Neuropeptide Y knockout mice reveal a central role of NPY in the coordination of bone mass to body weight. - Wikidata - wikimedia - TriplyDB

Neuropeptide Y knockout mice reveal a central role of NPY in the coordination of bone mass to body weight.

Neuropeptide Y knockout mice reveal a central role of NPY in the coordination of bone mass to body weight.

http://www.wikidata.org/entity/Q33520220

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Chronic Psychosocial Stress Impairs Bone Homeostasis: A Study in the Social Isolation Reared Rat.NPY neuron-specific Y2 receptors regulate adipose tissue and trabecular bone but not cortical bone homeostasis in mice Dissecting carboxypeptidase E: properties, functions and pathophysiological roles in disease.Obese carboxypeptidase E knockout mice exhibit multiple defects in peptide hormone processing contributing to low bone mineral density Peptide YY regulates bone remodeling in mice: a link between gut and skeletal biology Increased PTHrP and decreased estrogens alter bone turnover but do not reproduce the full effects of lactation on the skeleton.The hunger games of skeletal metabolism.Micellar nanomedicine of human neuropeptide Y.NPY signalling in early osteoblasts controls glucose homeostasis.High neuropeptide Y release associates with Ewing sarcoma bone dissemination - in vivo model of site-specific metastases Prader-Willi Critical Region, a Non-Translated, Imprinted Central Regulator of Bone Mass: Possible Role in Skeletal Abnormalities in Prader-Willi Syndrome.Osteoanabolics.Pleiotropic effects of incretins New roles of carboxypeptidase E in endocrine and neural function and cancer.Bone Injury and Repair Trigger Central and Peripheral NPY Neuronal Pathways.Bone-specific overexpression of NPY modulates osteogenesis Neuropeptide Y1 Receptor Regulates Glucocorticoid-Induced Inhibition of Osteoblast Differentiation in Murine MC3T3-E1 Cells via ERK Signaling.Neuropeptide Y and osteoblast differentiation--the balance between the neuro-osteogenic network and local control.The NPY system and its neural and neuroendocrine regulation of bone.Bariatric surgery, bone loss, obesity and possible mechanisms.Neuropeptide Y receptors: how to get subtype selectivity Influence of hormonal appetite and energy regulators on bone.Neuropeptide Y is a critical modulator of leptin's regulation of cortical bone.Y2 and Y4 receptor signalling attenuates the skeletal response of central NPY.Roles of leptin in bone metabolism and bone diseases.Osteoblastic Actions of the Neuropeptide Y System to Regulate Bone and Energy Homeostasis.The Central Nervous System and Bone Metabolism: An Evolving Story.Recent advances in the understanding of how neuropeptide Y and α-melanocyte stimulating hormone function in adipose physiology.Bone Cell Bioenergetics and Skeletal Energy Homeostasis.Neuropeptide Y modulates fracture healing through Y1 receptor signaling.Neuropeptide Y in the noradrenergic neurones induces obesity and inhibits sympathetic tone in mice.Additive actions of the cannabinoid and neuropeptide Y systems on adiposity and lipid oxidation.Endocrine regulation of bone and energy metabolism in hibernating mammals.Bone adaptation to mechanical loading in a mouse model of reduced peripheral sensory nerve function.Gut Microbiota and Bone Health.Neuropeptide y attenuates stress-induced bone loss through suppression of noradrenaline circuits.Neuropeptide Y mediates glucocorticoid-induced osteoporosis and marrow adiposity in mice.Long-term Consequences of Traumatic Brain Injury in Bone Metabolism.Brain to bone: What is the contribution of the brain to skeletal homeostasis?Osteoglycin, a novel coordinator of bone and glucose homeostasis.

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P2860

Neuropeptide Y knockout mice reveal a central role of NPY in the coordination of bone mass to body weight.

http://www.wikidata.org/entity/Q33520220

description

2009 nî lūn-bûn

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2009 թուականի Դեկտեմբերին հրատարակուած գիտական յօդուած

@hyw

2009 թվականի դեկտեմբերին հրատարակված գիտական հոդված

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2009年の論文

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2009年論文

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2009年論文

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2009年論文

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2009年論文

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2009年論文

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2009年论文

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Aygul Aljanova

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Bernhard Stehrer

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Dominique D Pierroz

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En-Ju D Lin

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John A Eisman

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Katy Slack

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P2860

Leptin inhibits bone formation through a hypothalamic relay: a central control of bone mass

Direct three-dimensional morphometric analysis of human cancellous bone: microstructural data from spine, femur, iliac crest, and calcaneus.

Caloric restriction decreases cortical bone mass but spares trabecular bone in the mouse skeleton: implications for the regulation of bone mass by body weight.

Leptin regulates bone formation via the sympathetic nervous system.

Hypothalamic Y2 receptors regulate bone formation

beta-Arrestin2 regulates the differential response of cortical and trabecular bone to intermittent PTH in female mice

Nutrition-related peptides and bone homeostasis.

Functional adaptation to loading of a single bone is neuronally regulated and involves multiple bones.

Relationships between fat and bone.

Neuropeptide Y is expressed by osteocytes and can inhibit osteoblastic activity.

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