Helicobacter pylori: molecular evolution of a bacterial quasi-species.
about
Helicobacter pylori and gastroduodenal pathology: new threats of the old friendInteraction of Helicobacter pylori with professional phagocytes: role of the cag pathogenicity island and translocation, phosphorylation and processing of CagA.Helicobacter pylori determinants of pathogenicity.Helicobacter pylori genetic diversity within the gastric niche of a single human hostInduction of monocyte chemoattractant protein 1 by Helicobacter pylori involves NF-kappaB.Comparison of genetic divergence and fitness between two subclones of Helicobacter pyloriIn silico evolutionary analysis of Helicobacter pylori outer membrane phospholipase A (OMPLA)Helicobacter pylori CagA protein can be tyrosine phosphorylated in gastric epithelial cells.Functional plasticity in the type IV secretion system of Helicobacter pyloriHelicobacter pylori: resurrection of the cancer link.Helicobacter pylori vacA s1a and s1b alleles from clinical isolates from different regions of Chile show a distinct geographic distribution.Nucleotide Binding Oligomerization Domain 1 Is an Essential Signal Transducer in Human Epithelial Cells Infected with Helicobacter pylori That Induces the Transepithelial Migration of Neutrophils.Cytokine gene polymorphisms influence mucosal cytokine expression, gastric inflammation, and host specific colonisation during Helicobacter pylori infection.Persistent bacterial infections: the interface of the pathogen and the host immune system.Correlation between cag pathogenicity island composition and Helicobacter pylori-associated gastroduodenal diseaseHelicobacter pylori: phenotypes, genotypes and virulence genes.Genomic structure and insertion sites of Helicobacter pylori prophages from various geographical origins.Strategies used by helicobacter pylori to establish persistent infectionHelicobacter pylori activates the histidine decarboxylase promoter through a mitogen-activated protein kinase pathway independent of pathogenicity island-encoded virulence factors.Pan-genomic analyses identify key Helicobacter pylori pathogenic loci modified by carcinogenic host microenvironments.Microaerophilic conditions permit to mimic in vitro events occurring during in vivo Helicobacter pylori infection and to identify Rho/Ras-associated proteins in cellular signaling.Activation of activator protein 1 and stress response kinases in epithelial cells colonized by Helicobacter pylori encoding the cag pathogenicity island.Molecular evolution between chemistry and biology : The interplay of competition, cooperation, and mutation.
P2860
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P2860
Helicobacter pylori: molecular evolution of a bacterial quasi-species.
description
1998 nî lūn-bûn
@nan
1998 թուականի Փետրուարին հրատարակուած գիտական յօդուած
@hyw
1998 թվականի փետրվարին հրատարակված գիտական հոդված
@hy
1998年の論文
@ja
1998年論文
@yue
1998年論文
@zh-hant
1998年論文
@zh-hk
1998年論文
@zh-mo
1998年論文
@zh-tw
1998年论文
@wuu
name
Helicobacter pylori: molecular evolution of a bacterial quasi-species.
@ast
Helicobacter pylori: molecular evolution of a bacterial quasi-species.
@en
Helicobacter pylori: molecular evolution of a bacterial quasi-species.
@nl
type
label
Helicobacter pylori: molecular evolution of a bacterial quasi-species.
@ast
Helicobacter pylori: molecular evolution of a bacterial quasi-species.
@en
Helicobacter pylori: molecular evolution of a bacterial quasi-species.
@nl
prefLabel
Helicobacter pylori: molecular evolution of a bacterial quasi-species.
@ast
Helicobacter pylori: molecular evolution of a bacterial quasi-species.
@en
Helicobacter pylori: molecular evolution of a bacterial quasi-species.
@nl
P1476
Helicobacter pylori: molecular evolution of a bacterial quasi-species.
@en
P304
P356
10.1016/S1369-5274(98)80148-3
P577
1998-02-01T00:00:00Z