Dimethylarginine dimethylaminohydrolase-1 transgenic mice are not protected from ischemic stroke. - Wikidata - wikimedia - TriplyDB

Dimethylarginine dimethylaminohydrolase-1 transgenic mice are not protected from ischemic stroke.

Dimethylarginine dimethylaminohydrolase-1 transgenic mice are not protected from ischemic stroke.

http://www.wikidata.org/entity/Q33509022

about

Paladin (X99384) is expressed in the vasculature and shifts from endothelial to vascular smooth muscle cells during mouse development Asymmetric dimethyarginine as marker and mediator in ischemic stroke Glutamate transporter type 3 knockout reduces brain tolerance to focal brain ischemia in mice Asymmetric dimethylarginine potentiates lung inflammation in a mouse model of allergic asthma.Dimethylarginine dimethylaminohydrolase1 is an organ-specific mediator of end organ damage in a murine model of hypertension Association of dimethylarginines and mediators of inflammation after acute ischemic stroke Method parameters' impact on mortality and variability in mouse stroke experiments: a meta-analysis.The role of asymmetric and symmetric dimethylarginines in renal disease.Effect of Treatment with Cyanidin-3-O-β-D-Glucoside on Rat Ischemic/Reperfusion Brain Damage.Endothelial dysfunction of the peripheral vascular bed in the acute phase after ischemic stroke.

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Dimethylarginine dimethylaminohydrolase-1 transgenic mice are not protected from ischemic stroke.

http://www.wikidata.org/entity/Q33509022

description

2009 nî lūn-bûn

@nan

2009 թուականի Հոկտեմբերին հրատարակուած գիտական յօդուած

@hyw

2009 թվականի հոտեմբերին հրատարակված գիտական հոդված

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2009年の論文

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2009年論文

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2009年論文

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2009年論文

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2009年論文

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2009年論文

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2009年论文

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Dimethylarginine dimethylamino ...... rotected from ischemic stroke.

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Dimethylarginine dimethylamino ...... rotected from ischemic stroke.

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Dimethylarginine dimethylamino ...... rotected from ischemic stroke.

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Chi-Un Choe

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Christian Gerloff

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Dorothee Atzler

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Eike-Christin von Leitner

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Rainer H Böger

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Tim Magnus

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P2860

Asymmetric dimethylarginine, smoking, and risk of coronary heart disease in apparently healthy men: prospective analysis from the population-based Monitoring of Trends and Determinants in Cardiovascular Disease/Kooperative Gesundheitsforschung in de

Overexpression of dimethylarginine dimethylaminohydrolase reduces tissue asymmetric dimethylarginine levels and enhances angiogenesis.

Dimethylarginine dimethylaminohydrolase regulates nitric oxide synthesis: genetic and physiological evidence.

Asymmetric dimethylarginine (ADMA): a novel risk marker in cardiovascular medicine and beyond.

Plasma asymmetric dimethylarginine and incidence of cardiovascular disease and death in the community

Association of the endogenous nitric oxide synthase inhibitor ADMA with carotid artery intimal media thickness in the Framingham Heart Study offspring cohort.

Asymmetric dimethylarginine causes hypertension and cardiac dysfunction in humans and is actively metabolized by dimethylarginine dimethylaminohydrolase.

Dimethylarginine dimethylaminohydrolase promotes endothelial repair after vascular injury.

Asymmetric dimethylarginine (ADMA) as a risk marker for stroke and TIA in a Swedish population.

Overexpression of dimethylarginine dimethylaminohydrolase inhibits asymmetric dimethylarginine-induced endothelial dysfunction in the cerebral circulation.

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Edzard Schwedhelm

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