Cytomegalovirus-specific T cells persist at very high levels during long-term antiretroviral treatment of HIV disease. - Wikidata - wikimedia - TriplyDB

Cytomegalovirus-specific T cells persist at very high levels during long-term antiretroviral treatment of HIV disease.

Cytomegalovirus-specific T cells persist at very high levels during long-term antiretroviral treatment of HIV disease.

http://www.wikidata.org/entity/Q33528999

about

Monocytes as regulators of inflammation and HIV-related comorbidities during cART Understanding frailty, aging, and inflammation in HIV infection Rational development of radiopharmaceuticals for HIV-1 Partners in Crime: The Role of CMV in Immune Dysregulation and Clinical Outcome During HIV Infection Cardiovascular Complications of HIV-Associated Immune Dysfunction Angiotensin converting enzyme inhibitor and HMG-CoA reductase inhibitor as adjunct treatment for persons with HIV infection: a feasibility randomized trial The CD4:CD8 ratio is associated with markers of age-associated disease in virally suppressed HIV-infected patients with immunological recovery Ageing and inflammation in patients with HIV infection.Cytomegalovirus and HIV: A Dangerous Pas de Deux HIV, Aging, and Viral Coinfections: Taking the Long View.Hepatitis C viremia is associated with cytomegalovirus IgG antibody levels in HIV-infected women.Systemic effects of inflammation on health during chronic HIV infection HIV-infected individuals with low CD4/CD8 ratio despite effective antiretroviral therapy exhibit altered T cell subsets, heightened CD8+ T cell activation, and increased risk of non-AIDS morbidity and mortality.Is HIV a model of accelerated or accentuated aging?A low T regulatory cell response may contribute to both viral control and generalized immune activation in HIV controllers.Inflammation, Immune Activation, and Antiretroviral Therapy in HIV Heterogeneity of CD4+ and CD8+ T-cell responses to cytomegalovirus in HIV-infected and HIV-uninfected men who have sex with men.Low proportions of CD28- CD8+ T cells expressing CD57 can be reversed by early ART initiation and predict mortality in treated HIV infection.Residual immune dysregulation syndrome in treated HIV infection.HIV infection, inflammation, immunosenescence, and aging.Chronic HIV disease and activation of the coagulation system Immune activation and HIV persistence: implications for curative approaches to HIV infection.Programmed death-1 expression on CD4⁺ and CD8⁺ T cells in treated and untreated HIV disease.T-cell phenotypes, apoptosis and inflammation in HIV+ patients on virologically effective cART with early atherosclerosis Biomarkers of immune dysfunction in HIV.Restoration of CMV-specific-CD4 T cells with ART occurs early and is greater in those with more advanced immunodeficiency.Predictors of CD4:CD8 ratio normalization and its effect on health outcomes in the era of combination antiretroviral therapy.Impact of HIV on CD8+ T cell CD57 expression is distinct from that of CMV and aging.Role of the immune system in HIV-associated neuroinflammation and neurocognitive implications.Delay in cART initiation results in persistent immune dysregulation and poor recovery of T-cell phenotype despite a decade of successful HIV suppression.Herpes simplex virus type 2 serostatus is not associated with inflammatory or metabolic markers in antiretroviral therapy-treated HIV Inhibition of HIV-1 by octadecyloxyethyl esters of (S)-[3-hydroxy-2-(phosphonomethoxy)propyl] nucleosides and evaluation of their mechanism of action.Expansion of CD8+ T cells lacking Sema4D/CD100 during HIV-1 infection identifies a subset of T cells with decreased functional capacity.Immunoactivation at the crossroads of human disease Differential Reduction in Monocyte Activation and Vascular Inflammation With Integrase Inhibitor-Based Initial Antiretroviral Therapy Among HIV-Infected Individuals.Physical function impairment of older, HIV-infected adults is associated with cytomegalovirus immunoglobulin response The immunological footprint of CMV in HIV-1 patients stable on long-term ART Cytomegalovirus immunoglobulin G antibody is associated with subclinical carotid artery disease among HIV-infected women.Virome analysis of antiretroviral-treated HIV patients shows no correlation between T-cell activation and anelloviruses levels.Shared monocyte subset phenotypes in HIV-1 infection and in uninfected subjects with acute coronary syndrome.

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Cytomegalovirus-specific T cells persist at very high levels during long-term antiretroviral treatment of HIV disease.

http://www.wikidata.org/entity/Q33528999

description

2010 nî lūn-bûn

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2010 թուականի Յունուարին հրատարակուած գիտական յօդուած

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2010 թվականի հունվարին հրատարակված գիտական հոդված

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2010年の論文

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2010年論文

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2010年論文

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2010年論文

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2010年論文

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2010年論文

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2010年论文

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name

Cytomegalovirus-specific T cel ...... iral treatment of HIV disease.

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Cytomegalovirus-specific T cel ...... iral treatment of HIV disease.

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Cytomegalovirus-specific T cel ...... iral treatment of HIV disease.

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Cytomegalovirus-specific T cel ...... iral treatment of HIV disease.

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Cytomegalovirus-specific T cel ...... iral treatment of HIV disease.

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Cytomegalovirus-specific T cel ...... iral treatment of HIV disease.

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Cytomegalovirus-specific T cel ...... iral treatment of HIV disease.

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Cytomegalovirus-specific T cel ...... iral treatment of HIV disease.

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Elizabeth Sinclair

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Jeffrey N Martin

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Joseph M McCune

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Peter W Hunt

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Priscilla Hsue

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P2860

Immune activation and CD8+ T-cell differentiation towards senescence in HIV-1 infection

Immunosenescence: deficits in adaptive immunity in the elderly

Inflammatory and coagulation biomarkers and mortality in patients with HIV infection

Biomarkers of immunosenescence within an evolutionary perspective: the challenge of heterogeneity and the role of antigenic load

Autocrine production of beta-chemokines protects CMV-Specific CD4 T cells from HIV infection

Long-term cytomegalovirus infection leads to significant changes in the composition of the CD8+ T-cell repertoire, which may be the basis for an imbalance in the cytokine production profile in elderly persons.

T cell depletion in HIV-1 infection: how CD4+ T cells go out of stock.

Risk factors for mortality in patients with AIDS in the era of highly active antiretroviral therapy.

Longitudinal studies of clonally expanded CD8 T cells reveal a repertoire shrinkage predicting mortality and an increased number of dysfunctional cytomegalovirus-specific T cells in the very elderly.

Cytomegalovirus infection and HIV-1 disease progression in infants born to HIV-1-infected women. Pediatric Pulmonary and Cardiovascular Complications of Vertically Transmitted HIV Infection Study Group.

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1

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David M. Naeger

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David R Bangsberg

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41401457

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20126452

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antiretroviral drug

Cytomegalovirus

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