Timing constraints of in vivo gag mutations during primary HIV-1 subtype C infection
about
Human leukocyte antigens and HIV type 1 viral load in early and chronic infection: predominance of evolving relationshipsHIV-1 subtype C-infected individuals maintaining high viral load as potential targets for the "test-and-treat" approach to reduce HIV transmissionTransmission of single and multiple viral variants in primary HIV-1 subtype C infection.Dynamics and timing of in vivo mutations at Gag residue 242 during primary HIV-1 subtype C infection.Reversion and T cell escape mutations compensate the fitness loss of a CD8+ T cell escape mutant in their cognate transmitted/founder virusIdentifying and characterizing recently transmitted viruses.Nef-mediated down-regulation of CD4 and HLA class I in HIV-1 subtype C infection: association with disease progression and influence of immune pressure.Influence of Gag-protease-mediated replication capacity on disease progression in individuals recently infected with HIV-1 subtype C.High frequency of transmitted HIV-1 Gag HLA class I-driven immune escape variants but minimal immune selection over the first year of clade C infection.Human leukocyte antigen variants B*44 and B*57 are consistently favorable during two distinct phases of primary HIV-1 infection in sub-Saharan Africans with several viral subtypes.Evolutionary gamut of in vivo Gag substitutions during early HIV-1 subtype C infection.Assessing Biases in the Evaluation of Classification Assays for HIV Infection Recency.Deciphering Multiplicity of HIV-1C Infection: Transmission of Closely Related Multiple Viral Lineages.A Generalized Entropy Measure of Within-Host Viral Diversity for Identifying Recent HIV-1 Infectionstat Exon 1 exhibits functional diversity during HIV-1 subtype C primary infection.Intra-host evolutionary rates in HIV-1C env and gag during primary infection.Pairwise diversity and tMRCA as potential markers for HIV infection recencyThe emerging role of HLA-C in HIV-1 infection.Genome-Wide Analyses Reveal Gene Influence on HIV Disease Progression and HIV-1C Acquisition in Southern Africa.Transmitted/Founder HIV-1 Subtype C Viruses Show Distinctive Signature Patterns in Vif, Vpr, and Vpu That Are Under Subsequent Immune Pressure During Early Infection.Exploring the benefits of antibody immune response in HIV-1 infection using a discrete model.Phylogenetic approach to recover integration dates of latent HIV sequences within-host
P2860
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P2860
Timing constraints of in vivo gag mutations during primary HIV-1 subtype C infection
description
2009 nî lūn-bûn
@nan
2009 թուականի Նոյեմբերին հրատարակուած գիտական յօդուած
@hyw
2009 թվականի նոյեմբերին հրատարակված գիտական հոդված
@hy
2009年の論文
@ja
2009年論文
@yue
2009年論文
@zh-hant
2009年論文
@zh-hk
2009年論文
@zh-mo
2009年論文
@zh-tw
2009年论文
@wuu
name
Timing constraints of in vivo gag mutations during primary HIV-1 subtype C infection
@ast
Timing constraints of in vivo gag mutations during primary HIV-1 subtype C infection
@en
Timing constraints of in vivo gag mutations during primary HIV-1 subtype C infection.
@nl
type
label
Timing constraints of in vivo gag mutations during primary HIV-1 subtype C infection
@ast
Timing constraints of in vivo gag mutations during primary HIV-1 subtype C infection
@en
Timing constraints of in vivo gag mutations during primary HIV-1 subtype C infection.
@nl
prefLabel
Timing constraints of in vivo gag mutations during primary HIV-1 subtype C infection
@ast
Timing constraints of in vivo gag mutations during primary HIV-1 subtype C infection
@en
Timing constraints of in vivo gag mutations during primary HIV-1 subtype C infection.
@nl
P2093
P2860
P50
P1433
P1476
Timing constraints of in vivo gag mutations during primary HIV-1 subtype C infection
@en
P2093
Caitlin Bonney
David Nkwe
Elias Woldegabriel
Jeannie Baca
Lauren Margolin
Lemme Kebaabetswe
Michaela Herzig
Raabya Rossenkhan
Rosemary Musonda
P2860
P356
10.1371/JOURNAL.PONE.0007727
P407
P577
2009-11-05T00:00:00Z