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Small molecule control of virulence gene expression in Francisella tularensis

Small molecule control of virulence gene expression in Francisella tularensis

http://www.wikidata.org/entity/Q33513964

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Identification of small RNAs in Francisella tularensis Identification of a small molecule that modifies MglA/SspA interaction and impairs intramacrophage survival of Francisella tularensis The CgrA and CgrC proteins form a complex that positively regulates cupA fimbrial gene expression in Pseudomonas aeruginosa Structural and Biochemical Characterization of the Francisella tularensis Pathogenicity Regulator, Macrophage Locus Protein A (MglA)Regulation of virulence gene transcripts by the Francisella novicida orphan response regulator PmrA: role of phosphorylation and evidence of MglA/SspA interaction.ppGpp conjures bacterial virulence Nooks and crannies in type VI secretion regulation.Coordinate control of virulence gene expression in Francisella tularensis involves direct interaction between key regulators Identification of a novel small RNA modulating Francisella tularensis pathogenicity.SspA up-regulates gene expression of the LEE pathogenicity island by decreasing H-NS levels in enterohemorrhagic Escherichia coli.The stringent response of Staphylococcus aureus and its impact on survival after phagocytosis through the induction of intracellular PSMs expression.Identification of Francisella novicida mutants that fail to induce prostaglandin E(2) synthesis by infected macrophages.Type A Francisella tularensis acid phosphatases contribute to pathogenesis.MglA/SspA complex interactions are modulated by inorganic polyphosphate Use of a capture-based pathogen transcript enrichment strategy for RNA-Seq analysis of the Francisella tularensis LVS transcriptome during infection of murine macrophages.Regulation of francisella tularensis virulence.Francisella tularensis metabolism and its relation to virulence.Immunoproteomic analysis of proteins expressed by two related pathogens, Burkholderia multivorans and Burkholderia cenocepacia, during human infection.Characterization of the RelBbu Regulon in Borrelia burgdorferi Reveals Modulation of Glycerol Metabolism by (p)ppGpp.Ubiquitous promoter-localization of essential virulence regulators in Francisella tularensis The acid phosphatase AcpA is secreted in vitro and in macrophages by Francisella spp.H2O2 production rate in Lactobacillus johnsonii is modulated via the interplay of a heterodimeric flavin oxidoreductase with a soluble 28 Kd PAS domain containing protein.Basis for the essentiality of H-NS family members in Pseudomonas aeruginosa The Francisella tularensis migR, trmE, and cphA genes contribute to F. tularensis pathogenicity island gene regulation and intracellular growth by modulation of the stress alarmone ppGpp Metabolic labeling to characterize the overall composition of Francisella lipid A and LPS grown in broth and in human phagocytes.Nucleotide, c-di-GMP, c-di-AMP, cGMP, cAMP, (p)ppGpp signaling in bacteria and implications in pathogenesis.Proteins involved in Francisella tularensis survival and replication inside macrophages.Uncovering the components of the Francisella tularensis virulence stealth strategy Dissection of the molecular circuitry controlling virulence in Francisella tularensis Differential Growth of Francisella tularensis, Which Alters Expression of Virulence Factors, Dominant Antigens, and Surface-Carbohydrate Synthases, Governs the Apparent Virulence of Ft SchuS4 to Immunized Animals.Bacterial Stress Responses during Host Infection Sleeper cells: the stringent response and persistence in the Borreliella (Borrelia) burgdorferi enzootic cycle.A response regulator promotes Francisella tularensis intramacrophage growth by repressing an anti-virulence factor.The metabolic enzyme fructose-1,6-bisphosphate aldolase acts as a transcriptional regulator in pathogenic Francisella.The Role of the Francisella Tularensis Pathogenicity Island in Type VI Secretion, Intracellular Survival, and Modulation of Host Cell Signaling.Exploitation of host cell biology and evasion of immunity by francisella tularensis.Whole genome transcriptomics reveals global effects including up-regulation of Francisella pathogenicity island gene expression during active stringent response in the highly virulent Francisella tularensis subsp. tularensis SCHU S4.Changes in proteome of the Δhfq strain derived from Francisella tularensis LVS correspond with its attenuated phenotype.Polyphosphate Kinase Antagonizes Virulence Gene Expression in Francisella tularensis.Overexpression of spoT gene in coccoid forms of clinical Helicobacter pylori isolates.

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P2860

Small molecule control of virulence gene expression in Francisella tularensis

http://www.wikidata.org/entity/Q33513964

description

2009 nî lūn-bûn

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2009 թուականի Հոկտեմբերին հրատարակուած գիտական յօդուած

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2009 թվականի հոտեմբերին հրատարակված գիտական հոդված

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2009年の論文

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2009年論文

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2009年論文

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2009年論文

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2009年論文

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2009年論文

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2009年论文

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name

Small molecule control of virulence gene expression in Francisella tularensis

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Small molecule control of virulence gene expression in Francisella tularensis

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Small molecule control of virulence gene expression in Francisella tularensis.

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type

label

Small molecule control of virulence gene expression in Francisella tularensis

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Small molecule control of virulence gene expression in Francisella tularensis

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Small molecule control of virulence gene expression in Francisella tularensis.

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Small molecule control of virulence gene expression in Francisella tularensis

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Small molecule control of virulence gene expression in Francisella tularensis

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Small molecule control of virulence gene expression in Francisella tularensis.

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Small molecule control of virulence gene expression in Francisella tularensis

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Dennis L Kasper

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James C Charity

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Leeann T Blalock

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Michelle M Costante-Hamm

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Simon L Dove

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P2860

Identification of a conserved bacterial protein secretion system in Vibrio cholerae using the Dictyostelium host model system

A virulence locus of Pseudomonas aeruginosa encodes a protein secretion apparatus

The tandem affinity purification (TAP) method: a general procedure of protein complex purification

Iron limitation induces SpoT-dependent accumulation of ppGpp in Escherichia coli

Control of bacterial transcription, translation and replication by (p)ppGpp

(p)ppGpp: still magical?

Twin RNA polymerase-associated proteins control virulence gene expression in Francisella tularensis

The stringent response of Mycobacterium tuberculosis is required for long-term survival

Pseudomonas aeruginosa relA contributes to virulence in Drosophila melanogaster

RegF, an SspA homologue, regulates the expression of the Neisseria gonorrhoeae pilE gene.

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