Genetic and physiological activation of osmosensitive gene expression mimics transcriptional signatures of pathogen infection in C. elegans. - Wikidata - wikimedia - TriplyDB

Genetic and physiological activation of osmosensitive gene expression mimics transcriptional signatures of pathogen infection in C. elegans.

Genetic and physiological activation of osmosensitive gene expression mimics transcriptional signatures of pathogen infection in C. elegans.

http://www.wikidata.org/entity/Q33528953

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GATA transcription factors as tissue-specific master regulators for induced responses The Caenorhabditis elegans epidermis as a model skin. II: differentiation and physiological roles FLCN and AMPK Confer Resistance to Hyperosmotic Stress via Remodeling of Glycogen Stores The Caenorhabditis elegans mucin-like protein OSM-8 negatively regulates osmosensitive physiology via the transmembrane protein PTR-23 TonEBP stimulates multiple cellular pathways for adaptation to hypertonic stress: organic osmolyte-dependent and -independent pathways Characterization of the proteostasis roles of glycerol accumulation, protein degradation and protein synthesis during osmotic stress in C. elegans Osmoprotective transcription factor NFAT5/TonEBP modulates nuclear factor-kappaB activity.Stress and aging induce distinct polyQ protein aggregation states Alterations in gene expression in Caenorhabditis elegans associated with organophosphate pesticide intoxication and recovery.Neuronal Chemosensation and Osmotic Stress Response Converge in the Regulation of aqp-8 in C. elegans Activation of a G protein-coupled receptor by its endogenous ligand triggers the innate immune response of Caenorhabditis elegans.Quantitative and automated high-throughput genome-wide RNAi screens in C. elegans.System wide analysis of the evolution of innate immunity in the nematode model species Caenorhabditis elegans and Pristionchus pacificus The GATA transcription factor egl-27 delays aging by promoting stress resistance in Caenorhabditis elegans.The transgenerational effects of heat stress in the nematode Caenorhabditis remanei are negative and rapidly eliminated under direct selection for increased stress resistance in larvae Gene-environment and protein-degradation signatures characterize genomic and phenotypic diversity in wild Caenorhabditis elegans populations.C. elegans Notch signaling regulates adult chemosensory response and larval molting quiescence Stabilization of RNT-1 protein, runt-related transcription factor (RUNX) protein homolog of Caenorhabditis elegans, by oxidative stress through mitogen-activated protein kinase pathway Contrasting invertebrate immune defense behaviors caused by a single gene, the Caenorhabditis elegans neuropeptide receptor gene npr-1 Abnormal Osmotic Avoidance Behavior in C. elegans Is Associated with Increased Hypertonic Stress Resistance and Improved Proteostasis A quantitative genome-wide RNAi screen in C. elegans for antifungal innate immunity genes.A UPR-independent infection-specific role for a BiP/GRP78 protein in the control of antimicrobial peptide expression in C. elegans epidermis The cystic-fibrosis-associated ΔF508 mutation confers post-transcriptional destabilization on the C. elegans ABC transporter PGP-3.Strength in numbers: "Omics" studies of C. elegans innate immunity.Physiological and molecular mechanisms of salt and water homeostasis in the nematode Caenorhabditis elegans.The Caenorhabditis elegans intestine.Survival assays using Caenorhabditis elegans.Glycogen: A must have storage to survive stressful emergencies Modulatory upregulation of an insulin peptide gene by different pathogens in C. elegans.Normal sleep bouts are not essential for C. elegans survival and FoxO is important for compensatory changes in sleep.A Damage Sensor Associated with the Cuticle Coordinates Three Core Environmental Stress Responses in Caenorhabditis elegans.Pathogenesis of the Candida parapsilosis Complex in the Model Host Caenorhabditis elegans

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P2860

Genetic and physiological activation of osmosensitive gene expression mimics transcriptional signatures of pathogen infection in C. elegans.

http://www.wikidata.org/entity/Q33528953

description

2010 nî lūn-bûn

@nan

2010 թուականի Փետրուարին հրատարակուած գիտական յօդուած

@hyw

2010 թվականի փետրվարին հրատարակված գիտական հոդված

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2010年の論文

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2010年論文

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2010年論文

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2010年論文

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2010年論文

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2010年論文

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2010年论文

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name

Genetic and physiological acti ...... hogen infection in C. elegans.

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Genetic and physiological acti ...... hogen infection in C. elegans.

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Genetic and physiological acti ...... hogen infection in C. elegans.

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Genetic and physiological acti ...... hogen infection in C. elegans.

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Genetic and physiological acti ...... hogen infection in C. elegans.

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Genetic and physiological acti ...... hogen infection in C. elegans.

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Genetic and physiological acti ...... hogen infection in C. elegans.

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Genetic and physiological acti ...... hogen infection in C. elegans.

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Genetic and physiological acti ...... hogen infection in C. elegans.

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Genetic and physiological acti ...... hogen infection in C. elegans.

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Anne-Katrin Rohlfing

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Sridhar Hannenhalli

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Todd Lamitina

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P2860

Tonicity-responsive enhancer binding protein, a rel-like protein that stimulates transcription in response to hypertonicity

Significance analysis of microarrays applied to the ionizing radiation response

Genetic interactions due to constitutive and inducible gene regulation mediated by the unfolded protein response in C. elegans.

p38 MAPK regulates expression of immune response genes and contributes to longevity in C. elegans

Genes that act downstream of DAF-16 to influence the lifespan of Caenorhabditis elegans

Pathogen recognition and innate immunity

Molecular mechanisms of bacterial virulence elucidated using a Pseudomonas aeruginosa-Caenorhabditis elegans pathogenesis model

Killing of Caenorhabditis elegans by Pseudomonas aeruginosa used to model mammalian bacterial pathogenesis

Activators and target genes of Rel/NF-kappaB transcription factors

DNA transformation

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