cells controls susceptibility to infection by Trypanosoma cruziAnalysis of the dynamics of infiltrating CD4(+) T cell subsets in the heart during experimental Trypanosoma cruzi infectionIL-10-IFN-γ double producers CD4+ T cells are induced by immunization with an amastigote stage specific derived recombinant protein of Trypanosoma cruzi.Cytokine, antibody and proliferative cellular responses elicited by Taenia solium calreticulin upon experimental infection in hamstersNADPH oxidase inhibition ameliorates Trypanosoma cruzi-induced myocarditis during Chagas disease.Trypanosoma cruzi trans-sialidase prevents elicitation of Th1 cell response via interleukin 10 and downregulates Th1 effector cells.Trypanosoma cruzi Infection through the Oral Route Promotes a Severe Infection in Mice: New Disease Form from an Old Infection?Oral Outbreak of Chagas Disease in Santa Catarina, Brazil: Experimental Evaluation of a Patient's StrainDynamics of Lymphocyte Populations during Trypanosoma cruzi Infection: From Thymocyte Depletion to Differential Cell Expansion/Contraction in Peripheral Lymphoid Organs.The Role of Sialic Acid-Binding Receptors (Siglecs) in the Immunomodulatory Effects of Trypanosoma cruzi Sialoglycoproteins on the Protective Immunity of the Host.Trypanosoma cruzi Entrance through Systemic or Mucosal Infection Sites Differentially Modulates Regional Immune Response Following Acute Infection in Mice.Thymus atrophy and double-positive escape are common features in infectious diseases.Mature peripheral T cells are important to preserve thymus function and selection of thymocytes during Mycobacterium tuberculosis infection.Interferon-γ- and glucocorticoid-mediated pathways synergize to enhance death of CD4(+) CD8(+) thymocytes during Salmonella enterica serovar Typhimurium infection.Role of Hormonal Circuitry Upon T Cell Development in Chagas Disease: Possible Implications on T Cell Dysfunctions.
P2860
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P2860
description
2009 nî lūn-bûn
@nan
2009 թուականի Յուլիսին հրատարակուած գիտական յօդուած
@hyw
2009 թվականի հուլիսին հրատարակված գիտական հոդված
@hy
2009年の論文
@ja
2009年論文
@yue
2009年論文
@zh-hant
2009年論文
@zh-hk
2009年論文
@zh-mo
2009年論文
@zh-tw
2009年论文
@wuu
name
Differential regional immune response in Chagas disease
@ast
Differential regional immune response in Chagas disease
@en
type
label
Differential regional immune response in Chagas disease
@ast
Differential regional immune response in Chagas disease
@en
prefLabel
Differential regional immune response in Chagas disease
@ast
Differential regional immune response in Chagas disease
@en
P2093
P2860
P1476
Differential regional immune response in Chagas disease
@en
P2093
Déa Maria Serra Villa-Verde
Désio Aurélio Farias-de-Oliveira
Juliana de Meis
Wilson Savino
P2860
P356
10.1371/JOURNAL.PNTD.0000417
P407
P5008
P577
2009-07-07T00:00:00Z